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Kumi Futawaka Department of Medicine and Clinical Science, Faculty of Pharmaceutical Sciences, Mukogawa Women’s University, Hyogo, Japan

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Tetsuya Tagami Clinical Research Institute for Endocrine and Metabolic Diseases, National Hospital Organization Kyoto Medical Center, Kyoto, Japan

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Yuki Fukuda Department of Medicine and Clinical Science, Faculty of Pharmaceutical Sciences, Mukogawa Women’s University, Hyogo, Japan

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Rie Koyama Department of Medicine and Clinical Science, Faculty of Pharmaceutical Sciences, Mukogawa Women’s University, Hyogo, Japan

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Ayaka Nushida Department of Medicine and Clinical Science, Faculty of Pharmaceutical Sciences, Mukogawa Women’s University, Hyogo, Japan

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Shoko Nezu Department of Medicine and Clinical Science, Faculty of Pharmaceutical Sciences, Mukogawa Women’s University, Hyogo, Japan

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Hironori Yamamoto Department of Health and Nutrition, Faculty of Human Life, Jin-ai University, Fukui, Japan

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Miyuki Imamoto Department of Nephrology and Blood Purification, Institute of Biomedical Research and Innovation, Kobe Medical Frontier Center, Kobe, Japan

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Masato Kasahara Department of Nephrology and Blood Purification, Institute of Biomedical Research and Innovation, Kobe Medical Frontier Center, Kobe, Japan

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Kenji Moriyama Department of Medicine and Clinical Science, Faculty of Pharmaceutical Sciences, Mukogawa Women’s University, Hyogo, Japan
Clinical Research Institute for Endocrine and Metabolic Diseases, National Hospital Organization Kyoto Medical Center, Kyoto, Japan
Department of Nephrology and Blood Purification, Institute of Biomedical Research and Innovation, Kobe Medical Frontier Center, Kobe, Japan

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Introduction The active form of Vitamin D 3 (VD 3 ) is a fat-soluble vitamin that performs transcriptional control of target genes by binding to and activating the VD receptor (VDR). In the liver, the proactive form of VD 3 is hydroxylated

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-16-0048
Volume/Issue:
Volume 57: Issue 1
Free access
Rachel S Fletcher Institute of Metabolism and Systems Research, University of Birmingham, Birmingham, UK

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Gareth G Lavery Institute of Metabolism and Systems Research, University of Birmingham, Birmingham, UK

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mammalian cells, a number of NAD + biosynthesis pathways have been established. These pathways can synthesise NAD + de novo from l -tryptophan or from the salvage of vitamin B3 NAD + precursors nicotinic acid (NA), nicotinamide (NAM) and nicotinamide

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JME-18-0085
Volume/Issue:
Volume 61: Issue 3
Open access
Francesco Maria Egro Department of Cellular and Molecular Medicine, University of Bristol, 53 Arley Hill, Bristol BS6 5PJ, UK

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to justify the rise in the incidence of this autoimmune disease. Vitamin D deficiency In a variety of epidemiological studies, type 1 diabetes appears to be conditioned by seasonal and geographical variation in u.v. light exposure ( Kimpimaki et al

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-13-0067
Volume/Issue:
Volume 51: Issue 1
Free access
M Rodríguez-Sanz IMIM (Hospital del Mar Research Institute), Internal Medicine Department, IDIAP Jordi Gol Primary Care Research Institute, Nuffield Department of Orthopaedics, MRC Lifecourse Epidemiology Unit, Medical Oncology Department, Departament de Genètica, Red Temática de Investigación Cooperativa en Envejecimiento y Fragilidad (RETICEF), ISCIII, Carrer del Doctor Aiguader 88, 08003 Barcelona, Spain

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N García-Giralt IMIM (Hospital del Mar Research Institute), Internal Medicine Department, IDIAP Jordi Gol Primary Care Research Institute, Nuffield Department of Orthopaedics, MRC Lifecourse Epidemiology Unit, Medical Oncology Department, Departament de Genètica, Red Temática de Investigación Cooperativa en Envejecimiento y Fragilidad (RETICEF), ISCIII, Carrer del Doctor Aiguader 88, 08003 Barcelona, Spain

