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J Kim Departments of Molecular Microbiology and Immunology,
Urology and Preventive Medicine, and
Pathology, Biochemistry and Molecular Biology, USC/Norris Comprehensive Cancer Center, Keck School of Medicine, University of Southern California, Los Angeles, CA 90089, USA

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L Jia Departments of Molecular Microbiology and Immunology,
Urology and Preventive Medicine, and
Pathology, Biochemistry and Molecular Biology, USC/Norris Comprehensive Cancer Center, Keck School of Medicine, University of Southern California, Los Angeles, CA 90089, USA

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M R Stallcup Departments of Molecular Microbiology and Immunology,
Urology and Preventive Medicine, and
Pathology, Biochemistry and Molecular Biology, USC/Norris Comprehensive Cancer Center, Keck School of Medicine, University of Southern California, Los Angeles, CA 90089, USA

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G A Coetzee Departments of Molecular Microbiology and Immunology,
Urology and Preventive Medicine, and
Pathology, Biochemistry and Molecular Biology, USC/Norris Comprehensive Cancer Center, Keck School of Medicine, University of Southern California, Los Angeles, CA 90089, USA

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Introduction Untreated prostate cancer progresses from androgen-dependent tumors that respond favorably to clinical therapies to recurrent, metastatic, androgen-independent tumors that are invariably fatal. Even at the latter

Article Type:
Research Article
DOI:
https://doi.org/10.1677/jme.1.01701
Volume/Issue:
Volume 34: Issue 1
Free access
Anuradha Mishra National Institute for Research in Reproductive and Child Health, ICMR, Parel, Mumbai, India

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Nirmalya Ganguli National Institute of Immunology, New Delhi, India
National Institute of Animal Biotechnology (NIAB), Hyderabad, Telangana, India

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Subeer S Majumdar National Institute of Immunology, New Delhi, India
National Institute of Animal Biotechnology (NIAB), Hyderabad, Telangana, India

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Deepak Modi National Institute for Research in Reproductive and Child Health, ICMR, Parel, Mumbai, India

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Introduction Endometrial cancer is the fourth most common malignancy in women. GLOBOCAN cancer statistics in 2020 estimated that 604,127 women globally, and 123,907 women in India alone had endometrial cancer ( Sung et al. 2021 ). Moreover

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-22-0051
Volume/Issue:
Volume 69: Issue 3
Restricted access
Cinzia Puppin Dipartimento di Scienze Mediche e Biologiche, Department of Medicine, Dipartimento di Medicina Interna e Specialità Mediche, Dipartimento di Scienze Farmacobiologiche, Azienda Ospedaliero-Universitaria ‘S. Maria della Misericordia’, Università di Udine, Piazzale Kolbe 1, 33100 Udine, Italy

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Nadia Passon Dipartimento di Scienze Mediche e Biologiche, Department of Medicine, Dipartimento di Medicina Interna e Specialità Mediche, Dipartimento di Scienze Farmacobiologiche, Azienda Ospedaliero-Universitaria ‘S. Maria della Misericordia’, Università di Udine, Piazzale Kolbe 1, 33100 Udine, Italy

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Jerome M Hershman Dipartimento di Scienze Mediche e Biologiche, Department of Medicine, Dipartimento di Medicina Interna e Specialità Mediche, Dipartimento di Scienze Farmacobiologiche, Azienda Ospedaliero-Universitaria ‘S. Maria della Misericordia’, Università di Udine, Piazzale Kolbe 1, 33100 Udine, Italy

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Sebastiano Filetti Dipartimento di Scienze Mediche e Biologiche, Department of Medicine, Dipartimento di Medicina Interna e Specialità Mediche, Dipartimento di Scienze Farmacobiologiche, Azienda Ospedaliero-Universitaria ‘S. Maria della Misericordia’, Università di Udine, Piazzale Kolbe 1, 33100 Udine, Italy

