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Julián Nevado Instituto de Investigaciones Biomédicas (CSIC)-Universidad Autónoma de Madrid, Arturo Duperier 4, 28029 Madrid, Spain
Unidad de Investigación, Hospital Universitario de Getafe, Getafe, 28905 Madrid, Spain
Centro Nacional de Investigaciones Oncológicas (CNIO), 28029 Madrid, Spain

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Stephan P Tenbaum Instituto de Investigaciones Biomédicas (CSIC)-Universidad Autónoma de Madrid, Arturo Duperier 4, 28029 Madrid, Spain
Unidad de Investigación, Hospital Universitario de Getafe, Getafe, 28905 Madrid, Spain
Centro Nacional de Investigaciones Oncológicas (CNIO), 28029 Madrid, Spain

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Ana Isabel Castillo Instituto de Investigaciones Biomédicas (CSIC)-Universidad Autónoma de Madrid, Arturo Duperier 4, 28029 Madrid, Spain
Unidad de Investigación, Hospital Universitario de Getafe, Getafe, 28905 Madrid, Spain
Centro Nacional de Investigaciones Oncológicas (CNIO), 28029 Madrid, Spain

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Aurora Sánchez-Pacheco Instituto de Investigaciones Biomédicas (CSIC)-Universidad Autónoma de Madrid, Arturo Duperier 4, 28029 Madrid, Spain
Unidad de Investigación, Hospital Universitario de Getafe, Getafe, 28905 Madrid, Spain
Centro Nacional de Investigaciones Oncológicas (CNIO), 28029 Madrid, Spain

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Ana Aranda Instituto de Investigaciones Biomédicas (CSIC)-Universidad Autónoma de Madrid, Arturo Duperier 4, 28029 Madrid, Spain
Unidad de Investigación, Hospital Universitario de Getafe, Getafe, 28905 Madrid, Spain
Centro Nacional de Investigaciones Oncológicas (CNIO), 28029 Madrid, Spain

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. 1989 ). In fact, data from our laboratory, among others, have demonstrated a role for T3 and retinoic acid (RA), other nuclear receptor ligands, on HIV-1 LTR regulation, demonstrating the binding of RA receptors (RARs) and T3 receptors (TRs) to

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JME-06-0065
Volume/Issue:
Volume 38: Issue 6
Free access
Changxue Lu Laboratory of Molecular Biology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, 37 Convent Drive, Room 5128, Bethesda, Maryland 20892-4264, USA

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Sheue-Yann Cheng Laboratory of Molecular Biology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, 37 Convent Drive, Room 5128, Bethesda, Maryland 20892-4264, USA

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receptors (RARs), Rev-erb, RAR-related orphan receptors (RORs), oxysterol receptors (LXRs), vitamin D3 receptors (VDRs), and the nuclear xenobiotic receptor (constitutive androstane receptor, CAR). PPARs and TRs share a conserved DNA-binding domain (DBD) and

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JME-09-0107
Volume/Issue:
Volume 44: Issue 3
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Diego Russo
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Giuseppe Damante Dipartimento di Scienze Farmacobiologiche, Dipartimento di Scienze e Tecnologie Biomediche, Dipartimento di Medicina Interna e Specialità Mediche, Dipartimento di Scienze Cliniche, Università di Catanzaro, 88100 Catanzaro, Italy

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Efisio Puxeddu Dipartimento di Scienze Farmacobiologiche, Dipartimento di Scienze e Tecnologie Biomediche, Dipartimento di Medicina Interna e Specialità Mediche, Dipartimento di Scienze Cliniche, Università di Catanzaro, 88100 Catanzaro, Italy

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Cosimo Durante Dipartimento di Scienze Farmacobiologiche, Dipartimento di Scienze e Tecnologie Biomediche, Dipartimento di Medicina Interna e Specialità Mediche, Dipartimento di Scienze Cliniche, Università di Catanzaro, 88100 Catanzaro, Italy

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Sebastiano Filetti Dipartimento di Scienze Farmacobiologiche, Dipartimento di Scienze e Tecnologie Biomediche, Dipartimento di Medicina Interna e Specialità Mediche, Dipartimento di Scienze Cliniche, Università di Catanzaro, 88100 Catanzaro, Italy

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metalloproteinase 53% PTC (associated with BRAF mutation) Hu et al . (2006) DAPK Ca/calmodulin-dependent ser/thr kinase protein 34% PTC (associated with BRAF mutation) Hu et al . (2006) RAR β 2 Negative regulator of cell growth 22% PTC (associated with BRAF

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-10-0150
Volume/Issue:
Volume 46: Issue 3
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Kyle T Helzer McArdle Laboratory for Cancer Research, Department of Oncology, 6151 Wisconsin Institutes for Medical Research, University of Wisconsin Carbone Cancer Center, University of Wisconsin–Madison, 1111 Highland Avenue, Madison, Wisconsin 53705, USA

