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Yali Ling National Clinical Research Center for Metabolic Diseases, Hunan Provincial Key Laboratory of Metabolic Bone Diseases, and Department of Metabolism and Endocrinology, The Second Xiangya Hospital of Central South University, Changsha, Hunan, China

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Feng Xu National Clinical Research Center for Metabolic Diseases, Hunan Provincial Key Laboratory of Metabolic Bone Diseases, and Department of Metabolism and Endocrinology, The Second Xiangya Hospital of Central South University, Changsha, Hunan, China

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Xuedi Xia National Clinical Research Center for Metabolic Diseases, Hunan Provincial Key Laboratory of Metabolic Bone Diseases, and Department of Metabolism and Endocrinology, The Second Xiangya Hospital of Central South University, Changsha, Hunan, China

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Dexing Dai National Clinical Research Center for Metabolic Diseases, Hunan Provincial Key Laboratory of Metabolic Bone Diseases, and Department of Metabolism and Endocrinology, The Second Xiangya Hospital of Central South University, Changsha, Hunan, China

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Ruoman Sun National Clinical Research Center for Metabolic Diseases, Hunan Provincial Key Laboratory of Metabolic Bone Diseases, and Department of Metabolism and Endocrinology, The Second Xiangya Hospital of Central South University, Changsha, Hunan, China

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Zhongjian Xie National Clinical Research Center for Metabolic Diseases, Hunan Provincial Key Laboratory of Metabolic Bone Diseases, and Department of Metabolism and Endocrinology, The Second Xiangya Hospital of Central South University, Changsha, Hunan, China

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cycling in VDR knockout mice ( Skorija et al. 2005 ), suggesting that the role of VDR in epidermal hair regulation is independent of its ligand. VDR interacts with β-catenin to regulate the transcription of target genes, thus participating in the

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-21-0167
Volume/Issue:
Volume 68: Issue 3
Open access
Custodia García-Jiménez
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Jose Manuel García-Martínez
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Ana Chocarro-Calvo
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Antonio De la Vieja Departamento de Fisiología y Bioquímica, Unidad Funcional de Investigación en Enfermedades Crónicas (UFIEC), Facultad de Ciencias de la Salud, Universidad Rey Juan Carlos, 28922 Alcorcon, Madrid, Spain

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unexplored aspect is the influence of high glucose levels on cancer signaling. In this context, high glucose has been shown to be essential for WNT/β-catenin signaling, a key cancer-associated pathway ( Anagnostou & Shepherd 2008 , Chocarro-Calvo et al

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JME-13-0152
Volume/Issue:
Volume 52: Issue 1
Free access
Jianqiu Gu Department of Endocrinology and Metabolism and the Institute of Endocrinology, The First Hospital of China Medical University, Shenyang, People’s Republic of China
Division of Advanced Diagnostics, Toronto General Hospital Research Institute, University Health Network, Toronto, Canada

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Weijuan Shao Division of Advanced Diagnostics, Toronto General Hospital Research Institute, University Health Network, Toronto, Canada

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Dinghui Liu Division of Advanced Diagnostics, Toronto General Hospital Research Institute, University Health Network, Toronto, Canada
Department of Cardiology, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, People’s Republic of China

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Jia Nuo Feng Division of Advanced Diagnostics, Toronto General Hospital Research Institute, University Health Network, Toronto, Canada
Department of Physiology, Temerty Faculty of Medicine, University of Toronto, Toronto, Canada

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Juan Pang Division of Advanced Diagnostics, Toronto General Hospital Research Institute, University Health Network, Toronto, Canada
Department of Nutrition, School of Public Health, Sun Yat-sen University, Guangzhou, People’s Republic of China

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Tianru Jin Division of Advanced Diagnostics, Toronto General Hospital Research Institute, University Health Network, Toronto, Canada
Department of Physiology, Temerty Faculty of Medicine, University of Toronto, Toronto, Canada
Banting and Best Diabetes Centre, Temerty Faculty of Medicine, University of Toronto, Toronto, Canada