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Stefania Bulotta Dipartimento di Scienze Mediche e Biologiche, Department of Medicine, Dipartimento di Medicina Interna e Specialità Mediche, Dipartimento di Scienze Farmacobiologiche, Azienda Ospedaliero-Universitaria ‘S. Maria della Misericordia’, Università di Udine, Piazzale Kolbe 1, 33100 Udine, Italy

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Marilena Celano Dipartimento di Scienze Mediche e Biologiche, Department of Medicine, Dipartimento di Medicina Interna e Specialità Mediche, Dipartimento di Scienze Farmacobiologiche, Azienda Ospedaliero-Universitaria ‘S. Maria della Misericordia’, Università di Udine, Piazzale Kolbe 1, 33100 Udine, Italy

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Diego Russo Dipartimento di Scienze Mediche e Biologiche, Department of Medicine, Dipartimento di Medicina Interna e Specialità Mediche, Dipartimento di Scienze Farmacobiologiche, Azienda Ospedaliero-Universitaria ‘S. Maria della Misericordia’, Università di Udine, Piazzale Kolbe 1, 33100 Udine, Italy

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Giuseppe Damante Dipartimento di Scienze Mediche e Biologiche, Department of Medicine, Dipartimento di Medicina Interna e Specialità Mediche, Dipartimento di Scienze Farmacobiologiche, Azienda Ospedaliero-Universitaria ‘S. Maria della Misericordia’, Università di Udine, Piazzale Kolbe 1, 33100 Udine, Italy
Dipartimento di Scienze Mediche e Biologiche, Department of Medicine, Dipartimento di Medicina Interna e Specialità Mediche, Dipartimento di Scienze Farmacobiologiche, Azienda Ospedaliero-Universitaria ‘S. Maria della Misericordia’, Università di Udine, Piazzale Kolbe 1, 33100 Udine, Italy

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, Hingorani et al . 2010 ). In the majority of thyroid cancers, and particularly in metastatic lesions, reduction or abolition of NIS gene expression occurs very often ( Lazar et al . 1999 , Arturi et al . 2000 ). Loss of NIS gene expression is related

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-11-0063
Volume/Issue:
Volume 48: Issue 3
Free access
Jack-Michel Renoir CNRS, Université Paris-Sud, IFR 141, Biologie moléculaire et cellulaire de la signalisation, UMR 8612, Pharmacologie Cellulaire et Moléculaire des Anticancéreux, Faculté de Pharmacie, 5 rue JB Clément, Châtenay-Malabry F-92296, France
CNRS, Université Paris-Sud, IFR 141, Biologie moléculaire et cellulaire de la signalisation, UMR 8612, Pharmacologie Cellulaire et Moléculaire des Anticancéreux, Faculté de Pharmacie, 5 rue JB Clément, Châtenay-Malabry F-92296, France
CNRS, Université Paris-Sud, IFR 141, Biologie moléculaire et cellulaire de la signalisation, UMR 8612, Pharmacologie Cellulaire et Moléculaire des Anticancéreux, Faculté de Pharmacie, 5 rue JB Clément, Châtenay-Malabry F-92296, France

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Céline Bouclier CNRS, Université Paris-Sud, IFR 141, Biologie moléculaire et cellulaire de la signalisation, UMR 8612, Pharmacologie Cellulaire et Moléculaire des Anticancéreux, Faculté de Pharmacie, 5 rue JB Clément, Châtenay-Malabry F-92296, France
CNRS, Université Paris-Sud, IFR 141, Biologie moléculaire et cellulaire de la signalisation, UMR 8612, Pharmacologie Cellulaire et Moléculaire des Anticancéreux, Faculté de Pharmacie, 5 rue JB Clément, Châtenay-Malabry F-92296, France
CNRS, Université Paris-Sud, IFR 141, Biologie moléculaire et cellulaire de la signalisation, UMR 8612, Pharmacologie Cellulaire et Moléculaire des Anticancéreux, Faculté de Pharmacie, 5 rue JB Clément, Châtenay-Malabry F-92296, France

