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E Grossini, C Molinari, L Sigaudo, M Biella, D A S G Mary and G Vacca

performed in porcine coronary artery endothelial cells (PCAEC) have shown an acute and dose-dependent endothelial NO synthase (eNOS)-related NO production in response to gastrin-17, which was modulated by β 2 -adrenoreceptor agonists/antagonists and was

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Leon A Bach

-independent actions. Endothelial cells line blood vessels and act as barriers that control the transfer of biomolecules and cells from the circulation to tissues ( Sumpio et al . 2002 ). They are metabolically active and regulate vascular tone, thrombosis

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R Gruemmer, L Klein-Hitpaß and J Neulen

2000 a ). In order to investigate the role of follicular fluid constituents and of granulosa cells in the expression of angiogenesis-related endothelial genes, human endothelial cells were incubated in vitro with human follicular fluid or

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Limin Tian, Luyan Zhang, Jing Liu, Tiankang Guo, Cuixia Gao and Jing Ni

accelerate AS. The central events of AS include endothelial dysfunction ( Atochin & Huang 2010 ), inflammation, and vascular smooth muscle cells (VSMCs) proliferation. It is worth noting that endothelial cell dysfunction is generally accepted as an early step

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Qi Zhang, Jing Liu, Jia Liu, Wenhui Huang, Limin Tian, Jinxing Quan, Yunfang Wang and Ruilan Niu

with hepatic sinusoid microcirculation ( Farrell et al . 2008 ). Liver sinusoidal endothelial cells (LSECs) play important roles in regulating hepatic sinus function. LSECs line the capillaries of the microvasculature and possess fenestrae that

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Özlem Erdogdu, Linnéa Eriksson, Hua Xu, Åke Sjöholm, Qimin Zhang and Thomas Nyström

endothelial cells (HCAECs) express the GLP1 receptor and that exendin-4 is able to activate the receptor, leading to increased cell proliferation, involving the eNOS pathway ( Erdogdu et al . 2010 ). GLP1 has also been described as an anti-apoptotic factor in

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Pabitra B Pal, Himangshu Sonowal, Kirtikar Shukla, Satish K Srivastava and Kota V Ramana

Introduction Hyperglycemia is a significant risk factor as well as a contributor to endothelial dysfunction and resultant cardiovascular complications frequently associated with diabetes. Generally, endothelial cells maintain the balance of

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Yanxia Tang and GuoDong Li

the study of endothelial dysfunction since it was recognized in 1987 ( Moncada et al . 1991 ). In endothelial cells (ECs), NO is synthesized by endothelial NO synthase (eNOS) and eNOS activity is regulated mainly by a change in cytosolic free Ca 2

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Ying Xing, Jingbo Lai, Xiangyang Liu, Nana Zhang, Jie Ming, Hengxin Liu and Xi Zhang

suppressing endothelial cell activation and leukocyte infiltration ( Wang et al . 2008 ). Emerging report demonstrated the reduction of plasma netrin-1 levels in patients diagnosed with type 2 DM, and netrin-1 levels was found to be negatively related to

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Philippe Huber, Christine Mallet, Elodie Faure, Christine Rampon, Marie-Hélène Prandini, Olivier Féraud, Stéphanie Bouillot and Isabelle Vilgrain

was correlated with a decrease in VE-cadherin content. However, endothelial/total cell ratios were maintained during one or both hormone treatments, suggesting that VE-cadherin expression was downregulated in endothelial cells of hypotrophic adrenals