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adipose tissues during adipogenesis and in adulthood are more complicated and require further investigations ( Chen et al. 2018 ). Another key effector of Wnt signaling pathway is β-catenin (β-cat). Functions of this co-transcriptional activator can be

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-22-0026
Volume/Issue:
Volume 69: Issue 2
Restricted access
Huixia Li Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Xi’an Jiaotong University Health Science Center, Xi’an, Shaanxi, China

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Hui Meng Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Xi’an Jiaotong University Health Science Center, Xi’an, Shaanxi, China

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Min Xu Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Xi’an Jiaotong University Health Science Center, Xi’an, Shaanxi, China

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Xin Gao Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Xi’an Jiaotong University Health Science Center, Xi’an, Shaanxi, China

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Xulei Sun Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Xi’an Jiaotong University Health Science Center, Xi’an, Shaanxi, China

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Xinxin Jin Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Xi’an Jiaotong University Health Science Center, Xi’an, Shaanxi, China

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Hongzhi Sun Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Xi’an Jiaotong University Health Science Center, Xi’an, Shaanxi, China

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osteoblast differentiation. Our results revealed that BMAL1 KD suppressed osteoblast differentiation and promoted inflammatory gene expression in vitro through glycogen synthase kinase-3 beta (GSK3β)/β-catenin pathway dependent on the mTOR activation. Our

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-22-0181
Volume/Issue:
Volume 70: Issue 4
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Kathryn L Auld
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Stephen P Berasi Pfizer Global Biotherapeutics Technologies, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer Oncology Research and Development, Former Wyeth Colleagues, Cambridge, Massachusetts, USA

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Yan Liu Pfizer Global Biotherapeutics Technologies, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer Oncology Research and Development, Former Wyeth Colleagues, Cambridge, Massachusetts, USA

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Michael Cain Pfizer Global Biotherapeutics Technologies, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer Oncology Research and Development, Former Wyeth Colleagues, Cambridge, Massachusetts, USA

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Ying Zhang Pfizer Global Biotherapeutics Technologies, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer Oncology Research and Development, Former Wyeth Colleagues, Cambridge, Massachusetts, USA

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Christine Huard Pfizer Global Biotherapeutics Technologies, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer Oncology Research and Development, Former Wyeth Colleagues, Cambridge, Massachusetts, USA

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Shoichi Fukayama Pfizer Global Biotherapeutics Technologies, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer Oncology Research and Development, Former Wyeth Colleagues, Cambridge, Massachusetts, USA

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Jing Zhang
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Sung Choe
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Wenyan Zhong Pfizer Global Biotherapeutics Technologies, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer Oncology Research and Development, Former Wyeth Colleagues, Cambridge, Massachusetts, USA

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Bheem M Bhat Pfizer Global Biotherapeutics Technologies, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer Oncology Research and Development, Former Wyeth Colleagues, Cambridge, Massachusetts, USA

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Ramesh A Bhat Pfizer Global Biotherapeutics Technologies, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer Oncology Research and Development, Former Wyeth Colleagues, Cambridge, Massachusetts, USA

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Eugene L Brown Pfizer Global Biotherapeutics Technologies, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer Oncology Research and Development, Former Wyeth Colleagues, Cambridge, Massachusetts, USA

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Robert V Martinez Pfizer Global Biotherapeutics Technologies, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer BioTherapeutics Research and Development, Pfizer Oncology Research and Development, Former Wyeth Colleagues, Cambridge, Massachusetts, USA

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ERRα acts as an inhibitor of osteoblast differentiation in vitro ( Teyssier et al . 2009 ). The Wnt pathway is another important player in bone development (reviewed in Bodine & Komm (2006) ). During canonical Wnt/β-catenin signaling, Wnt ligands