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Christopher Hooper McArdle Laboratory for Cancer Research, Department of Oncology, 6151 Wisconsin Institutes for Medical Research, University of Wisconsin Carbone Cancer Center, University of Wisconsin–Madison, 1111 Highland Avenue, Madison, Wisconsin 53705, USA

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Shigeki Miyamoto McArdle Laboratory for Cancer Research, Department of Oncology, 6151 Wisconsin Institutes for Medical Research, University of Wisconsin Carbone Cancer Center, University of Wisconsin–Madison, 1111 Highland Avenue, Madison, Wisconsin 53705, USA

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Elaine T Alarid McArdle Laboratory for Cancer Research, Department of Oncology, 6151 Wisconsin Institutes for Medical Research, University of Wisconsin Carbone Cancer Center, University of Wisconsin–Madison, 1111 Highland Avenue, Madison, Wisconsin 53705, USA

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receptors in mammals, including ERα (NR3A1) and ERβ (NR3A2), thyroid hormone receptor (TR, NR1A1), progesterone receptor (PR, NR3C3), androgen receptor (AR, NR3C4), retinoic acid receptor (RAR, NR1B1–3), retinoic X receptor (RXR, NR2B1–3), vitamin D receptor

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JME-14-0308
Volume/Issue:
Volume 54: Issue 3
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Shannon E Mullican Division of Endocrinology, Diabetes, and Metabolism, Department of Medicine, and The Institute for Diabetes, Obesity, and Metabolism, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania 19104, USA

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Joanna R DiSpirito Division of Endocrinology, Diabetes, and Metabolism, Department of Medicine, and The Institute for Diabetes, Obesity, and Metabolism, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania 19104, USA

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Mitchell A Lazar Division of Endocrinology, Diabetes, and Metabolism, Department of Medicine, and The Institute for Diabetes, Obesity, and Metabolism, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania 19104, USA

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/Rorα, Nr1f2/Rorβ, and Nr1f3/Rorγ The RAR-related orphan NRs comprise three structurally related orphan NRs with roles in retinal development, lipid and glucose homeostasis, atherosclerosis, carcinogenesis, circadian rhythm, bone formation, and immunity

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JME-13-0212
Volume/Issue:
Volume 51: Issue 3
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Jun Yang Prince Henry's Institute of Medical Research, Department of Medicine, Monash University, PO Box 5152, Clayton, Victoria 3168, Australia
Prince Henry's Institute of Medical Research, Department of Medicine, Monash University, PO Box 5152, Clayton, Victoria 3168, Australia

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Morag J Young Prince Henry's Institute of Medical Research, Department of Medicine, Monash University, PO Box 5152, Clayton, Victoria 3168, Australia

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sequence is sufficient for activation of the ER, different combinations of two LXXLL sequences are required for the TR, retinoic acid receptor (RAR), peroxisome proliferator activated receptor (PPAR), and PR ( McInerney et al . 1998 ). Distinct residues

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JME-09-0031
Volume/Issue:
Volume 43: Issue 2
Free access
Tina Di Palma
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Tiziana de Cristofaro
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Chiara D'Ambrosio Department di Biologia e Patologia Cellulare e Molecolare, Proteomics and Mass Spectrometry Laboratory, Istituto di Endocrinologia e Oncologia Sperimentale ‘G. Salvatore’-CNR, Universita' di Napoli Federico II, Via S. Pansini 5, 80131 Napoli, Italy

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Dolores Del Prete
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Andrea Scaloni Department di Biologia e Patologia Cellulare e Molecolare, Proteomics and Mass Spectrometry Laboratory, Istituto di Endocrinologia e Oncologia Sperimentale ‘G. Salvatore’-CNR, Universita' di Napoli Federico II, Via S. Pansini 5, 80131 Napoli, Italy

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Mariastella Zannini
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factors and (co)-factors, including NF-kB ( Hassa et al . 2003 , Nakajima et al . 2004 ), AP-2 ( Kannan et al . 1999 ), B-MYB ( Cervellera & Sala 2000 ), E2F-1 ( Simbulan-Rosenthal et al . 2003 ), RAR/RXR ( Miyamoto et al . 1999 , Pavri et al

Article Type:
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DOI:
https://doi.org/10.1677/JME-08-0056
Volume/Issue:
Volume 41: Issue 5
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A Pestka
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B Toth Department of Obstetrics and Gynaecology–Innenstadt, Department of Obstetrics and Gynaecology, Ludwig‐Maximilians‐University Munich, Maistrasse 11, 80337 Munich, Germany

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C Kuhn
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S Hofmann
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I Wiest
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G Wypior
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K Friese
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U Jeschke
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homozygous knockout mice demonstrate that those mice mainly die because of heart defects and disturbed placental development ( Kastner et al . 1994 , Sucov et al . 1994 , Sapin et al . 1997 ). RXR and its heterodimeric partner RAR can be activated by

