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Jenica H Kakadia Department of Biochemistry, Schulich School of Medicine and Dentistry, The University of Western Ontario, London, Ontario, Canada
Children's Health Research Institute, London, Ontario, Canada

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Muhammad U Khalid Department of Biochemistry, Schulich School of Medicine and Dentistry, The University of Western Ontario, London, Ontario, Canada

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Ilka U Heinemann Department of Biochemistry, Schulich School of Medicine and Dentistry, The University of Western Ontario, London, Ontario, Canada

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Victor K Han Department of Biochemistry, Schulich School of Medicine and Dentistry, The University of Western Ontario, London, Ontario, Canada
Children's Health Research Institute, London, Ontario, Canada
Department of Pediatrics, Schulich School of Medicine and Dentistry, The University of Western Ontario, London, Ontario, Canada

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lower, as seen in fetal development) in conjunction with glucose deprivation could increase cell stress, and confound results. Future experiments will show if these findings are translatable to FGR reveal if chronic hypoglycemia elicits IGFBP-1

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-23-0137
Volume/Issue:
Volume 72: Issue 3
Open access
Gemma Tan Department of Anatomy and Developmental Biology, Murdoch Childrens Research Institute, Department of Paediatrics, Monash University, Building 73, Clayton, Victoria 3800, Australia
Department of Anatomy and Developmental Biology, Murdoch Childrens Research Institute, Department of Paediatrics, Monash University, Building 73, Clayton, Victoria 3800, Australia

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Andrew G Elefanty Department of Anatomy and Developmental Biology, Murdoch Childrens Research Institute, Department of Paediatrics, Monash University, Building 73, Clayton, Victoria 3800, Australia
Department of Anatomy and Developmental Biology, Murdoch Childrens Research Institute, Department of Paediatrics, Monash University, Building 73, Clayton, Victoria 3800, Australia
Department of Anatomy and Developmental Biology, Murdoch Childrens Research Institute, Department of Paediatrics, Monash University, Building 73, Clayton, Victoria 3800, Australia

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Edouard G Stanley Department of Anatomy and Developmental Biology, Murdoch Childrens Research Institute, Department of Paediatrics, Monash University, Building 73, Clayton, Victoria 3800, Australia
Department of Anatomy and Developmental Biology, Murdoch Childrens Research Institute, Department of Paediatrics, Monash University, Building 73, Clayton, Victoria 3800, Australia
Department of Anatomy and Developmental Biology, Murdoch Childrens Research Institute, Department of Paediatrics, Monash University, Building 73, Clayton, Victoria 3800, Australia

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blood glucose afforded by properly functioning islets. Moreover, inappropriate balance between insulin and glucose intake can cause severe hypoglycemia, a situation that can be exacerbated during acute illnesses ( Krinsley et al . 2011 ). Critically

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JME-14-0188
Volume/Issue:
Volume 53: Issue 3
Open access
Eva A Rog-Zielinska Queen's Medical Research Institute, Centre for Cardiovascular Science, University of Edinburgh, Edinburgh EH16 4TJ, UK

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Rachel V Richardson Queen's Medical Research Institute, Centre for Cardiovascular Science, University of Edinburgh, Edinburgh EH16 4TJ, UK

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Martin A Denvir Queen's Medical Research Institute, Centre for Cardiovascular Science, University of Edinburgh, Edinburgh EH16 4TJ, UK

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Karen E Chapman Queen's Medical Research Institute, Centre for Cardiovascular Science, University of Edinburgh, Edinburgh EH16 4TJ, UK

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Thyrotropin-Releasing Hormone Study Group . American Journal of Obstetrics and Gynecology 181 709 – 717 . ( doi:10.1016/S0002-9378(99)70517-X ). Barker DJ Fall CH 1993 Fetal and infant origins of cardiovascular disease . Archives of Disease in Childhood 68

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JME-13-0204
Volume/Issue:
Volume 52: Issue 2
Free access
Sofia A Rahman Genetics and Genomic Medicine, Department of Paediatric Endocrinology, UCL Institute of Child Health, 30 Guilford Street, London WC1N 1EH, UK