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-11-0140
Volume/Issue:
Volume 48: Issue 2
Free access
Chen Chen Key Laboratory of Swine Genetics and Breeding of Agricultural Ministry and Key Laboratory of Agricultural Animal Genetics, Hunan Institute of Animal and Veterinary Science, Department of Animal Nutrition and Feed Science, Breeding and Reproduction of Ministry of Education, College of Animal Science and Technology, Huazhong Agricultural University, Wuhan 430070, People's Republic of China
Key Laboratory of Swine Genetics and Breeding of Agricultural Ministry and Key Laboratory of Agricultural Animal Genetics, Hunan Institute of Animal and Veterinary Science, Department of Animal Nutrition and Feed Science, Breeding and Reproduction of Ministry of Education, College of Animal Science and Technology, Huazhong Agricultural University, Wuhan 430070, People's Republic of China

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Yongdong Peng Key Laboratory of Swine Genetics and Breeding of Agricultural Ministry and Key Laboratory of Agricultural Animal Genetics, Hunan Institute of Animal and Veterinary Science, Department of Animal Nutrition and Feed Science, Breeding and Reproduction of Ministry of Education, College of Animal Science and Technology, Huazhong Agricultural University, Wuhan 430070, People's Republic of China

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Yinglin Peng Key Laboratory of Swine Genetics and Breeding of Agricultural Ministry and Key Laboratory of Agricultural Animal Genetics, Hunan Institute of Animal and Veterinary Science, Department of Animal Nutrition and Feed Science, Breeding and Reproduction of Ministry of Education, College of Animal Science and Technology, Huazhong Agricultural University, Wuhan 430070, People's Republic of China

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Jian Peng Key Laboratory of Swine Genetics and Breeding of Agricultural Ministry and Key Laboratory of Agricultural Animal Genetics, Hunan Institute of Animal and Veterinary Science, Department of Animal Nutrition and Feed Science, Breeding and Reproduction of Ministry of Education, College of Animal Science and Technology, Huazhong Agricultural University, Wuhan 430070, People's Republic of China

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Siwen Jiang Key Laboratory of Swine Genetics and Breeding of Agricultural Ministry and Key Laboratory of Agricultural Animal Genetics, Hunan Institute of Animal and Veterinary Science, Department of Animal Nutrition and Feed Science, Breeding and Reproduction of Ministry of Education, College of Animal Science and Technology, Huazhong Agricultural University, Wuhan 430070, People's Republic of China

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family impair preadipocyte differentiation ( Karbiener et al . 2009 , Sun et al . 2009 , Kim et al . 2010 ). In addition, miR-210 has been reported to promote adipogenesis by inhibiting the canonical Wnt/β-catenin signaling pathway ( Qin et al

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-14-0013
Volume/Issue:
Volume 52: Issue 3
Free access
Yan-ping Xu Department of Neonatology, Department of Neonatology, Department of Pediatric Neurology, Central Laboratory, Department of Endocrinology, The Children's Hospital, Zhejiang University School of Medicine and Zhejiang Key Laboratory for Diagnosis and Therapy of Neonatal Diseases, Hangzhou 310003, China

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Jia-jun Zhu Department of Neonatology, Department of Neonatology, Department of Pediatric Neurology, Central Laboratory, Department of Endocrinology, The Children's Hospital, Zhejiang University School of Medicine and Zhejiang Key Laboratory for Diagnosis and Therapy of Neonatal Diseases, Hangzhou 310003, China
Department of Neonatology, Department of Neonatology, Department of Pediatric Neurology, Central Laboratory, Department of Endocrinology, The Children's Hospital, Zhejiang University School of Medicine and Zhejiang Key Laboratory for Diagnosis and Therapy of Neonatal Diseases, Hangzhou 310003, China

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Fen Cheng Department of Neonatology, Department of Neonatology, Department of Pediatric Neurology, Central Laboratory, Department of Endocrinology, The Children's Hospital, Zhejiang University School of Medicine and Zhejiang Key Laboratory for Diagnosis and Therapy of Neonatal Diseases, Hangzhou 310003, China

