Search Results

You are looking at 81 - 90 of 117 items for :

  • All content x
Clear All
Free access

Yanjun Liu, Yuichi Nakagawa, Ying Wang, Limei Liu, Hongwei Du, Wei Wang, Xiuhai Ren, Kabirullah Lutfy, and Theodore C Friedman

adipose tissue and decreases lipolysis . Journal of Clinical Endocrinology and Metabolism 92 857 – 864 . Walker BR Connacher AA Lindsay RM Webb DJ Edwards CR 1995 Carbenoxolone increases hepatic insulin sensitivity in man: a novel role for 11

Free access

Changxue Lu and Sheue-Yann Cheng

H-mutated TRα1 manifest visceral obesity, hyperleptinemia, reduced catecholamine-stimulated lipolysis in WAT, and hepatic steatosis ( Liu et al . 2007 ). In the absence of T 3 , wild-type TRα1 and P398H mutant significantly reduce PPARα

Free access

Bo Zhou, Huixia Li, Jiali Liu, Lin Xu, Qinyue Guo, Hongzhi Sun, and Shufang Wu

476 – 491 . ( doi:10.1016/j.cell.2007.08.047 ). Stralfors P Honnor RC 1989 Insulin-induced dephosphorylation of hormone-sensitive lipase: correlation with lipolysis and cAMP-dependent protein kinase activity . European Journal of Biochemistry

Free access

Ana Luísa Neves, João Coelho, Luciana Couto, Adelino Leite-Moreira, and Roberto Roncon-Albuquerque Jr

of triglyceride, hepatic de novo fatty acid synthesis, and lipolysis. High doses of LPS produce hypertriglyceridemia by decreasing lipoprotein catabolism. Administration of TNF antibodies or IL1 receptor antagonist did not prevent the increase in

Free access

Eva Tudurí, Monica Imbernon, Rene Javier Hernández-Bautista, Marta Tojo, Johan Fernø, Carlos Diéguez, and Rubén Nogueiras

-Hyiaman et al . 2008 ). In addition, activation of CB1 increases lipogenesis and inflammation and reduces lipolysis, in the white adipose tissue (WAT) ( Silvestri & Di Marzo 2013 ). Furthermore, CB1 activation impairs insulin sensitivity, as evidenced by

Free access

Subhamoy Dasgupta and Bert W O'Malley

protected from high-fat diet-induced obesity and exhibit increased insulin sensitivity, higher lipolysis, and reduced fat uptake ( Picard et al . 2002 ). Loss of Src-2 −/− also affects the hepatic glucose release due to decreased expression of glucose-6

Free access

Carolina Gustavsson, Tomoyoshi Soga, Erik Wahlström, Mattias Vesterlund, Alireza Azimi, Gunnar Norstedt, and Petra Tollet-Egnell

. 2005 ) and antioxidant ( Louet et al . 2004 , Baba et al . 2005 , Borrás et al . 2005 ) properties. Oestrogens have also been shown to inhibit lipogenesis and stimulate lipolysis in abdominal visceral fat depots, to promote use of lipid as a fuel

Free access

Sofia A Rahman, Azizun Nessa, and Khalid Hussain

inappropriate release leads to persistent hyperinsulinaemic hypoglycaemia (HH). This drives glucose into the insulin sensitive tissues and simultaneously suppresses lipolysis and ketogenesis. The combined hypoketonaemic and hypoglycaemic characteristic of CHI

Free access

J H Duncan Bassett and Graham R Williams

hormone receptors . Bone 36 607 – 616 . Liu YY Schultz JJ Brent GA 2003 A thyroid hormone receptor alpha gene mutation (P398H) is associated with visceral adiposity and impaired catecholamine-stimulated lipolysis in mice . Journal of

Free access

Afreen Idris Shariff, Sohail Syed, Rebecca A Shelby, Jeremy Force, Jeffrey Melson Clarke, David D’Alessio, and Leonor Corsino

adipose tissue, CST increases the breakdown of lipids by inducing lipolysis and raising local accumulation of non-sterified fatty acids, a mechanism that has been associated with greater increasing insulin resistance ( Perez et al. 2014 ). Apart from