-gestation sheep fetuses whose mothers were undernourished (UN) during specific windows of gestation, but no clear consensus has emerged regarding the impact of nutrition on fetal growth, PAT depot mass or gene expression ( Symonds et al . 1998 , Bispham et al
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Jacqueline M Wallace, John S Milne, Raymond P Aitken, Dale A Redmer, Lawrence P Reynolds, Justin S Luther, Graham W Horgan, and Clare L Adam
Ashley A Able, Allison J Richard, and Jacqueline M Stephens
these direct STAT5 target genes, very little is known about the molecular mechanisms that contribute to the ability of STAT5A to regulate gene expression in adipocytes. To further investigate the functions of STAT5A in adipocytes, we sought to identify
Jacqueline M Wallace, John S Milne, Raymond P Aitken, and Clare L Adam
biomarkers may be an early presage of later metabolic health. Thus our aim herein was to examine the impact of poor prenatal growth followed by unlimited postnatal nutrition on plasma lipid status and adipose tissue gene expression in both male and female
Ciro Menale, Maria Teresa Piccolo, Grazia Cirillo, Raffaele A Calogero, Alfonso Papparella, Luigi Mita, Emanuele Miraglia Del Giudice, Nadia Diano, Stefania Crispi, and Damiano Gustavo Mita
studies have demonstrated that a large number of genes that are modulated by BPA induce negative health effects ( Singh & Li 2012 ). Specifically, BPA exposure during adipocyte differentiation affects gene expression and subsequently modulates adipose
Xi-Long Zheng and Norman C W Wong
diseases ( Rubin et al. 1991 , Barter & Rye 1996 a , Luoma 1997 ). Owing to the pivotal role of apo AI in the functions of HDL particles, it is critical to identify mechanisms underlying either stimulation or inhibition of apo AI gene expression
Alina Gajewska, Andrzej P Herman, Ewa Wolińska-Witort, Kazimierz Kochman, and Lech Zwierzchowski
single specific product. The identity of PCR products was further confirmed by direct sequencing in both directions (Oligo IBB, Warsaw, Poland). Relative gene expression was calculated using the comparative quantitation option of Rotor Gene 6000 software
Ann Lo, Weiming Zheng, Yimei Gong, John R Crochet, and Lisa M Halvorson
gonadotropin and GnRH receptor gene expression, including SF-1, LRH-1, Sp1, and AP1 ( Kawana et al . 1995 , Fluck & Miller 2004 , Bouchard et al . 2009 ). Although first characterized in the hematopoietic and cardiac systems, GATA factors are now known to
H Watanabe, A Suzuki, M Goto, S Ohsako, C Tohyama, H Handa, and T Iguchi
to investigate the estrogenic activity of TCDD. Accordingly, we determined the effects of TCDD and estrogen on gene expression and compared their gene expression patterns. By this approach, we estimated the estrogenic effects of TCDD
Toshihiro Kobayashi, Hitomi Imachi, Kensaku Fukunaga, Jingya Lyu, Seisuke Sato, Takanobu Saheki, Tomohiro Ibata, Mari Matsumoto, Salimah B Japar, and Koji Murao
this study, we have examined the effect of HDL-hSR-BI/CLA-1 signal transduction mechanism on APN gene expression. Materials and methods Cell culture The 3T3L1 cells originated from a mouse adipose cell line (American Type Culture Collection
Ke-Hung Tsui, Li-Chuan Chung, Shyi-Wu Wang, Tsui-Hsia Feng, Phei-Lang Chang, and Horng-Heng Juang
mACON gene expression. Fatty acid metabolism rather than glycolysis is reported as the energy fuel for prostate carcinoma cells ( Liu 2006 ). The glycolysis–citrate–lipogenesis pathways in providing the synthetic and bioenergetic requirement are