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Ke-wen Jiang Department of Neonatology, Department of Neonatology, Department of Pediatric Neurology, Central Laboratory, Department of Endocrinology, The Children's Hospital, Zhejiang University School of Medicine and Zhejiang Key Laboratory for Diagnosis and Therapy of Neonatal Diseases, Hangzhou 310003, China

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Wei-zhong Gu Department of Neonatology, Department of Neonatology, Department of Pediatric Neurology, Central Laboratory, Department of Endocrinology, The Children's Hospital, Zhejiang University School of Medicine and Zhejiang Key Laboratory for Diagnosis and Therapy of Neonatal Diseases, Hangzhou 310003, China

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Zheng Shen Department of Neonatology, Department of Neonatology, Department of Pediatric Neurology, Central Laboratory, Department of Endocrinology, The Children's Hospital, Zhejiang University School of Medicine and Zhejiang Key Laboratory for Diagnosis and Therapy of Neonatal Diseases, Hangzhou 310003, China

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Yi-dong Wu Department of Neonatology, Department of Neonatology, Department of Pediatric Neurology, Central Laboratory, Department of Endocrinology, The Children's Hospital, Zhejiang University School of Medicine and Zhejiang Key Laboratory for Diagnosis and Therapy of Neonatal Diseases, Hangzhou 310003, China

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Li Liang Department of Neonatology, Department of Neonatology, Department of Pediatric Neurology, Central Laboratory, Department of Endocrinology, The Children's Hospital, Zhejiang University School of Medicine and Zhejiang Key Laboratory for Diagnosis and Therapy of Neonatal Diseases, Hangzhou 310003, China

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Li-zhong Du Department of Neonatology, Department of Neonatology, Department of Pediatric Neurology, Central Laboratory, Department of Endocrinology, The Children's Hospital, Zhejiang University School of Medicine and Zhejiang Key Laboratory for Diagnosis and Therapy of Neonatal Diseases, Hangzhou 310003, China

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concentrated on the roles for Wnt/β-catenin in respiratory system development, including segregation of the conducting airway and alveolar compartments, specialization of the mesenchyme, and establishment of tracheal asymmetries and tracheal glands ( De Langhe

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-10-0143
Volume/Issue:
Volume 47: Issue 1
Free access
Michael A Gentile
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Pascale V Nantermet
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Robert L Vogel
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Robert Phillips Merck Research Laboratories, Department of Molecular Profiling, Merck Research Laboratories, Department of Molecular Endocrinology, West Point, Pennsylvania 19486, USA

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Daniel Holder Merck Research Laboratories, Department of Molecular Profiling, Merck Research Laboratories, Department of Molecular Endocrinology, West Point, Pennsylvania 19486, USA

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Paul Hodor Merck Research Laboratories, Department of Molecular Profiling, Merck Research Laboratories, Department of Molecular Endocrinology, West Point, Pennsylvania 19486, USA

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Chun Cheng Merck Research Laboratories, Department of Molecular Profiling, Merck Research Laboratories, Department of Molecular Endocrinology, West Point, Pennsylvania 19486, USA

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Hongyue Dai Merck Research Laboratories, Department of Molecular Profiling, Merck Research Laboratories, Department of Molecular Endocrinology, West Point, Pennsylvania 19486, USA

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Leonard P Freedman
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William J Ray
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myogenic lineage while inhibiting adipogenic differentiation by modulating nuclear translocation of β-catenin ( Bhasin et al . 2006 ). β-Catenin, through its action in cell adhesion and Wnt signal transduction, plays a critical role in embryonic

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JME-09-0048
Volume/Issue:
Volume 44: Issue 1
Free access
Yingying Zhu Wuya College of Innovation, Shenyang Pharmaceutical University, Shenyang, Liaoning, China

