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Yanxia Tang and GuoDong Li

increased plasma levels of free fatty acids (FFAs) resulting from lack of appropriate suppression of adipocyte lipolysis due to insulin resistance and deficiency ( Reaven 1988 , Imrie et al . 2010 ). Furthermore, circulating levels of FFAs are usually

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Edwin J W Geven, Folkert Verkaar, Gert Flik, and Peter H M Klaren

, Schreck 2000 ). Cortisol stimulates gluconeogenesis and lipolysis ( Sheridan 1988 , van der Boon et al. 1991 ), which result in increased plasma glucose and free fatty acid levels. These catabolic actions of cortisol reflect its glucocorticoid potency

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Kazuyoshi Ukena, Tomohiro Osugi, Jérôme Leprince, Hubert Vaudry, and Kazuyoshi Tsutsui

-induced lipolysis ( Malumba et al . 2010 ). As 3T3-L1 cells express the QRFPR-encoding gene, it appears that 26RFa/QRFP may act in an autocrine/paracrine manner to regulate adipogenesis ( Malumba et al . 2010 ). According to Alonzeau et al . (2013) , 26RFa

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Qi Zhang, Qin Zhu, Ruyuan Deng, Feiye Zhou, Linlin Zhang, Shushu Wang, Kecheng Zhu, Xiao Wang, Libin Zhou, and Qing Su

reticulum-tethered transcription factor cAMP responsive element-binding protein, hepatocyte specific, regulates hepatic lipogenesis, fatty acid oxidation, and lipolysis upon metabolic stress in mice . Hepatology 55 1070 – 1082 . ( https://doi.org/10

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Carolina Gustavsson, Tomoyoshi Soga, Erik Wahlström, Mattias Vesterlund, Alireza Azimi, Gunnar Norstedt, and Petra Tollet-Egnell

. 2005 ) and antioxidant ( Louet et al . 2004 , Baba et al . 2005 , Borrás et al . 2005 ) properties. Oestrogens have also been shown to inhibit lipogenesis and stimulate lipolysis in abdominal visceral fat depots, to promote use of lipid as a fuel

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Subhamoy Dasgupta and Bert W O'Malley

protected from high-fat diet-induced obesity and exhibit increased insulin sensitivity, higher lipolysis, and reduced fat uptake ( Picard et al . 2002 ). Loss of Src-2 −/− also affects the hepatic glucose release due to decreased expression of glucose-6

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J H Duncan Bassett and Graham R Williams

hormone receptors . Bone 36 607 – 616 . Liu YY Schultz JJ Brent GA 2003 A thyroid hormone receptor alpha gene mutation (P398H) is associated with visceral adiposity and impaired catecholamine-stimulated lipolysis in mice . Journal of

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Sofia A Rahman, Azizun Nessa, and Khalid Hussain

inappropriate release leads to persistent hyperinsulinaemic hypoglycaemia (HH). This drives glucose into the insulin sensitive tissues and simultaneously suppresses lipolysis and ketogenesis. The combined hypoketonaemic and hypoglycaemic characteristic of CHI

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Pierre Poinsot, Martin Schwarzer, Noël Peretti, and François Leulier

actions or lipolysis ( Bergan-Roller & Sheridan 2017 ). Anabolic actions of GH in juvenile growth act by well-known mechanism associated with positive nitrogen balance, protein synthesis in muscle and stimulation of the longitudinal bone growth ( Chikani

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Jin-Seung Choung, Young-Sun Lee, and Hee-Sook Jun

lipolysis and free fatty acid utilization, contributing to the activation of UCP-1. β 3 -adrenergic receptors are thought to play a role in thermogenesis in brown fat and skeletal muscle, induce adaptive non-shivering thermogenesis and stimulate UCP1