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Soojin Kim, Daksh Thaper, Samir Bidnur, Paul Toren, Shusuke Akamatsu, Jennifer L Bishop, Colin Colins, Sepideh Vahid, and Amina Zoubeidi

(LBD) ( Balbas et al. 2013 , Joseph et al. 2013 , Korpal et al. 2013 , Li et al. 2013 ). In other cases, after multi-agent AR-axis inhibition, aggressive CRPC cancers demonstrate a non-AR-driven phenotype with neuroendocrine (NE) features

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Sucharitha Iyer and Sunita K Agarwal

endocrine cell types that form tumors are located in the parathyroids, anterior pituitary and the duodenopancreatic neuroendocrine tissues ( Thakker et al . 2012 ). Other MEN1-associated endocrine tumors include foregut carcinoids that arise from

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William Rostene and Julia C Buckingham

neuromodulators of neuroendocrine functions ( Kitabgi et al. 2006 ). These emerging concepts were illustrated by pioneers in the field at a symposium entitled ‘Chemokines as modulators of neuroendocrine functions’ which was held at the sixth International

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Matei Bolborea, Gisela Helfer, Francis J P Ebling, and Perry Barrett

other unknown aspects of ependymal cell physiology that affect hypothalamic–neuroendocrine communication. Declaration of interest The authors declare that there is no conflict of interest that could be perceived as prejudicing the impartiality of the

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David García-Galiano, Victor M Navarro, Francisco Gaytan, and Manuel Tena-Sempere

gonads ( Fig. 3 ; our unpublished data) Unknown Neuroendocrine mechanisms and pathways for the anorectic effects of nesfatin-1 In addition to the above-mentioned neuroanatomical evidence, functional analyses on the metabolic regulation of NUCB2/nesfatin

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Shereen Ezzat and Sylvia L Asa

-opiomelanocortin (POMC), growth hormone (GH), prolactin (PRL), and GH-releasing hormone (GHRH). We have also identified a role for Ikaros in modulating neuroendocrine cell growth and survival functions ( Ezzat et al . 2003 , 2006 b ). In this review, we cover the

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Céline Callewaere, Ghazal Banisadr, William Rostène, and Stéphane Mélik Parsadaniantz

function as neuromodulators. Among the described neuromodulatory activities of chemokines, the current literature postulates that chemokines could directly interact with neuroendocrine functions. It is this latter new concept that we would like to

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B C J Dirven, J R Homberg, T Kozicz, and M J A G Henckens

activation of different response systems in the body. Crucial to the stress response is the neuroendocrine system, which tightly regulates adaptive processes following stress exposure ( Miller & O’Callaghan 2002 ). The primary endocrine effectors of the

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Jennifer A Chalmers, Shuo-Yen J Lin, Tami A Martino, Sara Arab, Peter Liu, Mansoor Husain, Michael J Sole, and Denise D Belsham

1997 , Charloux et al . 1999 ). In this study, we focused on POMC gene expression in normal murine heart and pressure-overload cardiac hypertrophy. In addition, we extended investigations toward additional neuroendocrine genes expressed in the

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Timon Vandamme, Marc Peeters, Fadime Dogan, Patrick Pauwels, Elvire Van Assche, Matthias Beyens, Geert Mortier, Geert Vandeweyer, Wouter de Herder, Guy Van Camp, Leo J Hofland, and Ken Op de Beeck

Introduction Pancreatic neuroendocrine tumors (PNETs) are malignant tumors probably arising from islet cells of the pancreas ( Schimmack et al . 2011 ). PNETs can secrete a variety of hormones including somatostatin, insulin, glucagon, serotonin