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Amélie Seguin CNRS, Université Paris-Sud, IFR 141, Biologie moléculaire et cellulaire de la signalisation, UMR 8612, Pharmacologie Cellulaire et Moléculaire des Anticancéreux, Faculté de Pharmacie, 5 rue JB Clément, Châtenay-Malabry F-92296, France

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Véronique Marsaud CNRS, Université Paris-Sud, IFR 141, Biologie moléculaire et cellulaire de la signalisation, UMR 8612, Pharmacologie Cellulaire et Moléculaire des Anticancéreux, Faculté de Pharmacie, 5 rue JB Clément, Châtenay-Malabry F-92296, France
CNRS, Université Paris-Sud, IFR 141, Biologie moléculaire et cellulaire de la signalisation, UMR 8612, Pharmacologie Cellulaire et Moléculaire des Anticancéreux, Faculté de Pharmacie, 5 rue JB Clément, Châtenay-Malabry F-92296, France
CNRS, Université Paris-Sud, IFR 141, Biologie moléculaire et cellulaire de la signalisation, UMR 8612, Pharmacologie Cellulaire et Moléculaire des Anticancéreux, Faculté de Pharmacie, 5 rue JB Clément, Châtenay-Malabry F-92296, France

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Brigitte Sola CNRS, Université Paris-Sud, IFR 141, Biologie moléculaire et cellulaire de la signalisation, UMR 8612, Pharmacologie Cellulaire et Moléculaire des Anticancéreux, Faculté de Pharmacie, 5 rue JB Clément, Châtenay-Malabry F-92296, France

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breast cancer (BC) tumour behaviour and is much more strongly expressed in BC tumours than ERβ ( Dahlman-Wright et al . 2006 ). It is currently thought that ERβ represses growth by inhibiting ERα-mediated transcriptional activity ( Hall & McDonnell 1999

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JME-07-0143
Volume/Issue:
Volume 40: Issue 3
Free access
Andrew P Trotta Cancer Biology Group, Dame Roma Mitchell Cancer Research Laboratories, Departments of Preventive Medicine and Urology, Level 1 Basil Hetzel Institute for Translational Health Research, Freemasons Foundation Centre for Men's Health, Queen Elizabeth Hospital, University of Adelaide, 28 Woodville Road, Woodville South, Adelaide, South Australia 5011, Australia
Cancer Biology Group, Dame Roma Mitchell Cancer Research Laboratories, Departments of Preventive Medicine and Urology, Level 1 Basil Hetzel Institute for Translational Health Research, Freemasons Foundation Centre for Men's Health, Queen Elizabeth Hospital, University of Adelaide, 28 Woodville Road, Woodville South, Adelaide, South Australia 5011, Australia

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Eleanor F Need Cancer Biology Group, Dame Roma Mitchell Cancer Research Laboratories, Departments of Preventive Medicine and Urology, Level 1 Basil Hetzel Institute for Translational Health Research, Freemasons Foundation Centre for Men's Health, Queen Elizabeth Hospital, University of Adelaide, 28 Woodville Road, Woodville South, Adelaide, South Australia 5011, Australia

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Lisa M Butler Cancer Biology Group, Dame Roma Mitchell Cancer Research Laboratories, Departments of Preventive Medicine and Urology, Level 1 Basil Hetzel Institute for Translational Health Research, Freemasons Foundation Centre for Men's Health, Queen Elizabeth Hospital, University of Adelaide, 28 Woodville Road, Woodville South, Adelaide, South Australia 5011, Australia

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Luke A Selth Cancer Biology Group, Dame Roma Mitchell Cancer Research Laboratories, Departments of Preventive Medicine and Urology, Level 1 Basil Hetzel Institute for Translational Health Research, Freemasons Foundation Centre for Men's Health, Queen Elizabeth Hospital, University of Adelaide, 28 Woodville Road, Woodville South, Adelaide, South Australia 5011, Australia

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Melissa A O'Loughlin Cancer Biology Group, Dame Roma Mitchell Cancer Research Laboratories, Departments of Preventive Medicine and Urology, Level 1 Basil Hetzel Institute for Translational Health Research, Freemasons Foundation Centre for Men's Health, Queen Elizabeth Hospital, University of Adelaide, 28 Woodville Road, Woodville South, Adelaide, South Australia 5011, Australia