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D Prieto-Alhambra IMIM (Hospital del Mar Research Institute), Internal Medicine Department, IDIAP Jordi Gol Primary Care Research Institute, Nuffield Department of Orthopaedics, MRC Lifecourse Epidemiology Unit, Medical Oncology Department, Departament de Genètica, Red Temática de Investigación Cooperativa en Envejecimiento y Fragilidad (RETICEF), ISCIII, Carrer del Doctor Aiguader 88, 08003 Barcelona, Spain
IMIM (Hospital del Mar Research Institute), Internal Medicine Department, IDIAP Jordi Gol Primary Care Research Institute, Nuffield Department of Orthopaedics, MRC Lifecourse Epidemiology Unit, Medical Oncology Department, Departament de Genètica, Red Temática de Investigación Cooperativa en Envejecimiento y Fragilidad (RETICEF), ISCIII, Carrer del Doctor Aiguader 88, 08003 Barcelona, Spain
IMIM (Hospital del Mar Research Institute), Internal Medicine Department, IDIAP Jordi Gol Primary Care Research Institute, Nuffield Department of Orthopaedics, MRC Lifecourse Epidemiology Unit, Medical Oncology Department, Departament de Genètica, Red Temática de Investigación Cooperativa en Envejecimiento y Fragilidad (RETICEF), ISCIII, Carrer del Doctor Aiguader 88, 08003 Barcelona, Spain
IMIM (Hospital del Mar Research Institute), Internal Medicine Department, IDIAP Jordi Gol Primary Care Research Institute, Nuffield Department of Orthopaedics, MRC Lifecourse Epidemiology Unit, Medical Oncology Department, Departament de Genètica, Red Temática de Investigación Cooperativa en Envejecimiento y Fragilidad (RETICEF), ISCIII, Carrer del Doctor Aiguader 88, 08003 Barcelona, Spain

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S Servitja IMIM (Hospital del Mar Research Institute), Internal Medicine Department, IDIAP Jordi Gol Primary Care Research Institute, Nuffield Department of Orthopaedics, MRC Lifecourse Epidemiology Unit, Medical Oncology Department, Departament de Genètica, Red Temática de Investigación Cooperativa en Envejecimiento y Fragilidad (RETICEF), ISCIII, Carrer del Doctor Aiguader 88, 08003 Barcelona, Spain

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S Balcells IMIM (Hospital del Mar Research Institute), Internal Medicine Department, IDIAP Jordi Gol Primary Care Research Institute, Nuffield Department of Orthopaedics, MRC Lifecourse Epidemiology Unit, Medical Oncology Department, Departament de Genètica, Red Temática de Investigación Cooperativa en Envejecimiento y Fragilidad (RETICEF), ISCIII, Carrer del Doctor Aiguader 88, 08003 Barcelona, Spain

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R Pecorelli IMIM (Hospital del Mar Research Institute), Internal Medicine Department, IDIAP Jordi Gol Primary Care Research Institute, Nuffield Department of Orthopaedics, MRC Lifecourse Epidemiology Unit, Medical Oncology Department, Departament de Genètica, Red Temática de Investigación Cooperativa en Envejecimiento y Fragilidad (RETICEF), ISCIII, Carrer del Doctor Aiguader 88, 08003 Barcelona, Spain
IMIM (Hospital del Mar Research Institute), Internal Medicine Department, IDIAP Jordi Gol Primary Care Research Institute, Nuffield Department of Orthopaedics, MRC Lifecourse Epidemiology Unit, Medical Oncology Department, Departament de Genètica, Red Temática de Investigación Cooperativa en Envejecimiento y Fragilidad (RETICEF), ISCIII, Carrer del Doctor Aiguader 88, 08003 Barcelona, Spain

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A Díez-Pérez IMIM (Hospital del Mar Research Institute), Internal Medicine Department, IDIAP Jordi Gol Primary Care Research Institute, Nuffield Department of Orthopaedics, MRC Lifecourse Epidemiology Unit, Medical Oncology Department, Departament de Genètica, Red Temática de Investigación Cooperativa en Envejecimiento y Fragilidad (RETICEF), ISCIII, Carrer del Doctor Aiguader 88, 08003 Barcelona, Spain
IMIM (Hospital del Mar Research Institute), Internal Medicine Department, IDIAP Jordi Gol Primary Care Research Institute, Nuffield Department of Orthopaedics, MRC Lifecourse Epidemiology Unit, Medical Oncology Department, Departament de Genètica, Red Temática de Investigación Cooperativa en Envejecimiento y Fragilidad (RETICEF), ISCIII, Carrer del Doctor Aiguader 88, 08003 Barcelona, Spain

