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Subhamoy Dasgupta and Bert W O'Malley

protected from high-fat diet-induced obesity and exhibit increased insulin sensitivity, higher lipolysis, and reduced fat uptake ( Picard et al . 2002 ). Loss of Src-2 −/− also affects the hepatic glucose release due to decreased expression of glucose-6

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Rebecca Roy, Caitlyn Nguyen-Ngo, and Martha Lappas

force in term pregnant rat myometrium . Journal of Physiology 593 4603 – 4614 . ( https://doi.org/10.1113/JP270631 ) Heimann E Nyman M Degerman E 2015 Propionic acid and butyric acid inhibit lipolysis and de novo lipogenesis and increase

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Kazuyoshi Ukena, Tomohiro Osugi, Jérôme Leprince, Hubert Vaudry, and Kazuyoshi Tsutsui

-induced lipolysis ( Malumba et al . 2010 ). As 3T3-L1 cells express the QRFPR-encoding gene, it appears that 26RFa/QRFP may act in an autocrine/paracrine manner to regulate adipogenesis ( Malumba et al . 2010 ). According to Alonzeau et al . (2013) , 26RFa

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Elisa Manieri and Guadalupe Sabio

secretion of adipokines ( Gavrilova et al . 2000 ). In the obese state, adipocyte enlargement induces molecular and cellular alterations that affect whole-body metabolic homeostasis. Increased lipolysis in adipocytes leads to increased hyperlipidaemia and

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Sofia A Rahman, Azizun Nessa, and Khalid Hussain

inappropriate release leads to persistent hyperinsulinaemic hypoglycaemia (HH). This drives glucose into the insulin sensitive tissues and simultaneously suppresses lipolysis and ketogenesis. The combined hypoketonaemic and hypoglycaemic characteristic of CHI

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Edwin J W Geven, Folkert Verkaar, Gert Flik, and Peter H M Klaren

, Schreck 2000 ). Cortisol stimulates gluconeogenesis and lipolysis ( Sheridan 1988 , van der Boon et al. 1991 ), which result in increased plasma glucose and free fatty acid levels. These catabolic actions of cortisol reflect its glucocorticoid potency

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Ting Qi, Yanming Chen, Honggui Li, Ya Pei, Shih-Lung Woo, Xin Guo, Jiajia Zhao, Xiaoxian Qian, Joseph Awika, Yuqing Huo, and Chaodong Wu

and resveratrol ameliorate muscle insulin resistance through preventing lipolysis and inflammation in hypoxic adipose tissue . Cell Signal 28 1401 – 1411 . ( doi:10.1016/j.cellsig.2016.06.018 ) Zheng J Woo S-L Hu X Botchlett R

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Carolina Gustavsson, Tomoyoshi Soga, Erik Wahlström, Mattias Vesterlund, Alireza Azimi, Gunnar Norstedt, and Petra Tollet-Egnell

. 2005 ) and antioxidant ( Louet et al . 2004 , Baba et al . 2005 , Borrás et al . 2005 ) properties. Oestrogens have also been shown to inhibit lipogenesis and stimulate lipolysis in abdominal visceral fat depots, to promote use of lipid as a fuel

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J H Duncan Bassett and Graham R Williams

hormone receptors . Bone 36 607 – 616 . Liu YY Schultz JJ Brent GA 2003 A thyroid hormone receptor alpha gene mutation (P398H) is associated with visceral adiposity and impaired catecholamine-stimulated lipolysis in mice . Journal of

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Fumihiko Hakuno and Shin-Ichiro Takahashi

, lipolysis and protein degradation. IGFs predominantly mediate long-term action to determine the cell fates, whereas insulin mainly possesses metabolic activity. Despite the profuseness and diversity of these effects of IGFs, the in vitro biological