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Gerhard A Coetzee Cancer Biology Group, Dame Roma Mitchell Cancer Research Laboratories, Departments of Preventive Medicine and Urology, Level 1 Basil Hetzel Institute for Translational Health Research, Freemasons Foundation Centre for Men's Health, Queen Elizabeth Hospital, University of Adelaide, 28 Woodville Road, Woodville South, Adelaide, South Australia 5011, Australia

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Wayne D Tilley Cancer Biology Group, Dame Roma Mitchell Cancer Research Laboratories, Departments of Preventive Medicine and Urology, Level 1 Basil Hetzel Institute for Translational Health Research, Freemasons Foundation Centre for Men's Health, Queen Elizabeth Hospital, University of Adelaide, 28 Woodville Road, Woodville South, Adelaide, South Australia 5011, Australia

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Grant Buchanan Cancer Biology Group, Dame Roma Mitchell Cancer Research Laboratories, Departments of Preventive Medicine and Urology, Level 1 Basil Hetzel Institute for Translational Health Research, Freemasons Foundation Centre for Men's Health, Queen Elizabeth Hospital, University of Adelaide, 28 Woodville Road, Woodville South, Adelaide, South Australia 5011, Australia

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be indicative of an altered status of receptor activation or response. This is most commonly observed in prostate cancer following the failure of androgen deprivation therapy, whereby the AR subcellular distribution shifts to become predominately

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-11-0152
Volume/Issue:
Volume 49: Issue 2
Free access
Angela Nebbioso Dipartimento di Patologia Generale, Dipartimento di Patologia e Microbiologia Sperimentale, University of Southern Denmark, Dipartimento di Studi Farmaceutici, Department of Cancer Biology, Università degli Studi di Milano, CNR-IGB, Seconda Università di Napoli, 80138 Napoli, Italy

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Carmela Dell'Aversana Dipartimento di Patologia Generale, Dipartimento di Patologia e Microbiologia Sperimentale, University of Southern Denmark, Dipartimento di Studi Farmaceutici, Department of Cancer Biology, Università degli Studi di Milano, CNR-IGB, Seconda Università di Napoli, 80138 Napoli, Italy
Dipartimento di Patologia Generale, Dipartimento di Patologia e Microbiologia Sperimentale, University of Southern Denmark, Dipartimento di Studi Farmaceutici, Department of Cancer Biology, Università degli Studi di Milano, CNR-IGB, Seconda Università di Napoli, 80138 Napoli, Italy

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Anne Bugge Dipartimento di Patologia Generale, Dipartimento di Patologia e Microbiologia Sperimentale, University of Southern Denmark, Dipartimento di Studi Farmaceutici, Department of Cancer Biology, Università degli Studi di Milano, CNR-IGB, Seconda Università di Napoli, 80138 Napoli, Italy

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Roberta Sarno Dipartimento di Patologia Generale, Dipartimento di Patologia e Microbiologia Sperimentale, University of Southern Denmark, Dipartimento di Studi Farmaceutici, Department of Cancer Biology, Università degli Studi di Milano, CNR-IGB, Seconda Università di Napoli, 80138 Napoli, Italy

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Sergio Valente Dipartimento di Patologia Generale, Dipartimento di Patologia e Microbiologia Sperimentale, University of Southern Denmark, Dipartimento di Studi Farmaceutici, Department of Cancer Biology, Università degli Studi di Milano, CNR-IGB, Seconda Università di Napoli, 80138 Napoli, Italy

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Dante Rotili Dipartimento di Patologia Generale, Dipartimento di Patologia e Microbiologia Sperimentale, University of Southern Denmark, Dipartimento di Studi Farmaceutici, Department of Cancer Biology, Università degli Studi di Milano, CNR-IGB, Seconda Università di Napoli, 80138 Napoli, Italy

