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Kun Chen Key Laboratory of Swine Genetics and Breeding of Agricultural Ministry, Wuhan Institute of Animal Science and Veterinary Medicine, and Key Laboratory of Agricultural Animal Genetics, Breeding and Reproduction of Ministry of Education, College of Animal Science and Technology, Huazhong Agricultural University, Wuhan 430070, People's Republic of China

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Ji-Dan Zhou Key Laboratory of Swine Genetics and Breeding of Agricultural Ministry, Wuhan Institute of Animal Science and Veterinary Medicine, and Key Laboratory of Agricultural Animal Genetics, Breeding and Reproduction of Ministry of Education, College of Animal Science and Technology, Huazhong Agricultural University, Wuhan 430070, People's Republic of China

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Feng Zhang Key Laboratory of Swine Genetics and Breeding of Agricultural Ministry, Wuhan Institute of Animal Science and Veterinary Medicine, and Key Laboratory of Agricultural Animal Genetics, Breeding and Reproduction of Ministry of Education, College of Animal Science and Technology, Huazhong Agricultural University, Wuhan 430070, People's Republic of China

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Fang Zhang Key Laboratory of Swine Genetics and Breeding of Agricultural Ministry, Wuhan Institute of Animal Science and Veterinary Medicine, and Key Laboratory of Agricultural Animal Genetics, Breeding and Reproduction of Ministry of Education, College of Animal Science and Technology, Huazhong Agricultural University, Wuhan 430070, People's Republic of China

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Rui-Rui Zhang Key Laboratory of Swine Genetics and Breeding of Agricultural Ministry, Wuhan Institute of Animal Science and Veterinary Medicine, and Key Laboratory of Agricultural Animal Genetics, Breeding and Reproduction of Ministry of Education, College of Animal Science and Technology, Huazhong Agricultural University, Wuhan 430070, People's Republic of China

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Meng-Si Zhan Key Laboratory of Swine Genetics and Breeding of Agricultural Ministry, Wuhan Institute of Animal Science and Veterinary Medicine, and Key Laboratory of Agricultural Animal Genetics, Breeding and Reproduction of Ministry of Education, College of Animal Science and Technology, Huazhong Agricultural University, Wuhan 430070, People's Republic of China

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Xiao-Yin Tang Key Laboratory of Swine Genetics and Breeding of Agricultural Ministry, Wuhan Institute of Animal Science and Veterinary Medicine, and Key Laboratory of Agricultural Animal Genetics, Breeding and Reproduction of Ministry of Education, College of Animal Science and Technology, Huazhong Agricultural University, Wuhan 430070, People's Republic of China

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Bing Deng Key Laboratory of Swine Genetics and Breeding of Agricultural Ministry, Wuhan Institute of Animal Science and Veterinary Medicine, and Key Laboratory of Agricultural Animal Genetics, Breeding and Reproduction of Ministry of Education, College of Animal Science and Technology, Huazhong Agricultural University, Wuhan 430070, People's Republic of China

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Ming-Gang Lei Key Laboratory of Swine Genetics and Breeding of Agricultural Ministry, Wuhan Institute of Animal Science and Veterinary Medicine, and Key Laboratory of Agricultural Animal Genetics, Breeding and Reproduction of Ministry of Education, College of Animal Science and Technology, Huazhong Agricultural University, Wuhan 430070, People's Republic of China

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Yuan-Zhu Xiong Key Laboratory of Swine Genetics and Breeding of Agricultural Ministry, Wuhan Institute of Animal Science and Veterinary Medicine, and Key Laboratory of Agricultural Animal Genetics, Breeding and Reproduction of Ministry of Education, College of Animal Science and Technology, Huazhong Agricultural University, Wuhan 430070, People's Republic of China

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Tsujimoto G 2005 Free fatty acids regulate gut incretin glucagon-like peptide-1 secretion through GPR120 . Nature Medicine 11 90 – 94 . ( doi:10.1038/nm1168 ). Ichimura A Hirasawa A Poulain-Godefroy O Bonnefond A Hara T Yengo L Kimura

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-15-0200
Volume/Issue:
Volume 56: Issue 2
Free access
Ping Li Department of Endocrinology, Drum Tower Hospital Affiliated to Nanjing University Medical School, Nanjing, People’s Republic of China

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Yan Hao Department of Endocrinology, Drum Tower Hospital Affiliated to Nanjing University Medical School, Nanjing, People’s Republic of China

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Feng-Hui Pan Department of Endocrinology, Drum Tower Hospital Affiliated to Nanjing University Medical School, Nanjing, People’s Republic of China

