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Joong Kwan Kim
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Yongchul Lim Department of Anatomy, Department of Surgery, Department of Pathology, Lee Gil Ya Cancer and Diabetes Institute, Korea University College of Medicine, 126‐1, Anam‐dong 5‐ga, Seongbuk‐gu, Seoul 136-701, Korea

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Jung Ok Lee
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Young-Sun Lee Department of Anatomy, Department of Surgery, Department of Pathology, Lee Gil Ya Cancer and Diabetes Institute, Korea University College of Medicine, 126‐1, Anam‐dong 5‐ga, Seongbuk‐gu, Seoul 136-701, Korea

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Nam Hee Won Department of Anatomy, Department of Surgery, Department of Pathology, Lee Gil Ya Cancer and Diabetes Institute, Korea University College of Medicine, 126‐1, Anam‐dong 5‐ga, Seongbuk‐gu, Seoul 136-701, Korea

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Hyun Kim
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Hyeon Soo Kim
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induced by several secretagogues, such as glucose ( Grill et al . 1978 ), some amino acids ( Newsholme & Krause 2012 ), glucagon-like peptide 1 ( Irwin et al . 2007 ), and free fatty acids ( Komatsu et al . 1999 ). Among these secretagogues, glucose is

Article Type:
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DOI:
https://doi.org/10.1530/JME-14-0325
Volume/Issue:
Volume 54: Issue 3
Free access
Sushi Jiang Department of Physiology, School of Medicine, Jinan University, Guangzhou, Guangdong, China

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Hening Zhai Department of Physiology, School of Medicine, Jinan University, Guangzhou, Guangdong, China
Endoscopy Center, The First Affiliated Hospital of Jinan University, Guangzhou, Guangdong, China

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Danjie Li Department of Physiology, School of Medicine, Jinan University, Guangzhou, Guangdong, China

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Jiana Huang Department of Physiology, School of Medicine, Jinan University, Guangzhou, Guangdong, China

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Heng Zhang Department of Physiology, School of Medicine, Jinan University, Guangzhou, Guangdong, China

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Ziru Li Shenzhen University Diabetes Center, Shenzhen University Health Science Center, Shenzhen, Guangdong, China

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Weizhen Zhang Shenzhen University Diabetes Center, Shenzhen University Health Science Center, Shenzhen, Guangdong, China
Department of Surgery, University of Michigan Medical Center, Ann Arbor, Michigan, USA

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Geyang Xu Department of Physiology, School of Medicine, Jinan University, Guangzhou, Guangdong, China

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AL Minaker KL Habener JF Andersen DK 1994 The insulinotropic actions of glucose-dependent insulinotropic polypeptide (GIP) and glucagon-like peptide-1 (7–37) in normal and diabetic subjects . Regulatory Peptides 51 63 – 74 . ( doi

Article Type:
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DOI:
https://doi.org/10.1530/JME-16-0099
Volume/Issue:
Volume 57: Issue 3
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Tomoko Kato Department of Diabetes, Endocrinology and Nutrition, Graduate School of Medicine, Kyoto University, Kyoto, Japan

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Norio Harada Department of Diabetes, Endocrinology and Nutrition, Graduate School of Medicine, Kyoto University, Kyoto, Japan

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Eri Ikeguchi-Ogura Department of Diabetes, Endocrinology and Nutrition, Graduate School of Medicine, Kyoto University, Kyoto, Japan

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Akiko Sankoda Department of Diabetes, Endocrinology and Nutrition, Graduate School of Medicine, Kyoto University, Kyoto, Japan

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Tomonobu Hatoko Department of Diabetes, Endocrinology and Nutrition, Graduate School of Medicine, Kyoto University, Kyoto, Japan

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Xuejing Lu Department of Diabetes, Endocrinology and Nutrition, Graduate School of Medicine, Kyoto University, Kyoto, Japan

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Takuma Yasuda Department of Diabetes, Endocrinology and Nutrition, Graduate School of Medicine, Kyoto University, Kyoto, Japan

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Shunsuke Yamane Department of Diabetes, Endocrinology and Nutrition, Graduate School of Medicine, Kyoto University, Kyoto, Japan

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Nobuya Inagaki Department of Diabetes, Endocrinology and Nutrition, Graduate School of Medicine, Kyoto University, Kyoto, Japan

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et al. 1998 , Batterham et al. 2002 ). In addition, glucose-dependent insulinotropic polypeptide/gastric inhibitory polypeptide (GIP) is an incretin secreted from enteroendocrine K cells, and plays an important role in obesity and insulin

Article Type:
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DOI:
https://doi.org/10.1530/JME-20-0134
Volume/Issue:
Volume 66: Issue 1
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Valérie Baldacchino Laboratory of Endocrine Pathophysiology and
Laboratory of Cellular Biology of Hypertension and Molecular Medicine, Department of Medicine, Centre Hospitalier de l’Université de Montréal, Montreal, Quebec, Canada

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Sylvie Oble Laboratory of Endocrine Pathophysiology and
Laboratory of Cellular Biology of Hypertension and Molecular Medicine, Department of Medicine, Centre Hospitalier de l’Université de Montréal, Montreal, Quebec, Canada