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Fabio Manzo Dipartimento di Patologia Generale, Dipartimento di Patologia e Microbiologia Sperimentale, University of Southern Denmark, Dipartimento di Studi Farmaceutici, Department of Cancer Biology, Università degli Studi di Milano, CNR-IGB, Seconda Università di Napoli, 80138 Napoli, Italy
Dipartimento di Patologia Generale, Dipartimento di Patologia e Microbiologia Sperimentale, University of Southern Denmark, Dipartimento di Studi Farmaceutici, Department of Cancer Biology, Università degli Studi di Milano, CNR-IGB, Seconda Università di Napoli, 80138 Napoli, Italy

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Diana Teti Dipartimento di Patologia Generale, Dipartimento di Patologia e Microbiologia Sperimentale, University of Southern Denmark, Dipartimento di Studi Farmaceutici, Department of Cancer Biology, Università degli Studi di Milano, CNR-IGB, Seconda Università di Napoli, 80138 Napoli, Italy

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Susanne Mandrup Dipartimento di Patologia Generale, Dipartimento di Patologia e Microbiologia Sperimentale, University of Southern Denmark, Dipartimento di Studi Farmaceutici, Department of Cancer Biology, Università degli Studi di Milano, CNR-IGB, Seconda Università di Napoli, 80138 Napoli, Italy

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Paolo Ciana Dipartimento di Patologia Generale, Dipartimento di Patologia e Microbiologia Sperimentale, University of Southern Denmark, Dipartimento di Studi Farmaceutici, Department of Cancer Biology, Università degli Studi di Milano, CNR-IGB, Seconda Università di Napoli, 80138 Napoli, Italy

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Adriana Maggi Dipartimento di Patologia Generale, Dipartimento di Patologia e Microbiologia Sperimentale, University of Southern Denmark, Dipartimento di Studi Farmaceutici, Department of Cancer Biology, Università degli Studi di Milano, CNR-IGB, Seconda Università di Napoli, 80138 Napoli, Italy

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Antonello Mai Dipartimento di Patologia Generale, Dipartimento di Patologia e Microbiologia Sperimentale, University of Southern Denmark, Dipartimento di Studi Farmaceutici, Department of Cancer Biology, Università degli Studi di Milano, CNR-IGB, Seconda Università di Napoli, 80138 Napoli, Italy

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Hinrich Gronemeyer Dipartimento di Patologia Generale, Dipartimento di Patologia e Microbiologia Sperimentale, University of Southern Denmark, Dipartimento di Studi Farmaceutici, Department of Cancer Biology, Università degli Studi di Milano, CNR-IGB, Seconda Università di Napoli, 80138 Napoli, Italy

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Lucia Altucci Dipartimento di Patologia Generale, Dipartimento di Patologia e Microbiologia Sperimentale, University of Southern Denmark, Dipartimento di Studi Farmaceutici, Department of Cancer Biology, Università degli Studi di Milano, CNR-IGB, Seconda Università di Napoli, 80138 Napoli, Italy
Dipartimento di Patologia Generale, Dipartimento di Patologia e Microbiologia Sperimentale, University of Southern Denmark, Dipartimento di Studi Farmaceutici, Department of Cancer Biology, Università degli Studi di Milano, CNR-IGB, Seconda Università di Napoli, 80138 Napoli, Italy

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Introduction In the past years, epigenetic therapies have come of age. The finding that histone deacetylases (HDACs) often are more expressed in tumor cells gave credit to the application of HDAC inhibitors (HDACIs) to target cancer cells, without

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JME-10-0043
Volume/Issue:
Volume 45: Issue 4
Free access
Jandee Lee Departments of Surgery, Internal Medicine, Pathology

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Mi-Youn Seol Departments of Surgery, Internal Medicine, Pathology

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Seonhyang Jeong Departments of Surgery, Internal Medicine, Pathology

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Hyeong Ju Kwon Departments of Surgery, Internal Medicine, Pathology

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Cho Rok Lee Departments of Surgery, Internal Medicine, Pathology