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Weiwei Liu Wuya College of Innovation, Shenyang Pharmaceutical University, Shenyang, Liaoning, China

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Shuaigao Chen Wuya College of Innovation, Shenyang Pharmaceutical University, Shenyang, Liaoning, China

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Fanxing Xu Wuya College of Innovation, Shenyang Pharmaceutical University, Shenyang, Liaoning, China

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Luxin Zhang Wuya College of Innovation, Shenyang Pharmaceutical University, Shenyang, Liaoning, China

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Toshihiko Hayashi Wuya College of Innovation, Shenyang Pharmaceutical University, Shenyang, Liaoning, China
Department of Chemistry and Life Science, School of Advanced Engineering Kogakuin University, Tokyo, Japan
Nippi Research Institute of Biomatrix, Toride, Ibaraki, Japan

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Kazunori Mizuno Nippi Research Institute of Biomatrix, Toride, Ibaraki, Japan

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Shunji Hattori Nippi Research Institute of Biomatrix, Toride, Ibaraki, Japan

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Hitomi Fujisaki Nippi Research Institute of Biomatrix, Toride, Ibaraki, Japan

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Takashi Ikejima Wuya College of Innovation, Shenyang Pharmaceutical University, Shenyang, Liaoning, China
Key Laboratory of Computational Chemistry-Based Natural Antitumor Drug Research and Development, Liaoning, China

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preadipocytes through the inhibition of autophagy ( Liu et al. 2019 ). Thus, the involvement of the IGF-1R-autophagy signaling pathway in collagen I-coated pancreatic INS-1 cells is discussed. β-Catenin, a transcription factor, is known as a crucial

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-21-0032
Volume/Issue:
Volume 67: Issue 3
Restricted access
Rui Wang Shanghai Clinical Center for Endocrine and Metabolic Diseases, Laboratory for Endocrine and Metabolism, Shanghai Institute of Endocrine and Metabolic Diseases, Department of Endocrinology and Metabolism, Shanghai Key Laboratory for Endocrine Tumors and E-Institutes of Shanghai Universities, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, 197 Ruijin 2nd Road, Shanghai 200025, China

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Jie Hong Shanghai Clinical Center for Endocrine and Metabolic Diseases, Laboratory for Endocrine and Metabolism, Shanghai Institute of Endocrine and Metabolic Diseases, Department of Endocrinology and Metabolism, Shanghai Key Laboratory for Endocrine Tumors and E-Institutes of Shanghai Universities, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, 197 Ruijin 2nd Road, Shanghai 200025, China

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Ruixin Liu Shanghai Clinical Center for Endocrine and Metabolic Diseases, Laboratory for Endocrine and Metabolism, Shanghai Institute of Endocrine and Metabolic Diseases, Department of Endocrinology and Metabolism, Shanghai Key Laboratory for Endocrine Tumors and E-Institutes of Shanghai Universities, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, 197 Ruijin 2nd Road, Shanghai 200025, China

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Maopei Chen Shanghai Clinical Center for Endocrine and Metabolic Diseases, Laboratory for Endocrine and Metabolism, Shanghai Institute of Endocrine and Metabolic Diseases, Department of Endocrinology and Metabolism, Shanghai Key Laboratory for Endocrine Tumors and E-Institutes of Shanghai Universities, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, 197 Ruijin 2nd Road, Shanghai 200025, China

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Min Xu Shanghai Clinical Center for Endocrine and Metabolic Diseases, Laboratory for Endocrine and Metabolism, Shanghai Institute of Endocrine and Metabolic Diseases, Department of Endocrinology and Metabolism, Shanghai Key Laboratory for Endocrine Tumors and E-Institutes of Shanghai Universities, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, 197 Ruijin 2nd Road, Shanghai 200025, China

