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Li Hu, Fengli He, Meifeng Huang, Meihua Peng, Zhiguang Zhou, Feng Liu, and Yan-Shan Dai

inflammation causes inflammation and insulin resistance in other insulin-sensitive tissues ( Rosen & Spiegelman 2006 , Guilherme et al . 2008 , Reilly & Saltiel 2017 ). Our results also were in line with previous studies that showed lipolysis in AT promotes

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I J Bujalska, M Quinkler, J W Tomlinson, C T Montague, D M Smith, and P M Stewart

electrolyte homeostasis and lipolysis in human adipocytes was decreased 2.7-fold by cortisol and was below detection levels in SC cells. Common adipose-specific genes Twenty-eight genes were upregulated by cortisol in both

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Jacqueline M Wallace, John S Milne, Raymond P Aitken, and Clare L Adam

the first report on adipose tissue HSL gene expression in relation to offspring gender, and the higher expression in males compared with females is commensurate with steroid hormone activation of HSL leading to enhanced lipolysis ( Mayes & Watson

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Xu-guang Zhu, Dong Wook Kim, Michael L Goodson, Martin L Privalsky, and Sheue-Yann Cheng

phenotype described here for the TRα1-PV mutation, a TRα1-P398H mutation produced visceral adiposity, hyperleptinemia, reduced sensitivity to catecholamine-stimulated lipolysis, and hepatic steatosis ( Liu et al . 2003 ). These divergent metabolic outcomes

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Stefania Carobbio, Barry Rosen, and Antonio Vidal-Puig

profile and mature brown adipocytes displayed functional properties such as increased lipolysis in response to β-adrenergic stimulation and oxygen consumption levels. Stable, faithful maintenance of both the brown and white adipocyte phenotypes could be

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Guojun Shi, Chen Sun, Weiqiong Gu, Minglan Yang, Xiaofang Zhang, Nan Zhai, Yan Lu, Zhijian Zhang, Peishun Shou, Zhiguo Zhang, and Guang Ning

and regulated gut hormones and motility ( Xiong et al . 2004 , Samuel et al . 2008 ). Then, several other groups argued that FFAR2 rather than FFAR3 inhibited lipolysis and triggered leptin secretion and adipogenesis ( Hong et al . 2005 , Ge et

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Amy Warner and Jens Mittag

JJ Brent GA 2003 A thyroid hormone receptor alpha gene mutation (P398H) is associated with visceral adiposity and impaired catecholamine-stimulated lipolysis in mice . Journal of Biological Chemistry 278 38913 – 38920 . doi:10.1074/jbc

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S M Jeyakumar, A Vajreswari, B Sesikeran, and N V Giridharan

study of obesity. Scandinavian Journal of Laboratory Animal Sciences 23 131 –139. Gong HX , Guo XR, Fei L & Guo M, Liu QQ & Chen RH 2003 Lipolysis and apoptosis of adipocytes induced by neuropeptide Y-Y5 receptor

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Loes P M Duivenvoorde, Evert M van Schothorst, Annelies Bunschoten, and Jaap Keijer

 kDa lysophospholipase 12.8 2.70×10 −12 Lipolysis   Lpcat3 Lysophospholipid acyltransferase 5 2.7 2.10×10 −12 Lipolysis   Rdh11 * Retinol dehydrogenase 11 9.6 6.10×10 −13 Reduction of short-chain  aldehydes   Srebf2 Sterol regulatory element

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Louise K Metcalfe, Greg C Smith, and Nigel Turner

. 2003 , Coll et al . 2008 , Henique et al . 2010 , Capel et al . 2016 ). Indeed, the apparently futile, ATP-consuming cycle of FA re-esterification to TAGs during adipocyte lipolysis appears designed to protect cells from lipotoxic stress