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D Grinberg IMIM (Hospital del Mar Research Institute), Internal Medicine Department, IDIAP Jordi Gol Primary Care Research Institute, Nuffield Department of Orthopaedics, MRC Lifecourse Epidemiology Unit, Medical Oncology Department, Departament de Genètica, Red Temática de Investigación Cooperativa en Envejecimiento y Fragilidad (RETICEF), ISCIII, Carrer del Doctor Aiguader 88, 08003 Barcelona, Spain

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I Tusquets IMIM (Hospital del Mar Research Institute), Internal Medicine Department, IDIAP Jordi Gol Primary Care Research Institute, Nuffield Department of Orthopaedics, MRC Lifecourse Epidemiology Unit, Medical Oncology Department, Departament de Genètica, Red Temática de Investigación Cooperativa en Envejecimiento y Fragilidad (RETICEF), ISCIII, Carrer del Doctor Aiguader 88, 08003 Barcelona, Spain

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X Nogués IMIM (Hospital del Mar Research Institute), Internal Medicine Department, IDIAP Jordi Gol Primary Care Research Institute, Nuffield Department of Orthopaedics, MRC Lifecourse Epidemiology Unit, Medical Oncology Department, Departament de Genètica, Red Temática de Investigación Cooperativa en Envejecimiento y Fragilidad (RETICEF), ISCIII, Carrer del Doctor Aiguader 88, 08003 Barcelona, Spain
IMIM (Hospital del Mar Research Institute), Internal Medicine Department, IDIAP Jordi Gol Primary Care Research Institute, Nuffield Department of Orthopaedics, MRC Lifecourse Epidemiology Unit, Medical Oncology Department, Departament de Genètica, Red Temática de Investigación Cooperativa en Envejecimiento y Fragilidad (RETICEF), ISCIII, Carrer del Doctor Aiguader 88, 08003 Barcelona, Spain

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in postmenopausal women with early breast cancer. Previously, we reported a genetic association with AI-related arthralgia and therapy discontinuation in this cohort. SNPs in CYP17A1, CYP27B1 and VDR genes (involved in the estrogen and vitamin D

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-15-0079
Volume/Issue:
Volume 55: Issue 1
Free access
Julián Nevado Instituto de Investigaciones Biomédicas (CSIC)-Universidad Autónoma de Madrid, Arturo Duperier 4, 28029 Madrid, Spain
Unidad de Investigación, Hospital Universitario de Getafe, Getafe, 28905 Madrid, Spain
Centro Nacional de Investigaciones Oncológicas (CNIO), 28029 Madrid, Spain

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Stephan P Tenbaum Instituto de Investigaciones Biomédicas (CSIC)-Universidad Autónoma de Madrid, Arturo Duperier 4, 28029 Madrid, Spain
Unidad de Investigación, Hospital Universitario de Getafe, Getafe, 28905 Madrid, Spain
Centro Nacional de Investigaciones Oncológicas (CNIO), 28029 Madrid, Spain

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Ana Isabel Castillo Instituto de Investigaciones Biomédicas (CSIC)-Universidad Autónoma de Madrid, Arturo Duperier 4, 28029 Madrid, Spain
Unidad de Investigación, Hospital Universitario de Getafe, Getafe, 28905 Madrid, Spain
Centro Nacional de Investigaciones Oncológicas (CNIO), 28029 Madrid, Spain

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Aurora Sánchez-Pacheco Instituto de Investigaciones Biomédicas (CSIC)-Universidad Autónoma de Madrid, Arturo Duperier 4, 28029 Madrid, Spain
Unidad de Investigación, Hospital Universitario de Getafe, Getafe, 28905 Madrid, Spain
Centro Nacional de Investigaciones Oncológicas (CNIO), 28029 Madrid, Spain

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Ana Aranda Instituto de Investigaciones Biomédicas (CSIC)-Universidad Autónoma de Madrid, Arturo Duperier 4, 28029 Madrid, Spain
Unidad de Investigación, Hospital Universitario de Getafe, Getafe, 28905 Madrid, Spain
Centro Nacional de Investigaciones Oncológicas (CNIO), 28029 Madrid, Spain

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different cell types. Further transient transfection experiments of HeLa cells with the 453HIV-luc reporter in presence of increasing concentrations of vitD3 (0–1000 nM) showed that the vitamin stimulated the HIV-1 LTR in a dose-dependent manner

