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concentrations, a threshold named ‘metabolic endotoxemia’ (ME; Cani et al . 2007 a ). ME may trigger toll-like receptor (TLR) 4-mediated inflammatory activation, eliciting a chronic low-grade proinflammatory and pro-oxidative stress status associated with
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Division of Endocrinology, Department of Cell Biology and Biochemistry, Instituto de Investigaciones Biomédicas Alberto Sols, Unidade de Enfermedades Metabólicas e Tiroideas (UETeM), Department of Internal Medicine
Division of Endocrinology, Department of Cell Biology and Biochemistry, Instituto de Investigaciones Biomédicas Alberto Sols, Unidade de Enfermedades Metabólicas e Tiroideas (UETeM), Department of Internal Medicine
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well as on the activity of DIO enzymes at a multi-tissue level, and iii) NF-kB nuclear binding activity and its correlation with the tissue-specific decrease in THRB gene expression observed during endotoxemia. Materials and methods Animals The study
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Laboratory of Endocrinology and Metabolism, Institute of Health Sciences, Shanghai Institutes for Biological Sciences (SIBS), Chinese Academy of Sciences (CAS) & Shanghai Jiao Tong University School of Medicine (SJTUSM), Shanghai, China
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richness in the microbiome ( Le Chatelier et al . 2013 ). Many studies demonstrated that gut microbiota maintain energy homeostasis by affecting gut barrier, which is important for metabolic endotoxemia and metabolic disorders ( Cani et al . 2009 ). These
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. Excessive body fat accumulation and accompanied chronic inflammation in adipose tissue are the main characteristics of obesity. Obesity and aging share many common biological and metabolic alterations, including metabolic endotoxemia ( Pérez et al. 2016
Institute of Liver Diseases, Shuguang Hospital Affiliated to Shanghai University of Traditional Chinese Medicine, Shanghai, China
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. Endotoxemia-mediated P300 induction through the activation of the IRE1-XBP1s pathway A high-fat, western-style diet is an important predisposing factor for the onset of diabetes and obesity. HFD feeding results in changes in gut microbiota that initiate
Department of Physiology and Pathophysiology, Department of Pathology, Division of Medicine, Department of Surgery, Peking University Health Science Center; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Beijing 100191, China
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Department of Physiology and Pathophysiology, Department of Pathology, Division of Medicine, Department of Surgery, Peking University Health Science Center; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Beijing 100191, China
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of LPS had no effect on insulin sensitivity. Our data suggest that low dose of LPS may improve the glucose tolerance. Consistent with our study, van der Crabben et al . (2009) showed that early endotoxemia increases peripheral and hepatic insulin
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Interactions between CBP, NF-kappaB, and CREB in the lungs after hemorrhage and endotoxemia. American Journal of Physiology. Lung Cellular and Molecular Physiology 281 L418 –L426. Sheppard KA , Rose DW, Haque ZK, Kurokawa R
Center for Reproductive Health Sciences, Department of Obstetrics and Gynecology, Washington University School of Medicine, St. Louis, Missouri, USA
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Center for Reproductive Health Sciences, Department of Obstetrics and Gynecology, Washington University School of Medicine, St. Louis, Missouri, USA
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, PCOS patients showed alterations in some, but not all, markers of gut barrier function and endotoxemia ( Lindheim et al. 2017 ). Finally, Qi et al. examined 43 healthy controls ( n = 43) and 50 PCOS patients ( n = 50) and found that Bacteroides
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of endotoxemia with carotid atherosclerosis and cardiovascular disease: prospective results from the Bruneck Study . Journal of the American College of Cardiology 34 1975 – 1981 . Wu J Gong J Geng J Song Y 2008 Deoxycholic acid
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. This drug combination has previously been shown to counter endotoxemia by rescuing the gut ultrastructure and mitigating hepatic steatosis via reduced TLR4 expression in the liver of high-fructose-fed mice ( Silva-Veiga et al. 2020 ). Previous