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-11-0002
Volume/Issue:
Volume 47: Issue 2
Free access
Rene Cortese Epigenomics AG Berlin, Kleine Präsidentenstr. 1, 10178 Berlin, Germany
Institute of Human Genetics, Otto-von-Guericke University, Leipziger Str. 44, 39120 Magdeburg, Germany
Institute for Human Genetics, Ernst-Moritz-Arndt University, Fleischmannstrasse 42/44, 17489 Greifswald, Germany
University Children’s Hospital, Department of Perinatology: Gerhard-Hauptmann-Str. 35, 39108 Magdeburg, Germany

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Florian Eckhardt Epigenomics AG Berlin, Kleine Präsidentenstr. 1, 10178 Berlin, Germany
Institute of Human Genetics, Otto-von-Guericke University, Leipziger Str. 44, 39120 Magdeburg, Germany
Institute for Human Genetics, Ernst-Moritz-Arndt University, Fleischmannstrasse 42/44, 17489 Greifswald, Germany
University Children’s Hospital, Department of Perinatology: Gerhard-Hauptmann-Str. 35, 39108 Magdeburg, Germany

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Marianne Volleth Epigenomics AG Berlin, Kleine Präsidentenstr. 1, 10178 Berlin, Germany
Institute of Human Genetics, Otto-von-Guericke University, Leipziger Str. 44, 39120 Magdeburg, Germany
Institute for Human Genetics, Ernst-Moritz-Arndt University, Fleischmannstrasse 42/44, 17489 Greifswald, Germany
University Children’s Hospital, Department of Perinatology: Gerhard-Hauptmann-Str. 35, 39108 Magdeburg, Germany

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Manfred Wehnert Epigenomics AG Berlin, Kleine Präsidentenstr. 1, 10178 Berlin, Germany
Institute of Human Genetics, Otto-von-Guericke University, Leipziger Str. 44, 39120 Magdeburg, Germany
Institute for Human Genetics, Ernst-Moritz-Arndt University, Fleischmannstrasse 42/44, 17489 Greifswald, Germany
University Children’s Hospital, Department of Perinatology: Gerhard-Hauptmann-Str. 35, 39108 Magdeburg, Germany

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Uwe Koelsch Epigenomics AG Berlin, Kleine Präsidentenstr. 1, 10178 Berlin, Germany
Institute of Human Genetics, Otto-von-Guericke University, Leipziger Str. 44, 39120 Magdeburg, Germany
Institute for Human Genetics, Ernst-Moritz-Arndt University, Fleischmannstrasse 42/44, 17489 Greifswald, Germany
University Children’s Hospital, Department of Perinatology: Gerhard-Hauptmann-Str. 35, 39108 Magdeburg, Germany

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Peter Wieacker Epigenomics AG Berlin, Kleine Präsidentenstr. 1, 10178 Berlin, Germany
Institute of Human Genetics, Otto-von-Guericke University, Leipziger Str. 44, 39120 Magdeburg, Germany
Institute for Human Genetics, Ernst-Moritz-Arndt University, Fleischmannstrasse 42/44, 17489 Greifswald, Germany
University Children’s Hospital, Department of Perinatology: Gerhard-Hauptmann-Str. 35, 39108 Magdeburg, Germany

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Thomas Brune Epigenomics AG Berlin, Kleine Präsidentenstr. 1, 10178 Berlin, Germany
Institute of Human Genetics, Otto-von-Guericke University, Leipziger Str. 44, 39120 Magdeburg, Germany
Institute for Human Genetics, Ernst-Moritz-Arndt University, Fleischmannstrasse 42/44, 17489 Greifswald, Germany
University Children’s Hospital, Department of Perinatology: Gerhard-Hauptmann-Str. 35, 39108 Magdeburg, Germany

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laminopathies, indicating an epigenetic component in this disease. The studied amplicon is located in intron 1 of the annotated RARB gene (Ensembl v42). The RARB gene encodes the RAR-β and mediates retinoid effects in controlling cell growth

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JME-07-0035
Volume/Issue:
Volume 38: Issue 6
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Tram B Doan Westmead Institute for Medical Research, Sydney Medical School – Westmead, University of Sydney, Sydney, New South Wales, Australia

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J Dinny Graham Westmead Institute for Medical Research, Sydney Medical School – Westmead, University of Sydney, Sydney, New South Wales, Australia

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Christine L Clarke Westmead Institute for Medical Research, Sydney Medical School – Westmead, University of Sydney, Sydney, New South Wales, Australia

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1B1 RARA 5914 All-trans and 9-cis retinoic acid Increased Yes a,b NR1B2 RARB 5915 All-trans and 9-cis retinoic acid Decreased NR1B3 RARG 5916 All-trans and 9-cis retinoic acid Decreased RAR

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JME-16-0082
Volume/Issue:
Volume 58: Issue 3
Free access