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Wiqiong Gu Shanghai Clinical Center for Endocrine and Metabolic Diseases, Laboratory for Endocrine and Metabolism, Shanghai Institute of Endocrine and Metabolic Diseases, Department of Endocrinology and Metabolism, Shanghai Key Laboratory for Endocrine Tumors and E-Institutes of Shanghai Universities, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, 197 Ruijin 2nd Road, Shanghai 200025, China

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Yifei Zhang Shanghai Clinical Center for Endocrine and Metabolic Diseases, Laboratory for Endocrine and Metabolism, Shanghai Institute of Endocrine and Metabolic Diseases, Department of Endocrinology and Metabolism, Shanghai Key Laboratory for Endocrine Tumors and E-Institutes of Shanghai Universities, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, 197 Ruijin 2nd Road, Shanghai 200025, China

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Qinyun Ma Shanghai Clinical Center for Endocrine and Metabolic Diseases, Laboratory for Endocrine and Metabolism, Shanghai Institute of Endocrine and Metabolic Diseases, Department of Endocrinology and Metabolism, Shanghai Key Laboratory for Endocrine Tumors and E-Institutes of Shanghai Universities, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, 197 Ruijin 2nd Road, Shanghai 200025, China

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Feng Wang Shanghai Clinical Center for Endocrine and Metabolic Diseases, Laboratory for Endocrine and Metabolism, Shanghai Institute of Endocrine and Metabolic Diseases, Department of Endocrinology and Metabolism, Shanghai Key Laboratory for Endocrine Tumors and E-Institutes of Shanghai Universities, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, 197 Ruijin 2nd Road, Shanghai 200025, China

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Juan Shi Shanghai Clinical Center for Endocrine and Metabolic Diseases, Laboratory for Endocrine and Metabolism, Shanghai Institute of Endocrine and Metabolic Diseases, Department of Endocrinology and Metabolism, Shanghai Key Laboratory for Endocrine Tumors and E-Institutes of Shanghai Universities, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, 197 Ruijin 2nd Road, Shanghai 200025, China

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Jiqiu Wang Shanghai Clinical Center for Endocrine and Metabolic Diseases, Laboratory for Endocrine and Metabolism, Shanghai Institute of Endocrine and Metabolic Diseases, Department of Endocrinology and Metabolism, Shanghai Key Laboratory for Endocrine Tumors and E-Institutes of Shanghai Universities, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, 197 Ruijin 2nd Road, Shanghai 200025, China

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Weiqing Wang Shanghai Clinical Center for Endocrine and Metabolic Diseases, Laboratory for Endocrine and Metabolism, Shanghai Institute of Endocrine and Metabolic Diseases, Department of Endocrinology and Metabolism, Shanghai Key Laboratory for Endocrine Tumors and E-Institutes of Shanghai Universities, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, 197 Ruijin 2nd Road, Shanghai 200025, China

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Guang Ning Shanghai Clinical Center for Endocrine and Metabolic Diseases, Laboratory for Endocrine and Metabolism, Shanghai Institute of Endocrine and Metabolic Diseases, Department of Endocrinology and Metabolism, Shanghai Key Laboratory for Endocrine Tumors and E-Institutes of Shanghai Universities, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, 197 Ruijin 2nd Road, Shanghai 200025, China
Shanghai Clinical Center for Endocrine and Metabolic Diseases, Laboratory for Endocrine and Metabolism, Shanghai Institute of Endocrine and Metabolic Diseases, Department of Endocrinology and Metabolism, Shanghai Key Laboratory for Endocrine Tumors and E-Institutes of Shanghai Universities, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, 197 Ruijin 2nd Road, Shanghai 200025, China

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that evoked WNT/β-catenin signalling in white and brown preadipocytes leads to the maintenance of the undifferentiated state and prevents the induction of CCAAT/enhancer-binding protein α (Cebpa) and peroxisome proliferator-activated receptor γ (Pparg

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-14-0037
Volume/Issue:
Volume 53: Issue 3
Free access