. Interestingly, cell death and differentiation of XL-B4 cells were regulated by T 3 , providing a new insights into the roles of T 3 in cell fate of myoblasts in degenerating tails during metamorphosis. Thus, the XL-B4 cells could be useful for studying the
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Kei Tamura, Shutaro Takayama, Takako Ishii, Shuuji Mawaribuchi, Nobuhiko Takamatsu, and Michihiko Ito
René Lafont, Maria Serova, Blaise Didry-Barca, Sophie Raynal, Louis Guibout, Laurence Dinan, Stanislas Veillet, Mathilde Latil, Waly Dioh, and Pierre J Dilda
specific pharmacological activators or inhibitors of ERs, the former being able to mimic and the latter to inhibit the effects of 20E on target cells such as osteoblasts ( Gao et al. 2008 ) or myoblasts ( Parr et al . 2014 ). These studies, however, do
Kesha Rana, Maria W S Chiu, Patricia K Russell, Jarrod P Skinner, Nicole K L Lee, Barbara C Fam, Jeffrey D Zajac, and Helen E MacLean
through an increase in the number of myonuclei, via activation of satellite cells (muscle stem cells) to proliferate as myoblasts, differentiate into myotubes and fuse with myofibers, or via an increase in protein content of postproliferative myofibers
X Fang, R Palanivel, X Zhou, Y Liu, A Xu, Y Wang, and G Sweeney
cell culture components were from Wisent (Quebec, Canada). Cell culture Rat L6 skeletal myoblasts were grown in α-minimum essential medium (αMEM) containing 10% (v/v)) fetal bovine serum (FBS) (growth medium) under 5
Magdalene O Wilson, Kathleen T Scougall, Jarupa Ratanamart, Elizabeth A McIntyre, and James A M Shaw
. Human myoblasts were cultured in Ham’s F10 medium (Invitrogen, Paisley, Strathclyde, UK) supplemented with 20% FBS, 2% chick embryo extract (ICN, Costa Mesa, CA, USA), 100 U/ml penicillin G and 100 μg/ml streptomycin. C2C12 and human myoblasts
Vivian Vu, Phuong Bui, Megumi Eguchi, Aimin Xu, and Gary Sweeney
to overexpress myc-tagged GLUT4 (a gift from Dr Amira Klip, The Hospital for Sick Children, Toronto) were used as myoblasts when grown to full confluency in α-MEM supplemented with 10% (volume/volume (v/v)) FBS and 1% (v/v) antibiotic
Ana P Irazoqui, Ricardo L Boland, and Claudia G Buitrago
Introduction Recruitment of cells in G0/G1 stage is a pre- and pro-differentiation arrest of skeletal muscle myoblasts, necessary to the subsequent development of myotubes ( Mercer et al . 2005 ). For cell cycle progression, the cellular exit from
Michael A Gentile, Pascale V Nantermet, Robert L Vogel, Robert Phillips, Daniel Holder, Paul Hodor, Chun Cheng, Hongyue Dai, Leonard P Freedman, and William J Ray
directly on satellite or other precursor cells. Murine C2C12 cells, which resemble myogenic precursors, do not express AR, but when AR expression is produced by transfection, AR-selective ligands increase myogenin expression and accelerate myoblast
Avraham I Jacob, Miriam Horovitz-Fried, Shlomit Aga-Mizrachi, Tamar Brutman-Barazani, Hana Okhrimenko, Yehiel Zick, Chaya Brodie, and Sanford R Sampson
and preplated for 20–30 min to reduce the number of fibroblasts. The myoblasts were diluted with growth medium to a concentration of 0.8×10 6 cells/ml for plating in collagen-coated 10-cm plastic tissue culture (10 ml/dish) plates. Cultures were grown
Colin D Clyne, Kevin P Kusnadi, Alexander Cowcher, James Morgan, Jun Yang, Peter J Fuller, and Morag J Young
modify multiple components of the cellular clock, which in turn will have a net effect on the regulation of cell function. In the present study, we demonstrated that the MR and TIMELESS colocalise in the nucleus of cardiac myoblast cells treated with