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Cheol Ryong Ku Departments of Surgery, Internal Medicine, Pathology

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Sang-Wook Kang Departments of Surgery, Internal Medicine, Pathology

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Jong Ju Jeong Departments of Surgery, Internal Medicine, Pathology

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Dong Yeob Shin Departments of Surgery, Internal Medicine, Pathology

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Kee-Hyun Nam Departments of Surgery, Internal Medicine, Pathology

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Eun Jig Lee Departments of Surgery, Internal Medicine, Pathology

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Woong Youn Chung Departments of Surgery, Internal Medicine, Pathology

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Young Suk Jo Departments of Surgery, Internal Medicine, Pathology

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Introduction Papillary thyroid cancer (PTC) is the most common endocrine malignancy, and generally has favorable outcomes ( DeGroot et al . 1990 , Wada et al . 2003 , Pacini et al . 2006 , Kweon et al . 2013 ); however, a significant

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-14-0270
Volume/Issue:
Volume 54: Issue 2
Free access
Sandra E Ghayad Université de Lyon, Université Lyon 1, INSERM, Centre Léon Bérard, Laboratoire d'Oncogénétique, Université Lyon 1, 69008 Lyon, France
Université de Lyon, Université Lyon 1, INSERM, Centre Léon Bérard, Laboratoire d'Oncogénétique, Université Lyon 1, 69008 Lyon, France
Université de Lyon, Université Lyon 1, INSERM, Centre Léon Bérard, Laboratoire d'Oncogénétique, Université Lyon 1, 69008 Lyon, France
Université de Lyon, Université Lyon 1, INSERM, Centre Léon Bérard, Laboratoire d'Oncogénétique, Université Lyon 1, 69008 Lyon, France

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Julie A Vendrell Université de Lyon, Université Lyon 1, INSERM, Centre Léon Bérard, Laboratoire d'Oncogénétique, Université Lyon 1, 69008 Lyon, France
Université de Lyon, Université Lyon 1, INSERM, Centre Léon Bérard, Laboratoire d'Oncogénétique, Université Lyon 1, 69008 Lyon, France
Université de Lyon, Université Lyon 1, INSERM, Centre Léon Bérard, Laboratoire d'Oncogénétique, Université Lyon 1, 69008 Lyon, France
Université de Lyon, Université Lyon 1, INSERM, Centre Léon Bérard, Laboratoire d'Oncogénétique, Université Lyon 1, 69008 Lyon, France

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Ivan Bieche Université de Lyon, Université Lyon 1, INSERM, Centre Léon Bérard, Laboratoire d'Oncogénétique, Université Lyon 1, 69008 Lyon, France

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Frédérique Spyratos Université de Lyon, Université Lyon 1, INSERM, Centre Léon Bérard, Laboratoire d'Oncogénétique, Université Lyon 1, 69008 Lyon, France

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Charles Dumontet Université de Lyon, Université Lyon 1, INSERM, Centre Léon Bérard, Laboratoire d'Oncogénétique, Université Lyon 1, 69008 Lyon, France
Université de Lyon, Université Lyon 1, INSERM, Centre Léon Bérard, Laboratoire d'Oncogénétique, Université Lyon 1, 69008 Lyon, France
Université de Lyon, Université Lyon 1, INSERM, Centre Léon Bérard, Laboratoire d'Oncogénétique, Université Lyon 1, 69008 Lyon, France

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Isabelle Treilleux Université de Lyon, Université Lyon 1, INSERM, Centre Léon Bérard, Laboratoire d'Oncogénétique, Université Lyon 1, 69008 Lyon, France

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Rosette Lidereau Université de Lyon, Université Lyon 1, INSERM, Centre Léon Bérard, Laboratoire d'Oncogénétique, Université Lyon 1, 69008 Lyon, France