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JME-06-0065
Volume/Issue:
Volume 38: Issue 6
Free access
Kent Wehmeier Division of Endocrinology, Diabetes, and Metabolism, Department of Medicine, University of Florida Jacksonville College of Medicine, Jacksonville, Florida, USA

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Luisa M Onstead-Haas Division of Endocrinology, Diabetes, and Metabolism, Department of Medicine, University of Florida Jacksonville College of Medicine, Jacksonville, Florida, USA

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Norman C W Wong Department of Medicine, Biochemistry and Molecular Biology, University of Calgary, Calgary, Alberta, Canada

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Arshag D Mooradian Division of Endocrinology, Diabetes, and Metabolism, Department of Medicine, University of Florida Jacksonville College of Medicine, Jacksonville, Florida, USA

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Michael J Haas Division of Endocrinology, Diabetes, and Metabolism, Department of Medicine, University of Florida Jacksonville College of Medicine, Jacksonville, Florida, USA

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Introduction The vitamin D metabolite 24,25-[OH] 2 D 3 is generated from 25-hydroxyvitamin D 3 (25-OHD 3 ) and 1,25-dihydroxyvitamin D 3 (1,25-[OH] 2 D 3 ) by the activity of the cytochrome P450 24-hydroxylase (24A2) that is expressed in

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-16-0009
Volume/Issue:
Volume 57: Issue 2
Free access
Ana P Irazoqui INBIOSUR – CONICET, Departamento de Biología, Bioquímica and Farmacia, Universidad Nacional del Sur, San Juan 670, 8000 Bahía Blanca, Argentina

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Ricardo L Boland INBIOSUR – CONICET, Departamento de Biología, Bioquímica and Farmacia, Universidad Nacional del Sur, San Juan 670, 8000 Bahía Blanca, Argentina

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Claudia G Buitrago INBIOSUR – CONICET, Departamento de Biología, Bioquímica and Farmacia, Universidad Nacional del Sur, San Juan 670, 8000 Bahía Blanca, Argentina

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, cyclins, and CKIs in skeletal muscle cells, nor on the role of the vitamin D receptor (VDR) and p38 in hormone-dependent events. 1,25D exerts most of its biological actions through VDR, which acts as a transcription factor ( MacDonald et al . 2001 ). VDR

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-14-0102
Volume/Issue:
Volume 53: Issue 3
Free access
Guozheng Gao Center of Reproductive Medicine, Luoyang Central Hospital Affiliated to Zhengzhou University, Luoyang City, Henan Province, People’s Republic of China

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Yong Zhao Center of Assisted Reproductive Medicine, The Sixth Medical Center of PLA General Hospital, Beijing, People’s Republic of China

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Ke Wang Center of Assisted Reproductive Medicine, The Sixth Medical Center of PLA General Hospital, Beijing, People’s Republic of China

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Fang Wang Center of Reproductive Medicine, Luoyang Central Hospital Affiliated to Zhengzhou University, Luoyang City, Henan Province, People’s Republic of China

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Introduction Vitamin C (also called ascorbic acid/AA), acting as a major antioxidant inside testis ( Angulo et al. 2011 ), is such a striking example. In guinea pig, it has been shown that the deficient diet causes massive degeneration of

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-20-0101
Volume/Issue:
Volume 66: Issue 2
Restricted access
Seiji Fukumoto Fujii Memorial Institute of Medical Sciences, Institute of Advanced Medical Sciences, Tokushima University, Tokushima, Tokushima, Japan

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paraneoplastic syndrome characterized by hypophosphatemia ( Shimada et al. 2001 ). Since then it has been shown that FGF23 works as a hormone regulating phosphate and vitamin D metabolism. In addition, several other kinds of hypophosphatemic rickets

Article Type:
Review Article
Collections:
Top-cited articles of 2023
DOI:
https://doi.org/10.1530/JME-20-0089
Volume/Issue:
Volume 66: Issue 2
Free access
Francesca Moramarco Institute of Medical Sciences, University of Aberdeen, Foresterhill, Aberdeen, Scotland, UK

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Peter McCaffery Institute of Medical Sciences, University of Aberdeen, Foresterhill, Aberdeen, Scotland, UK

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impairment of LTP ( Nomoto et al. 2012 ). These results indicate synaptic transmission dysfunction and synaptic activity alterations in the hippocampus following changes in RA signalling ( Nomoto et al. 2012 ). Vitamin A-deficient (VAD) rodents show a

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JME-22-0177
Volume/Issue:
Volume 72: Issue 1
Restricted access