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Azizun Nessa Genetics and Genomic Medicine, Department of Paediatric Endocrinology, UCL Institute of Child Health, 30 Guilford Street, London WC1N 1EH, UK

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Khalid Hussain Genetics and Genomic Medicine, Department of Paediatric Endocrinology, UCL Institute of Child Health, 30 Guilford Street, London WC1N 1EH, UK
Genetics and Genomic Medicine, Department of Paediatric Endocrinology, UCL Institute of Child Health, 30 Guilford Street, London WC1N 1EH, UK

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Practical management of hyperinsulinism in infancy . Archives of Disease in Childhood. Fetal and Neonatal Edition 82 F98 – F107 . ( doi:10.1136/fn.82.2.F98 ). Bruining GJ 1990 Recent advances in hyperinsulinism and the pathogenesis of diabetes

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JME-15-0016
Volume/Issue:
Volume 54: Issue 2
Free access
Ahter D Sanlioglu Human Gene and Cell Therapy Center, Division of Endocrinology and Metabolism, Division of Child Neurology, Department of Urology, Akdeniz University Hospitals and Clinics, B Block, 1st floor, Campus, Antalya 07058, Turkey
Human Gene and Cell Therapy Center, Division of Endocrinology and Metabolism, Division of Child Neurology, Department of Urology, Akdeniz University Hospitals and Clinics, B Block, 1st floor, Campus, Antalya 07058, Turkey

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Bahri Karacay Human Gene and Cell Therapy Center, Division of Endocrinology and Metabolism, Division of Child Neurology, Department of Urology, Akdeniz University Hospitals and Clinics, B Block, 1st floor, Campus, Antalya 07058, Turkey

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Mustafa Kemal Balci Human Gene and Cell Therapy Center, Division of Endocrinology and Metabolism, Division of Child Neurology, Department of Urology, Akdeniz University Hospitals and Clinics, B Block, 1st floor, Campus, Antalya 07058, Turkey
Human Gene and Cell Therapy Center, Division of Endocrinology and Metabolism, Division of Child Neurology, Department of Urology, Akdeniz University Hospitals and Clinics, B Block, 1st floor, Campus, Antalya 07058, Turkey

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Thomas S Griffith Human Gene and Cell Therapy Center, Division of Endocrinology and Metabolism, Division of Child Neurology, Department of Urology, Akdeniz University Hospitals and Clinics, B Block, 1st floor, Campus, Antalya 07058, Turkey

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Salih Sanlioglu Human Gene and Cell Therapy Center, Division of Endocrinology and Metabolism, Division of Child Neurology, Department of Urology, Akdeniz University Hospitals and Clinics, B Block, 1st floor, Campus, Antalya 07058, Turkey
Human Gene and Cell Therapy Center, Division of Endocrinology and Metabolism, Division of Child Neurology, Department of Urology, Akdeniz University Hospitals and Clinics, B Block, 1st floor, Campus, Antalya 07058, Turkey

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was reported in insulin-mediated experimentally induced hypoglycemia in PAC1 −/− mice ( Persson & Ahren 2002 ). Thus, PACAP-stimulated PAC1 receptor activation mediates glucagon response to insulin-induced hypoglycemia. Assessments of VIP receptor

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-12-0156
Volume/Issue:
Volume 49: Issue 3
Free access
Run Yu
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Chun-Rong Chen
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Xiaohong Liu Division of Endocrinology and Carcinoid and Neuroendocrine Tumor Center, Gilead Sciences, Novo Nordisk A/S, Cedars-Sinai Medical Center, B-131, 8700 Beverly Boulevard, Los Angeles, California 90048, USA

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János T Kodra Division of Endocrinology and Carcinoid and Neuroendocrine Tumor Center, Gilead Sciences, Novo Nordisk A/S, Cedars-Sinai Medical Center, B-131, 8700 Beverly Boulevard, Los Angeles, California 90048, USA

