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Inagadapa J N Padmavathi, Kalashikam Rajender Rao and Manchala Raghunath

response to oral glucose challenge ( Berney et al . 1997 , Reusens & Remacle 2001 ). We showed earlier that maternal micronutrient restriction increased body fat% especially the central adiposity, impaired plasma lipids, altered glucose tolerance

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Shaoqian Zhao, Wen Liu, Jiqiu Wang, Juan Shi, Yingkai Sun, Weiqing Wang, Guang Ning, Ruixin Liu and Jie Hong

improved adiposity, glucose tolerance and low-grade inflammation in chow diet-fed mice by A. muciniphila . Hence, our research provided new evidence for the metabolic benefits of A. muciniphila on chow diet-fed mice, pointing out the possible mechanisms

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Ji Seon Park, Su Jung Bae, Sik-Won Choi, You Hwa Son, Sung Bum Park, Sang Dal Rhee, Hee Youn Kim, Won Hoon Jung, Seung Kyu Kang, Jin Hee Ahn, Seong Hwan Kim and Ki Young Kim

tissue in 11β-HSD1-overexpressed mice showed metabolic syndrome-like phenotypes such as central obesity, glucose tolerance, and insulin resistance ( Masuzaki et al . 2001 , Paterson et al . 2004 ). By contrast, 11β-HSD1-deficient mice showed a

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Sufang Chen, Wei Wei, Minjie Chen, Xiaobo Qin, Lianglin Qiu, Li Zhang, Yuhao Zhang, Qi Cao and Zhekang Ying

impairs glucose tolerance, which is widely believed to be dependent on TNFα-induced serine phosphorylation of insulin receptor substrates (IRSs) ( Hotamisligil et al . 1993 ). In addition, the increased tolerance to glucose in TNFα − / − mice

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K Lindberg, S G Rønn, D Tornehave, H Richter, J A Hansen, J Rømer, M Jackerott and N Billestrup

from free insulin ( Bonnevie-Nielsen 1980 ). Plasma insulin Plasma insulin concentrations were determined using a rat insulin RIA kit (Linco Research, St Charles, MO, USA). Oral glucose-tolerance

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Zhipeng Li, Zhaoshui Shangguan, Yijie Liu, Jihua Wang, Xuejun Li, Shuyu Yang and Suhuan Liu

. 2012 ), anti-inflammation ( Huang et al . 2012 , Jin et al . 2012 ), and anti-apoptosis ( Liang et al . 2012 , Zhang et al . 2012 ) properties. It has been reported that puerarin improved insulin resistance and glucose tolerance ( Prasain et al

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Carolina Gustavsson, Tomoyoshi Soga, Erik Wahlström, Mattias Vesterlund, Alireza Azimi, Gunnar Norstedt and Petra Tollet-Egnell

during development of NAFLD and insulin resistance. For example, it was shown that oestrogen receptor α knockout mice develop fatty liver, hepatic insulin resistance and impaired glucose tolerance ( Bryzgalova et al . 2006 ). High-fat or high

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Guojun Shi, Chen Sun, Weiqiong Gu, Minglan Yang, Xiaofang Zhang, Nan Zhai, Yan Lu, Zhijian Zhang, Peishun Shou, Zhiguo Zhang and Guang Ning

and lymphocytes, which are critical components of the immune system in both defense and autoimmune diseases. Our results indicate that the activation of FFAR2 improves glucose tolerance by inducing apoptosis of infiltrated immune cells. The

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Pamela Petrocchi-Passeri, Cheryl Cero, Alessandro Cutarelli, Claudio Frank, Cinzia Severini, Alessandro Bartolomucci and Roberta Possenti

, Bartolomucci et al . 2006 ) and showing activity on synaptic plasticity ( Alder et al . 2003 , Bozdagi et al . 2008 , Thakker-Varia et al . 2014 ) – was the most potent in inducing insulin secretion and in improving glucose tolerance in vivo . Materials

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Qinyun Ma, Jianxia Fan, Jiqiu Wang, Shuai Yang, Qing Cong, Rui Wang, Qianqian Lv, Ruixin Liu and Guang Ning

, which possibly contributes to GDM development. Materials and methods Patients In this study, a standard 75 g oral glucose tolerance test (OGTT) was performed on pregnant women with fasting plasma glucose of 4.4 mmol/l or greater at 24–28 weeks of