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, however, did not appear to be at increased risk for this adenocarcinoma, suggesting that in utero programming may be a mechanism for this observation. Since then, interest in studying the endocrine effects of exogenous agents, and their role in
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( Park et al. 2010 , Grogg et al. 2015 ), has emerged both as a marker of outcome and as a potential treatment target ( Cordera & Jordan 2006 , Barton et al. 2015 ). A number of other nuclear receptors, including the vitamin D3 receptor (VDR
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tethering it to the promoter region in target DNA; simultaneously, there is a conformational change in ERα, permitting it to interact with a wide array of nuclear receptor coregulatory proteins ( McKenna & O'Malley 2002 , Smith & O'Malley 2004 , Edwards
Department of Veterinary Physiology and Pharmacology, Institute of Biosciences and Technology, Texas A&M University, College Station, Texas 77843-4466, USA
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ERα/AP-1 by reversing the effects of SRC2 ( Teyssier et al . 2003 ). By contrast, studies with the vitamin D-interacting protein 150 (DRIP150) coactivator demonstrated that DRIP150 coactivation of ERα/Sp was LXXLL-box-independent and required a novel
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Introduction The androgen receptor (AR) is a member of the nuclear receptor superfamily and mediates the effects of androgens ( Mangelsdorf et al. 1995 ). Upon binding of testosterone or 5α-dihydrotestosterone (DHT), the
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Introduction The tight junction (TJ) complex acts as a selectively permeable intercellular occlusion barrier in vertebrate epithelia and contributes to the regulated paracellular movement of solutes between fluid compartments ( Farquhar
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precise analysis of the lethal TR α knockin mutations. T3 is generally considered as a trophic factor whereas retinoic acid, a vitamin A derivative which is not chemically related to T3, has been originally proposed to be a morphogen ( Wolpert 1989
Sorbonne Université, CNRS -Institut de Biologie Paris Seine (BIOSIPE), Paris, France
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, vertebrate sex hormones (progestagens, estrogens, androgens), insect moulting hormones (ecdysteroids), as well as plant growth hormones (brassinosteroids). The rigid carbon skeleton of sterols is particularly suitable to generate a very large number of
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transcript encoding vitamin D 24-hydroxylase (Cyp24a1) ( Rasmussen et al . 1972 , Larkins et al . 1974 , Horiuchi et al . 1977 , Henry 1985 , Shigematsu et al . 1986 , Brenza et al . 1998 , Zierold et al . 2001 ). PTH inhibits the reabsorption
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Hereditary Endocrine Cancer Group, Centro de Investigación Biomédica en Red de Enfermedades Raras (CIBERER), Spanish National Cancer Research Centre (CNIO), Melchor Fernández Almagro 3, 28029 Madrid, Spain
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et al . 2001 ), probably due to the improvement in diagnostic techniques ( Davies & Welch 2006 ) and to the effect of environmental factors ( Chen et al . 2009 ). ‘Thyroid cancer’ is a general term that includes two main groups of neoplasias
