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K Lindberg, S G Rønn, D Tornehave, H Richter, J A Hansen, J Rømer, M Jackerott and N Billestrup

from free insulin ( Bonnevie-Nielsen 1980 ). Plasma insulin Plasma insulin concentrations were determined using a rat insulin RIA kit (Linco Research, St Charles, MO, USA). Oral glucose-tolerance

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Yueting Dong, Zhiye Xu, Ziyi Zhang, Xueyao Yin, Xihua Lin, Hong Li and Fenping Zheng

the same vehicle treatment by i.p. injection. Body weight was recorded once a week, and food intake was monitored every day. In vivo glucose homeostasis assays After 3 weeks of treatment, intraperitoneal glucose tolerance tests (IPGTT) and

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Guojun Shi, Chen Sun, Weiqiong Gu, Minglan Yang, Xiaofang Zhang, Nan Zhai, Yan Lu, Zhijian Zhang, Peishun Shou, Zhiguo Zhang and Guang Ning

and lymphocytes, which are critical components of the immune system in both defense and autoimmune diseases. Our results indicate that the activation of FFAR2 improves glucose tolerance by inducing apoptosis of infiltrated immune cells. The

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Shaoqian Zhao, Wen Liu, Jiqiu Wang, Juan Shi, Yingkai Sun, Weiqing Wang, Guang Ning, Ruixin Liu and Jie Hong

improved adiposity, glucose tolerance and low-grade inflammation in chow diet-fed mice by A. muciniphila . Hence, our research provided new evidence for the metabolic benefits of A. muciniphila on chow diet-fed mice, pointing out the possible mechanisms

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Ting-Ting Zhou, Fei Ma, Xiao-Fan Shi, Xin Xu, Te Du, Xiao-Dan Guo, Gai-Hong Wang, Liang Yu, Vatcharin Rukachaisirikul, Li-Hong Hu, Jing Chen and Xu Shen

demonstrated that DMT administration efficiently decreased fasting blood glucose and hemoglobin A1c (HbA1c) levels and improved glucose tolerance and pyruvate tolerance tests. To our knowledge, DMT might be the first reported small molecule as a Gαq signaling

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Ahter D Sanlioglu, Bahri Karacay, Mustafa Kemal Balci, Thomas S Griffith and Salih Sanlioglu

Obesity and inflammation Diabetes is the third most common disease and fourth leading cause of death in the world ( Tabak et al . 2012 ). Insulin resistance, impaired glucose tolerance, and excessive glucagon secretion are the prominent features of

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Hanze Du, Limei Liu, Ying Wang, Yuichi Nakagawa, Alexei Lyzlov, Kabirullah Lutfy, Theodore C Friedman, Xiaozhong Peng and Yanjun Liu

al . 2011 ). By contrast, 11B-Hsd1 knockout mice are resilient to diet-induced insulin resistance ( Kotelevtsev et al . 1997 ), although a liver-specific 11β-HSD1 knockout mouse showed mild improvement in glucose tolerance without affecting insulin

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Bo Zhou, Huixia Li, Jiali Liu, Lin Xu, Qinyue Guo, Hongzhi Sun and Shufang Wu

( Zhou et al . 2013 a , b ). Measurement of blood parameters Peripheral serum was subject to ELISA using standard kits (R&D Systems, Inc., Minneapolis, MN, USA) for progranulin. Morning blood glucose and insulin levels were measured and glucose tolerance

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Hisashi Masuyama and Yuji Hiramatsu

al . 2009 ). Therefore, we examined glucose tolerance test (GTT) and insulin tolerance test (ITT) and measured the expression of gluconeogenic and lipogenic genes in the livers of oophorectomized ICR mice treated with E 2 and progesterone in the

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Gilberto Paz-Filho, Claudio Alberto Mastronardi, Brian J Parker, Ainy Khan, Antonio Inserra, Klaus I Matthaei, Monika Ehrhart-Bornstein, Stefan Bornstein, Ma-Li Wong and Julio Licinio

/metabolite measurements Circulating leptin levels were determined at the end point using ELISA kits in duplicate (R&D systems, Minneapolis, MN, USA). Insulin levels were determined using an ultrasensitive ELISA (Alpco, Salem, NH, USA). Intraperitoneal glucose tolerance