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Min Zhang Department of Endocrinology, Drum Tower Hospital Affiliated to Nanjing University Medical School, Nanjing, People’s Republic of China

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Jian-Qiang Ma Department of Endocrinology, Drum Tower Hospital Affiliated to Nanjing University Medical School, Nanjing, People’s Republic of China

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Da-Long Zhu Department of Endocrinology, Drum Tower Hospital Affiliated to Nanjing University Medical School, Nanjing, People’s Republic of China

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investigate the effect of SGK1 inhibitors on the secretion of incretin. Excessive glucocorticoids result in whole-body insulin resistance, obesity, and hyperglycemia ( Geer et al . 2014 ). Metabolic syndrome shares many clinical features with excess

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-15-0285
Volume/Issue:
Volume 56: Issue 4
Free access
Daiana Araujo Santana-Oliveira Laboratory of Morphometry, Metabolism, and Cardiovascular Diseases, Biomedical Center, Institute of Biology, State University of Rio de Janeiro, Rio de Janeiro, Brazil

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Aline Fernandes-da-Silva Laboratory of Morphometry, Metabolism, and Cardiovascular Diseases, Biomedical Center, Institute of Biology, State University of Rio de Janeiro, Rio de Janeiro, Brazil

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Carolline Santos Miranda Laboratory of Morphometry, Metabolism, and Cardiovascular Diseases, Biomedical Center, Institute of Biology, State University of Rio de Janeiro, Rio de Janeiro, Brazil

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Fabiane Ferreira Martins Laboratory of Morphometry, Metabolism, and Cardiovascular Diseases, Biomedical Center, Institute of Biology, State University of Rio de Janeiro, Rio de Janeiro, Brazil

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Carlos Alberto Mandarim-de-Lacerda Laboratory of Morphometry, Metabolism, and Cardiovascular Diseases, Biomedical Center, Institute of Biology, State University of Rio de Janeiro, Rio de Janeiro, Brazil

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Vanessa Souza-Mello Laboratory of Morphometry, Metabolism, and Cardiovascular Diseases, Biomedical Center, Institute of Biology, State University of Rio de Janeiro, Rio de Janeiro, Brazil

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incretin hormone action . Cell Metabolism 17 819 – 837 . ( https://doi.org/10.1016/j.cmet.2013.04.008 ) 23684623 Chen FJ Yin Y Chua BT Li P 2020 CIDE family proteins control lipid homeostasis and the development of metabolic diseases . Traffic

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-21-0084
Volume/Issue:
Volume 68: Issue 4
Restricted access
G A Martens Diabetes Research Center, Department of Clinical Chemistry and Radioimmunology, Department of Medical Biochemistry, Department of Biomedical Sciences, Brussels Free University (VUB), Laarbeeklaan 103, B1090 Brussel, Belgium
Diabetes Research Center, Department of Clinical Chemistry and Radioimmunology, Department of Medical Biochemistry, Department of Biomedical Sciences, Brussels Free University (VUB), Laarbeeklaan 103, B1090 Brussel, Belgium

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E Motté Diabetes Research Center, Department of Clinical Chemistry and Radioimmunology, Department of Medical Biochemistry, Department of Biomedical Sciences, Brussels Free University (VUB), Laarbeeklaan 103, B1090 Brussel, Belgium

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G Kramer Diabetes Research Center, Department of Clinical Chemistry and Radioimmunology, Department of Medical Biochemistry, Department of Biomedical Sciences, Brussels Free University (VUB), Laarbeeklaan 103, B1090 Brussel, Belgium

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G Stangé Diabetes Research Center, Department of Clinical Chemistry and Radioimmunology, Department of Medical Biochemistry, Department of Biomedical Sciences, Brussels Free University (VUB), Laarbeeklaan 103, B1090 Brussel, Belgium

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L W Gaarn Diabetes Research Center, Department of Clinical Chemistry and Radioimmunology, Department of Medical Biochemistry, Department of Biomedical Sciences, Brussels Free University (VUB), Laarbeeklaan 103, B1090 Brussel, Belgium

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K Hellemans Diabetes Research Center, Department of Clinical Chemistry and Radioimmunology, Department of Medical Biochemistry, Department of Biomedical Sciences, Brussels Free University (VUB), Laarbeeklaan 103, B1090 Brussel, Belgium

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J H Nielsen Diabetes Research Center, Department of Clinical Chemistry and Radioimmunology, Department of Medical Biochemistry, Department of Biomedical Sciences, Brussels Free University (VUB), Laarbeeklaan 103, B1090 Brussel, Belgium

