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Jenica H Kakadia Department of Biochemistry, Schulich School of Medicine and Dentistry, The University of Western Ontario, London, Ontario, Canada
Children's Health Research Institute, London, Ontario, Canada

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Muhammad U Khalid Department of Biochemistry, Schulich School of Medicine and Dentistry, The University of Western Ontario, London, Ontario, Canada

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Ilka U Heinemann Department of Biochemistry, Schulich School of Medicine and Dentistry, The University of Western Ontario, London, Ontario, Canada

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Victor K Han Department of Biochemistry, Schulich School of Medicine and Dentistry, The University of Western Ontario, London, Ontario, Canada
Children's Health Research Institute, London, Ontario, Canada
Department of Pediatrics, Schulich School of Medicine and Dentistry, The University of Western Ontario, London, Ontario, Canada

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established, studies have implicated the insulin-like growth factor (IGF) system to play an important role ( Holmes et al. 1997 ). IGFs are critical for optimal fetal and placental growth and development, secreted by the fetal liver and placenta in pregnancy

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M H Abel
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D Baban
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S Lee
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H M Charlton
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P J O'Shaughnessy Department of Physiology, Institute of Comparative Medicine, Anatomy and Genetics, University of Oxford, Le Gros Clarke Building, Oxford OX1 3QX, UK

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decarboxylase 5.0 H19 H19 fetal liver mRNA 3.8 Igf1 Insulin-like growth factor 1 5.0 Pdgfc Platelet-derived growth factor C 3.7 Fah Fumarylacetoacetate hydrolase 4.9 Hsd17b11 Hydroxysteroid (17β) dehydrogenase 11 3.7 Mpzl2 Myelin protein zero-like 2 4.9 Cabc1

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Dorka Nagy Department of Metabolism, Digestion and Reproduction, Section of Genetics and Genomics, Imperial College London, London, UK
National Heart and Lung Institute, Imperial College London, London, UK

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Hannah Maude Department of Metabolism, Digestion and Reproduction, Section of Genetics and Genomics, Imperial College London, London, UK

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Graeme M Birdsey National Heart and Lung Institute, Imperial College London, London, UK

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Anna M Randi National Heart and Lung Institute, Imperial College London, London, UK

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Inês Cebola Department of Metabolism, Digestion and Reproduction, Section of Genetics and Genomics, Imperial College London, London, UK

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cell (HSC) migration into the fetal liver ( Géraud et al. 2017 ). Overexpression of GATA4 in HUVECs suppressed expression of the junctional molecule Cadherin-5 (VE-cadherin), suggesting that GATA4 decreases junctional stability in fetal LSECs and thus

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