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Patrick-Olivier Décarie Laboratory of Endocrine Pathophysiology and
Laboratory of Cellular Biology of Hypertension and Molecular Medicine, Department of Medicine, Centre Hospitalier de l’Université de Montréal, Montreal, Quebec, Canada

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Isabelle Bourdeau Laboratory of Endocrine Pathophysiology and
Laboratory of Cellular Biology of Hypertension and Molecular Medicine, Department of Medicine, Centre Hospitalier de l’Université de Montréal, Montreal, Quebec, Canada

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Pavel Hamet Laboratory of Endocrine Pathophysiology and
Laboratory of Cellular Biology of Hypertension and Molecular Medicine, Department of Medicine, Centre Hospitalier de l’Université de Montréal, Montreal, Quebec, Canada

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Johanne Tremblay Laboratory of Endocrine Pathophysiology and
Laboratory of Cellular Biology of Hypertension and Molecular Medicine, Department of Medicine, Centre Hospitalier de l’Université de Montréal, Montreal, Quebec, Canada

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André Lacroix Laboratory of Endocrine Pathophysiology and
Laboratory of Cellular Biology of Hypertension and Molecular Medicine, Department of Medicine, Centre Hospitalier de l’Université de Montréal, Montreal, Quebec, Canada

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1575 –1585. Boylan MO , Jepeal LI & Wolfe MM 1999 Structure of the rat glucose-dependent insulinotropic polypeptide receptor gene. Peptides 20 219 –228. Buggy J , Hull J & Yoo-Warren H 1995

Article Type:
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DOI:
https://doi.org/10.1677/jme.1.01765
Volume/Issue:
Volume 35: Issue 1
Free access
Paola Moreno Department of Metabolism, CIBERDEM, Nutrition and Hormones, IIS-Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain
Department of Metabolism, CIBERDEM, Nutrition and Hormones, IIS-Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain

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Bernardo Nuche-Berenguer Department of Metabolism, CIBERDEM, Nutrition and Hormones, IIS-Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain
Department of Metabolism, CIBERDEM, Nutrition and Hormones, IIS-Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain

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Irene Gutiérrez-Rojas Department of Metabolism, CIBERDEM, Nutrition and Hormones, IIS-Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain
Department of Metabolism, CIBERDEM, Nutrition and Hormones, IIS-Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain

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Alicia Acitores Department of Metabolism, CIBERDEM, Nutrition and Hormones, IIS-Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain
Department of Metabolism, CIBERDEM, Nutrition and Hormones, IIS-Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain

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Verónica Sancho Department of Metabolism, CIBERDEM, Nutrition and Hormones, IIS-Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain

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Isabel Valverde Department of Metabolism, CIBERDEM, Nutrition and Hormones, IIS-Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain
Department of Metabolism, CIBERDEM, Nutrition and Hormones, IIS-Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain

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Nieves González Department of Metabolism, CIBERDEM, Nutrition and Hormones, IIS-Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain
Department of Metabolism, CIBERDEM, Nutrition and Hormones, IIS-Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain

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María L Villanueva-Peñacarrillo Department of Metabolism, CIBERDEM, Nutrition and Hormones, IIS-Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain
Department of Metabolism, CIBERDEM, Nutrition and Hormones, IIS-Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain

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developed in our laboratory ( Valverde et al . 1988 ). Glucose transport The soleus muscles in rats were individually attached by the tendon to a steel clip ( Valverde et al . 1994 ) and were incubated in the absence of added peptides (control), for 60 min

Article Type:
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DOI:
https://doi.org/10.1530/JME-11-0127
Volume/Issue:
Volume 48: Issue 1
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Haihua Yang Division of Endocrinology, Zhengzhou Children’s Hospital, Zhengzhou, Henan, China

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Linghai Yang Department of Pharmacology, University of Washington, Seattle, Washington, USA

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-like peptide I and glucose-dependent insulinotropic polypeptide stimulate Ca2+-induced secretion in rat alpha-cells by a protein kinase A-mediated mechanism . Diabetes 46 792 – 800 . ( doi:10.2337/diab.46.5.792 ) Dunning BE Gerich JE 2007 The

Article Type:
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DOI:
https://doi.org/10.1530/JME-15-0316
Volume/Issue:
Volume 57: Issue 2
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Verónica Sancho Department of Metabolism, Nutrition and Hormones, Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain
Laboratory of Experimental Hormonology, Brussels Free University, Brussels, Belgium

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María V Trigo Department of Metabolism, Nutrition and Hormones, Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain
Laboratory of Experimental Hormonology, Brussels Free University, Brussels, Belgium

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Nieves González Department of Metabolism, Nutrition and Hormones, Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain
Laboratory of Experimental Hormonology, Brussels Free University, Brussels, Belgium

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Isabel Valverde Department of Metabolism, Nutrition and Hormones, Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain
Laboratory of Experimental Hormonology, Brussels Free University, Brussels, Belgium

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Willy J Malaisse Department of Metabolism, Nutrition and Hormones, Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain
Laboratory of Experimental Hormonology, Brussels Free University, Brussels, Belgium