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J M Aerts Diabetes Research Center, Department of Clinical Chemistry and Radioimmunology, Department of Medical Biochemistry, Department of Biomedical Sciences, Brussels Free University (VUB), Laarbeeklaan 103, B1090 Brussel, Belgium

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Z Ling Diabetes Research Center, Department of Clinical Chemistry and Radioimmunology, Department of Medical Biochemistry, Department of Biomedical Sciences, Brussels Free University (VUB), Laarbeeklaan 103, B1090 Brussel, Belgium

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D Pipeleers Diabetes Research Center, Department of Clinical Chemistry and Radioimmunology, Department of Medical Biochemistry, Department of Biomedical Sciences, Brussels Free University (VUB), Laarbeeklaan 103, B1090 Brussel, Belgium

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vesicle exocytosis, and incretin signaling. Our data argue against this: in fact, when specifically analyzed by oligonucleotide array for their mRNA expression of a large panel of conserved β cell marker genes ( Martens et al . 2011 ), neonatal β cells

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-13-0106
Volume/Issue:
Volume 52: Issue 1
Free access
Haihua Yang Division of Endocrinology, Zhengzhou Children’s Hospital, Zhengzhou, Henan, China

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Linghai Yang Department of Pharmacology, University of Washington, Seattle, Washington, USA

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homeostasis by cAMP/PKA pathway in pancreatic β cell, hepatocyte, and adipocyte. (A) Regulation of insulin production by cAMP/PKA signaling in pancreatic β cell. Upon stimulation by incretins such as glucagon-like peptide 1 (GLP1), PKA is activated to promote

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JME-15-0316
Volume/Issue:
Volume 57: Issue 2
Free access
Brenda Vidrio-Huerta Institute of Clinical Biochemistry, Hannover Medical School, Hannover, Germany

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Thomas Plötz Institute of Clinical Biochemistry, Hannover Medical School, Hannover, Germany

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Stephan Lortz Institute of Clinical Biochemistry, Hannover Medical School, Hannover, Germany

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hypoglycaemia to maturity-onset diabetes of the young, and incretin response . Diabetic Medicine 31 e11 – e15 . ( https://doi.org/10.1111/dme.12369 ) Bestetti S Galli M Sorrentino I Pinton P Rimessi A Sitia R & Medrano-Fernandez I 2020 Human

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-23-0087
Volume/Issue:
Volume 72: Issue 2
Restricted access
Zhaoqian Xu Department of Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Key Laboratory for Endocrine Tumor, State Key Laboratory of Medical Genomics, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Key Laboratory for Endocrine Tumor, State Key Laboratory of Medical Genomics, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China

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Yiru Wang Department of VIP Clinic, Shanghai East Hospital, Tongji University School of Medicine, Shanghai, China
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Key Laboratory for Endocrine Tumor, State Key Laboratory of Medical Genomics, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China

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Qianqian Liu Department of Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Key Laboratory for Endocrine Tumor, State Key Laboratory of Medical Genomics, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China

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Shushu Wang Department of Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Key Laboratory for Endocrine Tumor, State Key Laboratory of Medical Genomics, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China

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Chunxiang Sheng Department of Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Key Laboratory for Endocrine Tumor, State Key Laboratory of Medical Genomics, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China

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Junmin Chen Department of Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Academy of Integrative Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai, China

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Jialin Tan Department of Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Key Laboratory for Endocrine Tumor, State Key Laboratory of Medical Genomics, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China

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Xiao Wang Department of Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Key Laboratory for Endocrine Tumor, State Key Laboratory of Medical Genomics, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China

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Li Shao Department of VIP Clinic, Shanghai East Hospital, Tongji University School of Medicine, Shanghai, China

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Libin Zhou Department of Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Key Laboratory for Endocrine Tumor, State Key Laboratory of Medical Genomics, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China

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Menshikov M Vorotnikov AV Parfyonova YV , 2019 Low AS160 and high SGK basal phosphorylation associates with impaired incretin profile and type 2 diabetes in adipose tissue of obese patients . Diabetes Research and Clinical Practice 158 107928

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-23-0046
Volume/Issue:
Volume 71: Issue 2
Restricted access
Sheng Y Ang Drug Discovery Biology, Monash Institute of Pharmaceutical Sciences, Monash University, Parkville, Victoria, Australia

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Bronwyn A Evans Drug Discovery Biology, Monash Institute of Pharmaceutical Sciences, Monash University, Parkville, Victoria, Australia

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Daniel P Poole Drug Discovery Biology, Monash Institute of Pharmaceutical Sciences, Monash University, Parkville, Victoria, Australia

