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Norman G Nicolson Department of Surgery, Yale School of Medicine, Yale Endocrine Neoplasia Laboratory, New Haven, Connecticut, USA

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Reju Korah Department of Surgery, Yale School of Medicine, Yale Endocrine Neoplasia Laboratory, New Haven, Connecticut, USA

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Tobias Carling Department of Surgery, Yale School of Medicine, Yale Endocrine Neoplasia Laboratory, New Haven, Connecticut, USA

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targeted molecular therapies, which have been effective in a number of other solid tumors ( Fassnacht et al. 2010 , Konda & Kirschner 2016 ). Recent genomic analyses supported the previously implicated roles of dysregulated WNT and TP53 pathways in

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-18-0262
Volume/Issue:
Volume 62: Issue 4
Free access
Nirvay Sah Department of Pathology, University of California San Diego, La Jolla, California, USA
Sanford Consortium for Regenerative Medicine, University of California San Diego, La Jolla, California, USA

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Francesca Soncin Department of Pathology, University of California San Diego, La Jolla, California, USA
Sanford Consortium for Regenerative Medicine, University of California San Diego, La Jolla, California, USA

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differentiation; not required ND Maintains differentiation potential of TSC; dispensable Wnt signaling Promotes proliferation; activation required Promotes early trophoblast lineage development; inhibition required Promotes proliferation; required

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JME-23-0131
Volume/Issue:
Volume 72: Issue 4
Restricted access
Rosa Chung School of Pharmacy and Medical Sciences, Sansom Institute for Health Research, University of South Australia, City East Campus, GPO Box 2471, Adelaide, South Australia 5001, Australia

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Cory J Xian School of Pharmacy and Medical Sciences, Sansom Institute for Health Research, University of South Australia, City East Campus, GPO Box 2471, Adelaide, South Australia 5001, Australia

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et al . 1994 ), bone morphogenic protein (BMP; Chen et al . 2004 , Jing et al . 2013 ), Wnt/B-catenin ( Macsai et al . 2008 , Dao et al . 2012 , Golovchenko et al . 2013 , Lu et al . 2013 ), fibroblast growth factor (FGF; Mancilla et al

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JME-14-0062
Volume/Issue:
Volume 53: Issue 1
Free access
I J Bujalska
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M Quinkler
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J W Tomlinson
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C T Montague
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D M Smith
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P M Stewart
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and WARS ), oncogenes ( ENPP2 ), anti-apoptotic ( OXTR ). Interestingly, in SC preadipocytes F-treatment decreased expression of two genes associated with Wnt-signalling pathways ( FZD7 and SFRP4 ; 3.5- and 3.3-fold respectively; Fig. 3

Article Type:
Research Article
DOI:
https://doi.org/10.1677/jme.1.02048
Volume/Issue:
Volume 37: Issue 2
Free access
Lekha Jain Liggins Institute, University of Auckland, Auckland, New Zealand

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Tayaza Fadason Liggins Institute, University of Auckland, Auckland, New Zealand

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William Schierding Liggins Institute, University of Auckland, Auckland, New Zealand

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Mark H Vickers Liggins Institute, University of Auckland, Auckland, New Zealand

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Justin M O’Sullivan Liggins Institute, University of Auckland, Auckland, New Zealand

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Jo K Perry Liggins Institute, University of Auckland, Auckland, New Zealand

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involved in regulation of GH signalling is the Wnt pathway, which was also among the enriched pathways (Supplementary data 1-m). There was also a significant representation of these genes in carcinogenic pathways. Five cancer terms were enriched in the KEGG

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-20-0010
Volume/Issue:
Volume 64: Issue 4
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Maria Cernea Departments of Obstetrics and Gynaecology, and Physiology and Pharmacology, Children's Health Research Institute and Lawson Health Research Institute, Western University, Room A5-132, 800 Commissioners Road East, London, Ontario, Canada, N6C 2V5

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Wei Tang Departments of Obstetrics and Gynaecology, and Physiology and Pharmacology, Children's Health Research Institute and Lawson Health Research Institute, Western University, Room A5-132, 800 Commissioners Road East, London, Ontario, Canada, N6C 2V5

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Haiyan Guan Departments of Obstetrics and Gynaecology, and Physiology and Pharmacology, Children's Health Research Institute and Lawson Health Research Institute, Western University, Room A5-132, 800 Commissioners Road East, London, Ontario, Canada, N6C 2V5

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Kaiping Yang Departments of Obstetrics and Gynaecology, and Physiology and Pharmacology, Children's Health Research Institute and Lawson Health Research Institute, Western University, Room A5-132, 800 Commissioners Road East, London, Ontario, Canada, N6C 2V5

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proliferation, we conducted genome-wide gene expression profiling and identified 134 and 164 genes that were up- and downregulated by Bmp3 respectively (data not shown). Among the significantly upregulated genes, WNT-inducible secreted protein 1 ( Wisp1/CCN4

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-15-0217
Volume/Issue:
Volume 56: Issue 1
Free access
Alberto Cascón Hereditary Endocrine Cancer Group, Spanish National Cancer Research Centre (CNIO), Madrid, Spain
Centro de Investigación Biomédica en Red de Enfermedades Raras (CIBERER), Madrid, Spain

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Bruna Calsina Hereditary Endocrine Cancer Group, Spanish National Cancer Research Centre (CNIO), Madrid, Spain

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María Monteagudo Hereditary Endocrine Cancer Group, Spanish National Cancer Research Centre (CNIO), Madrid, Spain

