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Leping Zhao, Yong Pan, Kesong Peng, Zhe Wang, Jieli Li, Dan Li, Chao Tong, Yi Wang and Guang Liang

drops obtained by clipping the tail of the mice using a ONE TOUCH BASIC plus Glucose Monitor (Lifescan, Milpitas, CA, USA). The blood glucose levels of these animals were recorded every week. An oral glucose tolerance test (OGTT) was carried out in mice

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Hisashi Masuyama and Yuji Hiramatsu

al . 2009 ). Therefore, we examined glucose tolerance test (GTT) and insulin tolerance test (ITT) and measured the expression of gluconeogenic and lipogenic genes in the livers of oophorectomized ICR mice treated with E 2 and progesterone in the

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Louise K Metcalfe, Greg C Smith and Nigel Turner

, particularly in studies demonstrating the beneficial effects of their deficiency. The genetic ablation of sphingomyelin synthase 2 ( Sms2 ), for instance, has been found in HFD-fed rodents to improve whole-body insulin sensitivity, glucose tolerance and weight

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Loes P M Duivenvoorde, Evert M van Schothorst, Annelies Bunschoten and Jaap Keijer

restricted (30%) or ad libitum (HF-AL) basis. We show that, although an animal is fed a HF (western) diet, the moderate reduction in the total amount of calories improves metabolic health, as measured by a number of parameters, such as glucose tolerance and

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C Folgueira, S Barja-Fernandez, P Gonzalez-Saenz, V Pena-Leon, C Castelao, M Ruiz-Piñon, F F Casanueva, R Nogueiras and L M Seoane

2C-knockout mice had unaltered body weight, adiposity and glucose tolerance ( Begg et al . 2014 ). Therefore, the physiological significance of the UGN-GUCY2C system for the control of energy homeostasis is not fully understood

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Lynley D Pound, Suparna A Sarkar, Stéphane Cauchi, Yingda Wang, James K Oeser, Catherine E Lee, Philippe Froguel, John C Hutton and Richard M O'Brien

Slc30a8 expression can be achieved, have also failed to demonstrate a critical role for ZnT-8 in the control of glucose metabolism. Thus, mice in which the Slc30a8 gene was globally deleted have normal glucose tolerance ( Lemaire et al . 2009

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Maria Sörhede Winzell and Bo Ahrén

. Journal of Biological Chemistry 272 21201 – 21206 . Pacini G Thomaseth K Ahrén B 2001 Contribution to glucose tolerance of insulin-independent vs. insulin-dependent mechanisms in mice . American Journal of Physiology Endocrinology and Metabolism 281

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Ana Luiza R Rolim, Susan C Lindsey, Ilda S Kunii, Felipe Crispim, Regina Célia M S Moisés, Rui M B Maciel and Magnus R Dias-da-Silva

a glucose tolerance test and a higher platelet 3Na + /2K + ATPase activity in TPP patients compared with healthy control subjects and thyrotoxicosis patients without paralysis ( Chan et al . 1994 ). Recently, we have proposed a feedforward model

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C Attané, D Daviaud, C Dray, R Dusaulcy, M Masseboeuf, D Prévot, C Carpéné, I Castan-Laurell and P Valet

subjects ( Boucher et al . 2005 , Castan-Laurell et al . 2008 ). Plasma apelin levels are also raised in morbidly obese ( Heinonen et al . 2005 ) and type 2 diabetic ( Soriguer et al . 2009 ) subjects. Nonobese patients with impaired glucose tolerance

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Ping Li, Yan Hao, Feng-Hui Pan, Min Zhang, Jian-Qiang Ma and Da-Long Zhu

, USA). Systolic blood pressure was measured in conscious animals using a tail-cuff blood pressure analyzer (BP-98A; Softron Corporation, Japan) every 2 weeks. To estimate glucose tolerance, at 17 weeks of age, mice were fasted for 16 h, and a basal