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Michela Zamboni Institute of Biochemistry and Cell Biology, National Council of Research, Monterotondo, Rome, Italy

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Georgios Strimpakos Institute of Biochemistry and Cell Biology, National Council of Research, Monterotondo, Rome, Italy

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Eleonora Poggiogalle Department of Experimental Medicine – Medical Pathophysiology, Food Science and Endocrinology Section, Sapienza University of Rome, Rome, Italy

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Lorenzo M Donini Department of Experimental Medicine – Medical Pathophysiology, Food Science and Endocrinology Section, Sapienza University of Rome, Rome, Italy

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Donato Civitareale Institute of Biochemistry and Cell Biology, National Council of Research, Monterotondo, Rome, Italy

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mechanisms behind this correlation are not fully understood. Since TTF-2 plays a critical role as mediator between extracellular inputs in the maintenance of thyroid-specific gene expression, we hypothesized that it could play a role in adipocyte signaling on

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-22-0129
Volume/Issue:
Volume 70: Issue 1
Free access
I Robertshaw Department of Obstetrics and Gynecology, University of Cincinnati, West Chester, Ohio, USA
Division of Reproductive Sciences, Cincinnati Children’s Hospital Medical Center, Cincinnati, Ohio, USA

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F Bian Division of Reproductive Sciences, Cincinnati Children’s Hospital Medical Center, Cincinnati, Ohio, USA
Perinatal Institute, Cincinnati Children’s Hospital Medical Center, Cincinnati, Ohio, USA

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S K Das Division of Reproductive Sciences, Cincinnati Children’s Hospital Medical Center, Cincinnati, Ohio, USA
Perinatal Institute, Cincinnati Children’s Hospital Medical Center, Cincinnati, Ohio, USA
Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, USA

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of implantation. The mechanisms by which estrogen transforms a progesterone-primed uterus to the receptive state, activates blastocysts, and initiates implantation are not clearly delineated. The classical estrogen signaling pathway is through

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JME-15-0300
Volume/Issue:
Volume 56: Issue 3
Free access
Custodia García-Jiménez
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Jose Manuel García-Martínez
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Ana Chocarro-Calvo
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Antonio De la Vieja Departamento de Fisiología y Bioquímica, Unidad Funcional de Investigación en Enfermedades Crónicas (UFIEC), Facultad de Ciencias de la Salud, Universidad Rey Juan Carlos, 28922 Alcorcon, Madrid, Spain

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exogenous), and subsequent increase in insulin-like growth factor 1 (IGF1) signaling. Altered balances of some adipokines may also contribute to an inflammatory state and increased proliferation; iii) chronic inflammation characterized by increased

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JME-13-0152
Volume/Issue:
Volume 52: Issue 1
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Yefei Pang Marine Science Institute, University of Texas at Austin, Port Aransas, Texas, USA

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Peter Thomas Marine Science Institute, University of Texas at Austin, Port Aransas, Texas, USA

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that multiple mPRs are expressed on the cell membranes of human VSMCs and that both progesterone and 02-0, a selective mPR agonist, but not R5020, a selective PR agonist, rapidly activated MAP kinase and Akt signaling pathways and caused muscle

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-19-0019
Volume/Issue:
Volume 63: Issue 3
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I K Lund Signal Transduction, Novo Nordisk A/S, DK-2880 Bagsværd, Denmark
MedChem Research II, Novo Nordisk A/S, DK-2760 Måløv, Denmark
Steno Diabetes Center, Niels Steensens Vej 6, DK-2820 Gentofte, Denmark

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J A Hansen Signal Transduction, Novo Nordisk A/S, DK-2880 Bagsværd, Denmark
MedChem Research II, Novo Nordisk A/S, DK-2760 Måløv, Denmark
Steno Diabetes Center, Niels Steensens Vej 6, DK-2820 Gentofte, Denmark

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H S Andersen Signal Transduction, Novo Nordisk A/S, DK-2880 Bagsværd, Denmark
MedChem Research II, Novo Nordisk A/S, DK-2760 Måløv, Denmark
Steno Diabetes Center, Niels Steensens Vej 6, DK-2820 Gentofte, Denmark