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Pascale A Cohen Université de Lyon, Université Lyon 1, INSERM, Centre Léon Bérard, Laboratoire d'Oncogénétique, Université Lyon 1, 69008 Lyon, France
Université de Lyon, Université Lyon 1, INSERM, Centre Léon Bérard, Laboratoire d'Oncogénétique, Université Lyon 1, 69008 Lyon, France
Université de Lyon, Université Lyon 1, INSERM, Centre Léon Bérard, Laboratoire d'Oncogénétique, Université Lyon 1, 69008 Lyon, France
Université de Lyon, Université Lyon 1, INSERM, Centre Léon Bérard, Laboratoire d'Oncogénétique, Université Lyon 1, 69008 Lyon, France

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Introduction Breast carcinoma, the most common female cancer, is currently undergoing an alarming increase. Breast cancer growth is regulated by estrogen, which acts by binding to the estrogen receptor α (ERα). ERα is routinely used as a prognostic

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JME-08-0076
Volume/Issue:
Volume 42: Issue 2
Free access
Klaus W Frommer
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Katharina Reichenmiller
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Burkhardt S Schutt
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Andreas Hoeflich
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Michael B Ranke
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Gabriele Dodt
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Martin W Elmlinger
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in detail so far. One possible approach towards the identification of IGF-I-independent pathways of IGFBP-2 actions involves the human breast cancer cell line Hs578T, which possesses no functional IGF-I receptor, thereby excluding IGF

Article Type:
Research Article
DOI:
https://doi.org/10.1677/jme.1.01955
Volume/Issue:
Volume 37: Issue 1
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Kyounghyun Kim Department of Veterinary Physiology and Pharmacology, Institute of Biosciences and Technology, Veterinary Integrative Biosciences, Biochemistry and Biophysics, Texas A&M University, 4466 TAMU, Veterinary Research Building 410, College Station, Texas 77843-4466, USA
Department of Veterinary Physiology and Pharmacology, Institute of Biosciences and Technology, Veterinary Integrative Biosciences, Biochemistry and Biophysics, Texas A&M University, 4466 TAMU, Veterinary Research Building 410, College Station, Texas 77843-4466, USA

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Robert Burghardt Department of Veterinary Physiology and Pharmacology, Institute of Biosciences and Technology, Veterinary Integrative Biosciences, Biochemistry and Biophysics, Texas A&M University, 4466 TAMU, Veterinary Research Building 410, College Station, Texas 77843-4466, USA

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Rola Barhoumi Department of Veterinary Physiology and Pharmacology, Institute of Biosciences and Technology, Veterinary Integrative Biosciences, Biochemistry and Biophysics, Texas A&M University, 4466 TAMU, Veterinary Research Building 410, College Station, Texas 77843-4466, USA

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Syng-ook Lee Department of Veterinary Physiology and Pharmacology, Institute of Biosciences and Technology, Veterinary Integrative Biosciences, Biochemistry and Biophysics, Texas A&M University, 4466 TAMU, Veterinary Research Building 410, College Station, Texas 77843-4466, USA

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Xinyi Liu Department of Veterinary Physiology and Pharmacology, Institute of Biosciences and Technology, Veterinary Integrative Biosciences, Biochemistry and Biophysics, Texas A&M University, 4466 TAMU, Veterinary Research Building 410, College Station, Texas 77843-4466, USA

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Stephen Safe Department of Veterinary Physiology and Pharmacology, Institute of Biosciences and Technology, Veterinary Integrative Biosciences, Biochemistry and Biophysics, Texas A&M University, 4466 TAMU, Veterinary Research Building 410, College Station, Texas 77843-4466, USA
Department of Veterinary Physiology and Pharmacology, Institute of Biosciences and Technology, Veterinary Integrative Biosciences, Biochemistry and Biophysics, Texas A&M University, 4466 TAMU, Veterinary Research Building 410, College Station, Texas 77843-4466, USA

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/tissue context-dependent manner ( Brooks & Gu 2006 ). Since MDM2 inhibits p53 function as a tumor suppressor gene, it is not surprising that expression of MDM2 plays a role in cancer and in cancer prognosis ( Levav-Cohen et al . 2005 ). Overexpression of MDM2 is

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JME-10-0110
Volume/Issue:
Volume 46: Issue 2
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