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Romke Bron Drug Discovery Biology, Monash Institute of Pharmaceutical Sciences, Monash University, Parkville, Victoria, Australia

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Jesse J DiCello Drug Discovery Biology, Monash Institute of Pharmaceutical Sciences, Monash University, Parkville, Victoria, Australia

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Ross A D Bathgate The Florey Institute of Neuroscience and Mental Health, University of Melbourne, Parkville, Victoria, Australia
Department of Biochemistry and Molecular Biology, University of Melbourne, Melbourne, Victoria, Australia

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Martina Kocan The Florey Institute of Neuroscience and Mental Health, University of Melbourne, Parkville, Victoria, Australia
Department of Biochemistry and Molecular Biology, University of Melbourne, Melbourne, Victoria, Australia

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Dana S Hutchinson Drug Discovery Biology, Monash Institute of Pharmaceutical Sciences, Monash University, Parkville, Victoria, Australia
Department of Pharmacology, Monash University, Clayton, Victoria, Australia

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Roger J Summers Drug Discovery Biology, Monash Institute of Pharmaceutical Sciences, Monash University, Parkville, Victoria, Australia

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transcriptome . Genome Research 23 1554 – 1562 . ( https://doi.org/10.1101/gr.150706.112 ) 23716500 10.1101/gr.150706.112 Ørskov C Wettergren A Holst JJ 1996 Secretion of the incretin hormones glucagon-like peptide-1 and gastric inhibitory

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-17-0152
Volume/Issue:
Volume 60: Issue 3
Free access
Jianqiu Gu Department of Endocrinology and Metabolism and the Institute of Endocrinology, The First Hospital of China Medical University, Shenyang, People’s Republic of China
Division of Advanced Diagnostics, Toronto General Hospital Research Institute, University Health Network, Toronto, Canada

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Weijuan Shao Division of Advanced Diagnostics, Toronto General Hospital Research Institute, University Health Network, Toronto, Canada

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Dinghui Liu Division of Advanced Diagnostics, Toronto General Hospital Research Institute, University Health Network, Toronto, Canada
Department of Cardiology, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, People’s Republic of China

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Jia Nuo Feng Division of Advanced Diagnostics, Toronto General Hospital Research Institute, University Health Network, Toronto, Canada
Department of Physiology, Temerty Faculty of Medicine, University of Toronto, Toronto, Canada

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Juan Pang Division of Advanced Diagnostics, Toronto General Hospital Research Institute, University Health Network, Toronto, Canada
Department of Nutrition, School of Public Health, Sun Yat-sen University, Guangzhou, People’s Republic of China

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Tianru Jin Division of Advanced Diagnostics, Toronto General Hospital Research Institute, University Health Network, Toronto, Canada
Department of Physiology, Temerty Faculty of Medicine, University of Toronto, Toronto, Canada
Banting and Best Diabetes Centre, Temerty Faculty of Medicine, University of Toronto, Toronto, Canada

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tolerance. Following the recognition of GLP-1 as an incretin ( Mojsov et al. 1987 ), GLP-1R has been identified and its expression in pancreatic islets and elsewhere has been investigated by various means ( Campos et al. 1994 , Bullock et al. 1996

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-22-0026
Volume/Issue:
Volume 69: Issue 2
Restricted access
He-jun Zhao Department of Endocrinology, Tianjin First Central Hospital, Tianjin, China

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Xia Jiang Department of Endocrinology, Tianjin First Central Hospital, Tianjin, China

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Li-juan Hu Laboratory of Acute Abdomen Disease, Tianjin NanKai Hospital, Tianjin, China

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Lei Yang Clinical Laboratory, Tianjin First Central Hospital, Tianjin, China

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Lian-dong Deng Department of Polymer Science and Technology, School of Chemical Engineering and Technology, Key Laboratory of Systems Bioengineering (Ministry of Education), Tianjin University, Tianjin, China

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Ya-ping Wang Department of Polymer Science and Technology, School of Chemical Engineering and Technology, Key Laboratory of Systems Bioengineering (Ministry of Education), Tianjin University, Tianjin, China

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Zhi-peng Ren Department of Orthopaedics, Tianjin Hospital, Tianjin, China

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drugs in patients complicated with pancreatic cancer and diabetes mellitus has important clinical significance. Glucagon-like peptide 1 (GLP-1) is known as an incretin hormone secreted from intestinal endocrine cells, and it augments insulin production

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-19-0186
Volume/Issue:
Volume 64: Issue 2
Restricted access