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N P H Møller Signal Transduction, Novo Nordisk A/S, DK-2880 Bagsværd, Denmark
MedChem Research II, Novo Nordisk A/S, DK-2760 Måløv, Denmark
Steno Diabetes Center, Niels Steensens Vej 6, DK-2820 Gentofte, Denmark

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N Billestrup Signal Transduction, Novo Nordisk A/S, DK-2880 Bagsværd, Denmark
MedChem Research II, Novo Nordisk A/S, DK-2760 Måløv, Denmark
Steno Diabetes Center, Niels Steensens Vej 6, DK-2820 Gentofte, Denmark

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hypothalamus, through which it exerts its key regulatory role by controlling the overall energy homeostasis, i.e. induction of reduced food intake and promotion of weight loss ( Ahima & Flier 2000 , Korner & Aronne 2003 ). Defective leptin signalling due to

Article Type:
Research Article
DOI:
https://doi.org/10.1677/jme.1.01694
Volume/Issue:
Volume 34: Issue 2
Free access
Gunnar Kleinau Institute of Experimental Pediatric Endocrinology (IEPE), Charité-Universitätsmedizin, Berlin, Germany

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Anne Müller Institute of Experimental Pediatric Endocrinology (IEPE), Charité-Universitätsmedizin, Berlin, Germany

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Heike Biebermann Institute of Experimental Pediatric Endocrinology (IEPE), Charité-Universitätsmedizin, Berlin, Germany

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G-protein-coupled receptors The importance of GPCRs Physiological functions such as development, growth, behavior, learning, emotions, senses and aging are strongly dependent on the control of endocrine circuits. Hormonal signals exert

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JME-16-0049
Volume/Issue:
Volume 57: Issue 1
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Dhivya Kumar Departments of Molecular Biology and Biophysics, University of Connecticut Health Center, Farmington, Connecticut, USA

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Richard E Mains Department of Neuroscience, University of Connecticut Health Center, Farmington, Connecticut, USA

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Betty A Eipper Departments of Molecular Biology and Biophysics, University of Connecticut Health Center, Farmington, Connecticut, USA
Department of Neuroscience, University of Connecticut Health Center, Farmington, Connecticut, USA

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, cloning, and structural/mechanistic studies focused on PAM and then expanded to include cell biological studies on secretory granule biogenesis, retrograde signaling from the granule lumen to the nucleus, and the delivery of essential cofactors (ascorbate

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JME-15-0266
Volume/Issue:
Volume 56: Issue 4
Free access
Antara A Banerjee Division of Structural Biology, National Institute for Research in Reproductive Health (Indian Council of Medical Research), National Institute for Research in Reproductive Health (Indian Council of Medical Research), Jehangir Merwanji Street, Parel, Mumbai 400 012, India

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Madhavi Dupakuntla Division of Structural Biology, National Institute for Research in Reproductive Health (Indian Council of Medical Research), National Institute for Research in Reproductive Health (Indian Council of Medical Research), Jehangir Merwanji Street, Parel, Mumbai 400 012, India

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Bhakti R Pathak Division of Structural Biology, National Institute for Research in Reproductive Health (Indian Council of Medical Research), National Institute for Research in Reproductive Health (Indian Council of Medical Research), Jehangir Merwanji Street, Parel, Mumbai 400 012, India

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Smita D Mahale Division of Structural Biology, National Institute for Research in Reproductive Health (Indian Council of Medical Research), National Institute for Research in Reproductive Health (Indian Council of Medical Research), Jehangir Merwanji Street, Parel, Mumbai 400 012, India
Division of Structural Biology, National Institute for Research in Reproductive Health (Indian Council of Medical Research), National Institute for Research in Reproductive Health (Indian Council of Medical Research), Jehangir Merwanji Street, Parel, Mumbai 400 012, India

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downstream signaling pathways, but the exact molecular mechanism remains elusive. The recently solved crystal structure of FSH in complex with the entire ECD of FSHR including the hinge region provides some explanation. FSH interacts with FSHR in a two

