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Patrizia Morera, Loredana Basiricò, Kenji Hosoda and Umberto Bernabucci

rectal temperature (RT) were recorded daily. On 0 and 5 days, six animals from each group were used to perform a glucose tolerance test (GTT). At the beginning (day 0) and after 2 and 5 days of the experimental period, six animals from each group were

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Laure Nivlet, Joel Herrmann, Delia Esteban Martin, Aline Meunier, Christophe Orvain and Gérard Gradwohl

fast for 16 h. For Oral Glucose Tolerance Test (OGTT), mice received glucose by intragastric gavage (1 g/kg body weight). For Intraperitoneal Glucose Tolerance Test (IPGTT), mice received glucose by i.p. injection (2 g/kg body weight). Circulating blood

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Yuka Toyoshima, Reiko Tokita, Yoichiro Ohne, Fumihiko Hakuno, Tadashi Noguchi, Shiro Minami, Hisanori Kato and Shin-Ichiro Takahashi

. Values for glucose tolerance test (GTT), PTT, and gene expression levels are given as means± s.e.m. for five rats. When we compared the two groups, the results were analyzed statistically using unpaired Student's t -test. Where multiple comparisons were

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Lucia Kořínková, Martina Holubová, Barbora Neprašová, Lucie Hrubá, Veronika Pražienková, Michal Bencze, Martin Haluzík, Jaroslav Kuneš, Lenka Maletínská and Blanka Železná

glucose tolerance test (OGTT) was measured in Experiment 2 ( Fig. 1 ): 6-h-fasted mice were administered a glucose solution at a dose of 2 g/kg BW by gavage. Blood samples were obtained from the tail vessels. The blood glucose concentrations were measured

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Juliane Dinter, Jessica Mühlhaus, Simon Friedrich Jacobi, Carolin Leonie Wienchol, Maxi Cöster, Jaroslawna Meister, Carolin Stephanie Hoefig, Anne Müller, Josef Köhrle, Annette Grüters, Heiko Krude, Jens Mittag, Torsten Schöneberg, Gunnar Kleinau and Heike Biebermann

:2500). Color reaction was performed as described previously ( Schoneberg et al . 1996 ), and absorption was measured at 492/620 nm using an Anthos Reader 2001. i.p. glucose tolerance test and liver glycogen determination C57BL/6J male mice that were 3–4 months

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C Ruth González, María J Vázquez, Miguel López and Carlos Diéguez

et al . 2006 ). Moreover, ablation of the ghrelin gene improves glucose tolerance, insulin secretion, and insulin sensitivity in genetically leptin-deficient ( ob/ob ) obese mice ( Sun et al . 2006 ). Administration of exogenous ghrelin suppresses

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Lingyun Zhang, Takashi Sugiyama, Nao Murabayashi, Takashi Umekawa, Ning Ma, Yuki Kamimoto, Yoshihiro Ogawa and Norimasa Sagawa

% paraformaldehyde (Nakarai Tesque, Kyoto, Japan), and the other tissues were frozen in liquid nitrogen and stored at −80 °C prior to RNA extraction. Metabolic testing For glucose tolerance test (GTT), the mice were deprived of food for 14–16 h overnight and then

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Shinobu Shimizu, Tetsuya Hosooka, Tomokazu Matsuda, Shun-ichiro Asahara, Maki Koyanagi-Kimura, Ayumi Kanno, Alberto Bartolome, Hiroaki Etoh, Megumi Fuchita, Kyoko Teruyama, Hiroaki Takahashi, Hiroyuki Inoue, Yusuke Mieda, Naoko Hashimoto, Susumu Seino and Yoshiaki Kido

ELISA kit (Shibayagi, Gunma, Japan). This study was performed according to the guidelines of the Animal Ethics Committee of Kobe University Graduate School of Medicine. Oral glucose tolerance tests Mice were deprived of food for 16 h before the oral

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Elin Swedenborg, Joëlle Rüegg, Sari Mäkelä and Ingemar Pongratz

syndrome or syndrome X is a term for a combination of disorders that may include impaired glucose tolerance or insulin resistance, dyslipidemia, high blood pressure, and obesity (reviewed in Despres et al . (2008) and Phillips & Prins (2008) ). Key

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Jiazhong Sun, Yancheng Xu, Haohua Deng, Suxin Sun, Zhe Dai and Yanlei Sun

spikes in the pathogenesis of diabetic complications . Diabetic Medicine 15 188 – 193 . Ceriello A 2004 Impaired glucose tolerance and cardiovascular disease: the possible role of post-prandial hyperglycemia . American Heart Journal 147