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) and grown in DMEM supplemented with 10% fetal bovine serum at 37 °C (unless specified otherwise) in a humidified incubator circulated with 5% CO 2 . Tunicamycin, DMSO, glycerol, trimethylamine N -oxide (TMAO), 4-phenylbutyric acid (PBA), thapsigargin

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-12-0051
Volume/Issue:
Volume 49: Issue 2
Free access
Sushi Jiang Department of Physiology, School of Medicine, Jinan University, Guangzhou, Guangdong, China

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Hening Zhai Department of Physiology, School of Medicine, Jinan University, Guangzhou, Guangdong, China
Endoscopy Center, The First Affiliated Hospital of Jinan University, Guangzhou, Guangdong, China

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Danjie Li Department of Physiology, School of Medicine, Jinan University, Guangzhou, Guangdong, China

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Jiana Huang Department of Physiology, School of Medicine, Jinan University, Guangzhou, Guangdong, China

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Heng Zhang Department of Physiology, School of Medicine, Jinan University, Guangzhou, Guangdong, China

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Ziru Li Shenzhen University Diabetes Center, Shenzhen University Health Science Center, Shenzhen, Guangdong, China

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Weizhen Zhang Shenzhen University Diabetes Center, Shenzhen University Health Science Center, Shenzhen, Guangdong, China
Department of Surgery, University of Michigan Medical Center, Ann Arbor, Michigan, USA

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Geyang Xu Department of Physiology, School of Medicine, Jinan University, Guangzhou, Guangdong, China

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( Herrmann et al . 1995 ). Such glucose-dependent action is attractive particularly because when the plasma glucose concentration is in the normal fasting range, GLP1 no longer stimulates insulin release, thus avoiding hypoglycemia ( Elahi et al . 1994

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-16-0099
Volume/Issue:
Volume 57: Issue 3
Free access
Raul M Luque
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Manuel D Gahete
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Rudy J Valentine
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Rhonda D Kineman
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the majority are confined to studies involving GH-producing adenomas or fetal pituitaries ( Oosterom et al. 1984 , Nakagawa et al. 1985 , Goodyer et al. 1986 , Yamashita et al. 1986 , Isaacs et al. 1987 , Lloyd et al. 1993 ). The bulk

Article Type:
Research Article
DOI:
https://doi.org/10.1677/jme.1.02042
Volume/Issue:
Volume 37: Issue 1
Free access
E Anastasi Department of Clinical Sciences, University ‘La Sapienza’, Rome, Italy
National Centre for Food Quality and Risk Assessment, Section of Nutrition, Istituto Superiore di Sanità, Rome, Italy
Division of Endocrinology and Metabolism, Cedars-Sinai Medical Center, University of California, Los Angeles, CA, USA
Department of Experimental Medicine and Pathology, University ‘La Sapienza’, Rome, Italy

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C Santangelo Department of Clinical Sciences, University ‘La Sapienza’, Rome, Italy
National Centre for Food Quality and Risk Assessment, Section of Nutrition, Istituto Superiore di Sanità, Rome, Italy
Division of Endocrinology and Metabolism, Cedars-Sinai Medical Center, University of California, Los Angeles, CA, USA
Department of Experimental Medicine and Pathology, University ‘La Sapienza’, Rome, Italy

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A Bulotta Department of Clinical Sciences, University ‘La Sapienza’, Rome, Italy
National Centre for Food Quality and Risk Assessment, Section of Nutrition, Istituto Superiore di Sanità, Rome, Italy
Division of Endocrinology and Metabolism, Cedars-Sinai Medical Center, University of California, Los Angeles, CA, USA
Department of Experimental Medicine and Pathology, University ‘La Sapienza’, Rome, Italy

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F Dotta Department of Clinical Sciences, University ‘La Sapienza’, Rome, Italy
National Centre for Food Quality and Risk Assessment, Section of Nutrition, Istituto Superiore di Sanità, Rome, Italy
Division of Endocrinology and Metabolism, Cedars-Sinai Medical Center, University of California, Los Angeles, CA, USA
Department of Experimental Medicine and Pathology, University ‘La Sapienza’, Rome, Italy