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Sara Mellid Hereditary Endocrine Cancer Group, Spanish National Cancer Research Centre (CNIO), Madrid, Spain

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Alberto Díaz-Talavera Hereditary Endocrine Cancer Group, Spanish National Cancer Research Centre (CNIO), Madrid, Spain
Centro de Investigación Biomédica en Red de Enfermedades Raras (CIBERER), Madrid, Spain

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Maria Currás-Freixes Hereditary Endocrine Cancer Group, Spanish National Cancer Research Centre (CNIO), Madrid, Spain

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Mercedes Robledo Hereditary Endocrine Cancer Group, Spanish National Cancer Research Centre (CNIO), Madrid, Spain
Centro de Investigación Biomédica en Red de Enfermedades Raras (CIBERER), Madrid, Spain

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TCGA Project ( Fishbein et al. 2017 ). Fishbein et al. identified, for the first time in PPGLs, a third transcriptional cluster composed of tumors with Wnt signaling-pathway activation carrying MAML3 fusions and CSDE1 mutations. PPGL

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JME-22-0167
Volume/Issue:
Volume 70: Issue 3
Free access
Parmita Kar Department of Endocrinology and Metabolism, All India Institute of Medical Sciences, New Delhi, India

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Ravinder Goswami Department of Endocrinology and Metabolism, All India Institute of Medical Sciences, New Delhi, India

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). Besides, the expression of several other vitamin-D-modulated molecules, calcium channels, and phosphate transporters was assessed. These molecules included Wnt-3A, bone sialoprotein1 ( Bsp1 ), alkaline phosphatase ( Alp ), c ollagen 1α , osteoprotegerin

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-22-0108
Volume/Issue:
Volume 70: Issue 2
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Elisabeth Sambroni
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Antoine D Rolland INRA, INSERM, UR1037, Laboratoire de Physiologie et Génomique des Poissons (LPGP), Campus de Beaulieu, Testicular Physiology and Puberty, Biosit,Biogenouest, F-35000 Rennes, France

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Jean-Jacques Lareyre
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Florence Le Gac
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.02 Q6F6A1 ctsl Cathepsin L Somatic  tcbk0048.o.16 Q29VH6 smtnb Smoothelin-b Somatic  tcay0013.g.08 Q92088 cyp2m1 Cytochrome P450 2M1 Somatic  tcba0029.p.03 O95388 wisp1 WNT1-inducible-signaling pathway protein 1  precursor (WISP-1) Somatic  tcab0002.j

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-12-0197
Volume/Issue:
Volume 50: Issue 1
Free access
Siyi Zhu Rehabilitation Medicine Center, West China Hospital, Sichuan University, Chengdu, China
Rehabilitation Key Laboratory of Sichuan Province, West China Hospital, Sichuan University, Chengdu, China
Laboratory of Endocrinology and Metabolism, Department of Endocrinology, National Key Laboratory of Biotherapy/Collaborative Innovation Center of Biotherapy and Cancer Center, West China Hospital, Sichuan University, Chengdu, China

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Hongchen He Rehabilitation Medicine Center, West China Hospital, Sichuan University, Chengdu, China

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Chengfei Gao Rehabilitation Medicine Center, West China Hospital, Sichuan University, Chengdu, China
Rehabilitation Key Laboratory of Sichuan Province, West China Hospital, Sichuan University, Chengdu, China
Institute for Disaster Management and Reconstruction, Sichuan University-The Hong Kong Polytechnic University, Chengdu, China

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Guojing Luo Laboratory of Endocrinology and Metabolism, Department of Endocrinology, National Key Laboratory of Biotherapy/Collaborative Innovation Center of Biotherapy and Cancer Center, West China Hospital, Sichuan University, Chengdu, China

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Ying Xie Laboratory of Endocrinology and Metabolism, Department of Endocrinology, National Key Laboratory of Biotherapy/Collaborative Innovation Center of Biotherapy and Cancer Center, West China Hospital, Sichuan University, Chengdu, China

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Haiming Wang Rehabilitation Medicine Center, West China Hospital, Sichuan University, Chengdu, China
Rehabilitation Key Laboratory of Sichuan Province, West China Hospital, Sichuan University, Chengdu, China

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Li Tian Laboratory of Endocrinology and Metabolism, Department of Endocrinology, National Key Laboratory of Biotherapy/Collaborative Innovation Center of Biotherapy and Cancer Center, West China Hospital, Sichuan University, Chengdu, China

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Xiang Chen Laboratory of Endocrinology and Metabolism, Department of Endocrinology, National Key Laboratory of Biotherapy/Collaborative Innovation Center of Biotherapy and Cancer Center, West China Hospital, Sichuan University, Chengdu, China

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Xijie Yu Laboratory of Endocrinology and Metabolism, Department of Endocrinology, National Key Laboratory of Biotherapy/Collaborative Innovation Center of Biotherapy and Cancer Center, West China Hospital, Sichuan University, Chengdu, China

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Chengqi He Rehabilitation Medicine Center, West China Hospital, Sichuan University, Chengdu, China
Rehabilitation Key Laboratory of Sichuan Province, West China Hospital, Sichuan University, Chengdu, China
Institute for Disaster Management and Reconstruction, Sichuan University-The Hong Kong Polytechnic University, Chengdu, China

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-wasting effect of estrogen deficiency and preserve osteoporotic bone mass, possibly via the inhibition of NF-κB, AP1 component and WNT signaling inhibitors. However, TNFα potentially appears to exert an important role in the inhibition of bone formation and

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JME-17-0218
Volume/Issue:
Volume 60: Issue 3
Free access