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-14-0275
Volume/Issue:
Volume 54: Issue 3
Free access
Pamela Petrocchi-Passeri Department of Medicine of Systems, Institute of Cell Biology and Neurobiology, Department of Integrative Biology and Physiology, Istituto Superiore di Sanità, EBRI Foundation, University of Rome Tor Vergata, Via Montpellier, 100133 Rome, Italy
Department of Medicine of Systems, Institute of Cell Biology and Neurobiology, Department of Integrative Biology and Physiology, Istituto Superiore di Sanità, EBRI Foundation, University of Rome Tor Vergata, Via Montpellier, 100133 Rome, Italy

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Cheryl Cero Department of Medicine of Systems, Institute of Cell Biology and Neurobiology, Department of Integrative Biology and Physiology, Istituto Superiore di Sanità, EBRI Foundation, University of Rome Tor Vergata, Via Montpellier, 100133 Rome, Italy

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Alessandro Cutarelli Department of Medicine of Systems, Institute of Cell Biology and Neurobiology, Department of Integrative Biology and Physiology, Istituto Superiore di Sanità, EBRI Foundation, University of Rome Tor Vergata, Via Montpellier, 100133 Rome, Italy
Department of Medicine of Systems, Institute of Cell Biology and Neurobiology, Department of Integrative Biology and Physiology, Istituto Superiore di Sanità, EBRI Foundation, University of Rome Tor Vergata, Via Montpellier, 100133 Rome, Italy

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Claudio Frank Department of Medicine of Systems, Institute of Cell Biology and Neurobiology, Department of Integrative Biology and Physiology, Istituto Superiore di Sanità, EBRI Foundation, University of Rome Tor Vergata, Via Montpellier, 100133 Rome, Italy

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Cinzia Severini Department of Medicine of Systems, Institute of Cell Biology and Neurobiology, Department of Integrative Biology and Physiology, Istituto Superiore di Sanità, EBRI Foundation, University of Rome Tor Vergata, Via Montpellier, 100133 Rome, Italy
Department of Medicine of Systems, Institute of Cell Biology and Neurobiology, Department of Integrative Biology and Physiology, Istituto Superiore di Sanità, EBRI Foundation, University of Rome Tor Vergata, Via Montpellier, 100133 Rome, Italy

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Alessandro Bartolomucci Department of Medicine of Systems, Institute of Cell Biology and Neurobiology, Department of Integrative Biology and Physiology, Istituto Superiore di Sanità, EBRI Foundation, University of Rome Tor Vergata, Via Montpellier, 100133 Rome, Italy

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Roberta Possenti Department of Medicine of Systems, Institute of Cell Biology and Neurobiology, Department of Integrative Biology and Physiology, Istituto Superiore di Sanità, EBRI Foundation, University of Rome Tor Vergata, Via Montpellier, 100133 Rome, Italy
Department of Medicine of Systems, Institute of Cell Biology and Neurobiology, Department of Integrative Biology and Physiology, Istituto Superiore di Sanità, EBRI Foundation, University of Rome Tor Vergata, Via Montpellier, 100133 Rome, Italy

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various peptides stored in large, dense core granules and secreted via the regulated secretory pathway in response to different stimuli, such as membrane depolarization and intracellular signaling activation ( Possenti et al . 1989 , 1999 , Trani et al

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-14-0313
Volume/Issue:
Volume 54: Issue 3
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Nicole Gallo-Payet Division of Endocrinology, Department of Medicine, Faculté de médecine et des sciences de la santé, Université de Sherbrooke, Sherbrooke, Quebec, Canada
Division of Endocrinology, Department of Medicine, Faculté de médecine et des sciences de la santé, Université de Sherbrooke, Sherbrooke, Quebec, Canada

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determined that ACTH(1–16) is the minimal sequence required for ACTH binding to MC2R and downstream signaling ( Kapas et al. 1996 , Chen et al. 2007 ). In addition, some ACTH fragments not only lack activity, but act as competitive antagonists of full

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JME-15-0257
Volume/Issue:
Volume 56: Issue 4
Free access