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B Argenti Department of Clinical Sciences, University ‘La Sapienza’, Rome, Italy
National Centre for Food Quality and Risk Assessment, Section of Nutrition, Istituto Superiore di Sanità, Rome, Italy
Division of Endocrinology and Metabolism, Cedars-Sinai Medical Center, University of California, Los Angeles, CA, USA
Department of Experimental Medicine and Pathology, University ‘La Sapienza’, Rome, Italy

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C Mincione Department of Clinical Sciences, University ‘La Sapienza’, Rome, Italy
National Centre for Food Quality and Risk Assessment, Section of Nutrition, Istituto Superiore di Sanità, Rome, Italy
Division of Endocrinology and Metabolism, Cedars-Sinai Medical Center, University of California, Los Angeles, CA, USA
Department of Experimental Medicine and Pathology, University ‘La Sapienza’, Rome, Italy

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A Gulino Department of Clinical Sciences, University ‘La Sapienza’, Rome, Italy
National Centre for Food Quality and Risk Assessment, Section of Nutrition, Istituto Superiore di Sanità, Rome, Italy
Division of Endocrinology and Metabolism, Cedars-Sinai Medical Center, University of California, Los Angeles, CA, USA
Department of Experimental Medicine and Pathology, University ‘La Sapienza’, Rome, Italy

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M Maroder Department of Clinical Sciences, University ‘La Sapienza’, Rome, Italy
National Centre for Food Quality and Risk Assessment, Section of Nutrition, Istituto Superiore di Sanità, Rome, Italy
Division of Endocrinology and Metabolism, Cedars-Sinai Medical Center, University of California, Los Angeles, CA, USA
Department of Experimental Medicine and Pathology, University ‘La Sapienza’, Rome, Italy

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R Perfetti Department of Clinical Sciences, University ‘La Sapienza’, Rome, Italy
National Centre for Food Quality and Risk Assessment, Section of Nutrition, Istituto Superiore di Sanità, Rome, Italy
Division of Endocrinology and Metabolism, Cedars-Sinai Medical Center, University of California, Los Angeles, CA, USA
Department of Experimental Medicine and Pathology, University ‘La Sapienza’, Rome, Italy

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U Di Mario Department of Clinical Sciences, University ‘La Sapienza’, Rome, Italy
National Centre for Food Quality and Risk Assessment, Section of Nutrition, Istituto Superiore di Sanità, Rome, Italy
Division of Endocrinology and Metabolism, Cedars-Sinai Medical Center, University of California, Los Angeles, CA, USA
Department of Experimental Medicine and Pathology, University ‘La Sapienza’, Rome, Italy

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) containing 10% fetal bovine serum (FBS) (Gibco), 50 μg/ml streptomycin, 100 μg/ml penicillin at 37°C under a humidified condition of 95% air, and 5% CO 2 . Hep-G2 cells were cultured in Minimum Alpha Essential Medium (Gibco, BRL) supplemented with FBS and

Article Type:
Research Article
DOI:
https://doi.org/10.1677/jme.1.01595
Volume/Issue:
Volume 34: Issue 2
Free access
Amanda M Ackermann Departments of Medicine,
Molecular Physiology and Biophysics and
Program in Developmental Biology, Vanderbilt University Medical Center, 2220 Pierce Avenue 746 PRB, Nashville, Tennessee 37232, USA

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Maureen Gannon Departments of Medicine,
Molecular Physiology and Biophysics and
Program in Developmental Biology, Vanderbilt University Medical Center, 2220 Pierce Avenue 746 PRB, Nashville, Tennessee 37232, USA

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levels of glucocorticoids, which can independently reduce fetal β-cell mass when exogenously administered to the mother. IUGR caused by protein restriction also results in underdeveloped β-cell mass in newborn rats, but this is due to reduced β

Article Type:
Review Article
DOI:
https://doi.org/10.1677/JME-06-0053
Volume/Issue:
Volume 38: Issue 2
Free access