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María L Villanueva-Peñacarrillo Department of Metabolism, Nutrition and Hormones, Fundación Jiménez Díaz, Avda. Reyes Católicos, 2, 28040 Madrid, Spain
Laboratory of Experimental Hormonology, Brussels Free University, Brussels, Belgium

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peptide, shares 53% of its amino acid sequence with GLP-1; it is also insulinotropic ( Cancelas et al. 2001 a ) and exerts GLP-1-like effects in rat liver and skeletal muscle, such as stimulation of glycogen synthase α, glycogen synthesis and glucose

Article Type:
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DOI:
https://doi.org/10.1677/jme.1.01747
Volume/Issue:
Volume 35: Issue 1
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Drake Hechter Department of Food and Human Nutritional Sciences, University of Manitoba, Winnipeg, Manitoba, Canada

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Brett Vahkal Department of Biology, The University of Winnipeg, Winnipeg, Manitoba, Canada
Department of Biochemistry, Microbiology and Immunology, University of Ottawa, Ottawa, Ontario, Canada

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Tiana Tiede Department of Biology, The University of Winnipeg, Winnipeg, Manitoba, Canada

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Sara V Good Department of Biology, The University of Winnipeg, Winnipeg, Manitoba, Canada
Department of Biological Sciences, The University of Manitoba, Winnipeg, Manitoba, Canada

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insulin secretion at 16.8 mM glucose. Similar results were seen with 100 nM GLP-1 treatment at both glucose concentrations, leading the authors to conclude that the insulinotropic effect of INSL5 on mouse pancreatic islets is glucose-dependent ( Luo et al

Article Type:
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DOI:
https://doi.org/10.1530/JME-21-0241
Volume/Issue:
Volume 69: Issue 1
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Seisuke Sato Department of Endocrinology and Metabolism, Faculty of Medicine, Kagawa University, Kita-gun, Kagawa, Japan

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Hitomi Imachi Department of Endocrinology and Metabolism, Faculty of Medicine, Kagawa University, Kita-gun, Kagawa, Japan

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Jingya Lyu Department of Endocrinology and Metabolism, Faculty of Medicine, Kagawa University, Kita-gun, Kagawa, Japan

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Yumi Miyai Department of Endocrinology and Metabolism, Faculty of Medicine, Kagawa University, Kita-gun, Kagawa, Japan

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Kensaku Fukunaga Department of Endocrinology and Metabolism, Faculty of Medicine, Kagawa University, Kita-gun, Kagawa, Japan

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Tao Dong Department of Endocrinology and Metabolism, Faculty of Medicine, Kagawa University, Kita-gun, Kagawa, Japan

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Tomohiro Ibata Department of Endocrinology and Metabolism, Faculty of Medicine, Kagawa University, Kita-gun, Kagawa, Japan

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Toshihiro Kobayashi Department of Endocrinology and Metabolism, Faculty of Medicine, Kagawa University, Kita-gun, Kagawa, Japan

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Takuo Yoshimoto Department of Endocrinology and Metabolism, Faculty of Medicine, Kagawa University, Kita-gun, Kagawa, Japan

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Fumi Kikuchi Department of Endocrinology and Metabolism, Faculty of Medicine, Kagawa University, Kita-gun, Kagawa, Japan

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Kazuko Yonezaki Department of Endocrinology and Metabolism, Faculty of Medicine, Kagawa University, Kita-gun, Kagawa, Japan

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Nao Yamaji Department of Endocrinology and Metabolism, Faculty of Medicine, Kagawa University, Kita-gun, Kagawa, Japan

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Hisakazu Iwama Life Science Research Center, Kagawa University, Kita-gun, Kagawa, Japan

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Koji Murao Department of Endocrinology and Metabolism, Faculty of Medicine, Kagawa University, Kita-gun, Kagawa, Japan

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glucose for 48 h, and also high glucose-induced ABCA1 suppression was sensitive to p38-MAPK inhibitors. On the other hand, glucose-dependent insulinotropic polypeptide potentiated GSIS, insulin biosynthesis and β-cell proliferation and survival

Article Type:
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DOI:
https://doi.org/10.1530/JME-18-0167
Volume/Issue:
Volume 61: Issue 4
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Ahmed A Hasan Institute of Nutritional Science, University of Potsdam, Potsdam, Germany
Department of Biochemistry, Faculty of Pharmacy, Zagazig University, Zagazig, Egypt

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Berthold Hocher Institute of Nutritional Science, University of Potsdam, Potsdam, Germany
Institut für Laboriatorumsmedizin IFLb, Berlin, Germany
Departments of Embryology and Nephrology, Basic Medical College, Jinan University, Guangzhou, China

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-1; SDF-1a, stromal derived factor-1 alpha; GIP, glucose dependent insulinotropic polypeptide; BNP, brain natriuretic peptide; NPY, neuropeptide Y. Table 1 DPP-4 inhibitors in animal models of diabetic nephropathy. Animal

Article Type:
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DOI:
https://doi.org/10.1530/JME-17-0005
Volume/Issue:
Volume 59: Issue 1
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