Search Results

You are looking at 101 - 106 of 106 items for :

  • Refine by Access: All content x
Clear All
Free access

Pia Kiilerich, Gérard Triqueneaux, Nynne Meyn Christensen, Vincent Trayer, Xavier Terrien, Marc Lombès, and Patrick Prunet

receptors ( Chen et al . 1997 ). In all of these studies, the inhibition of nuclear receptor transactivation activity is associated with mechanisms involving heterodimerization formation that have been demonstrated through gel shift (EMSA) experiments ( Liu

Free access

S Zhang, X Li, R Burghardt, R Smith III, and S H Safe

electrophoretic mobility shift assay (EMSA) The probe containing a consensus estrogen-responsive element (ERE; 5′-GTCCAAAGTCAGGTCACAGTGACCTGATCAAAGTT-3′) was synthesized, annealed and 32 P-labeled at the 5′-end using T4 polynucleotide kinase (Roche) and

Free access

Young Sun Kang, Yun Gyu Park, Bo Kyung Kim, Sang Youb Han, Yi Hwa Jee, Kum Hyun Han, Mi Hwa Lee, Hye Kyoung Song, Dae Ryong Cha, Shin Wook Kang, and Dae Suk Han

μl color reagent per well. The intensity of the color was measured in an ELISA reader at 540 nm. Electrophoretic mobility shift assay (EMSA) Podocytes were treated with 100 nM angiotensin II for 8 h. Nuclear

Free access

Luc J Martin and Jacques J Tremblay

& Tremblay 2008 , Martin et al . 2008 ), in vitro approaches such as EMSA showed a weak binding of NUR77 to the −95 bp SF1/NBRE element ( Martin et al . 2008 ), suggesting that recruitment of NUR77 to the Star promoter might be mediated by interactions

Free access

Suryaprakash Raichur, Patrick Lau, Bart Staels, and George E O Muscat

cell line. Whether RORγ regulates the LPL promoter by primary (direct) and/or secondary mechanisms remain unclear at present. Future studies utilizing ChIP, deletion and EMSA analysis will be used to address these questions. These latter

Free access

Zhen-Yu She and Wan-Xi Yang

assay (EMSA) and ChIP demonstrate that SOX9 recognizes and binds to the paired SOX-binding sites within the Pgd 2 promoter and then regulates the expression of Pgd 2 to ensure the sufficient Sertoli cells differentiation during normal sex development